|1.||Goedert, Michel: 2 articles (01/2005 - 03/2002)|
|2.||Cuenda, Ana: 1 article (01/2005)|
|3.||Jakes, Ross: 1 article (01/2005)|
|4.||Campbell, David G: 1 article (01/2005)|
|5.||Feijoo, Carmen: 1 article (01/2005)|
|6.||Buée-Scherrer, Valérie: 1 article (03/2002)|
01/15/2005 - "These findings suggest a role for Tau in the adaptative response of neurons to stress and indicate that SAPK4/p38delta and/or SAPK3/p38delta may contribute to the hyperphosphorylation of Tau in the human tauopathies."
03/27/2002 - "These findings indicate that the aberrant activation of SAP kinases, especially SAPK3/p38gamma and SAPK4/p38delta, could play an important role in the abnormal hyperphosphorylation of tau that is an invariant feature of the tauopathies."
01/15/1997 - "Stress-activated protein kinase-3 (SAPK3), a recently described MAP kinase family member with a wide-spread tissue distribution, was transfected into several mammalian cell lines and shown to be activated in response to cellular stresses, interleukin-1 (IL-1) and tumour necrosis factor (TNF) in a similar manner to SAPK1 (also termed JNK) and SAPK2 (also termed p38, RK, CSBP and Mxi2). "
|1.||Mitogen-Activated Protein Kinase 13
|2.||Mitogen-Activated Protein Kinase 12
|4.||Mitogen-Activated Protein Kinase 11
|5.||Interleukin-1 (Interleukin 1)