|1.||Bonny, Christophe: 14 articles (01/2012 - 09/2003)|
|2.||Zhang, Guang-Yi: 11 articles (04/2012 - 02/2005)|
|3.||Firestein, Gary S: 7 articles (06/2015 - 03/2002)|
|4.||Pei, Dong-Sheng: 7 articles (01/2012 - 02/2005)|
|5.||Hirt, Lorenz: 7 articles (01/2012 - 09/2003)|
|6.||Yoshimura, Koichi: 6 articles (01/2015 - 12/2005)|
|7.||Chen, Jun: 6 articles (01/2012 - 06/2005)|
|8.||Ji, Ru-Rong: 6 articles (11/2010 - 03/2006)|
|9.||Borsello, Tiziana: 6 articles (11/2009 - 09/2003)|
|10.||Kaneto, Hideaki: 6 articles (02/2008 - 10/2004)|
09/01/2015 - "Leukocyte cell-derived chemotaxin 2 (LECT2) is a recently discovered novel hepatokine, leading to skeletal muscle insulin resistance by activating c-Jun N-terminal kinase (JNK). "
07/01/2015 - "Moreover, cocoa-rich diet suppressed c-Jun N-terminal kinase and p38 activation caused by insulin resistance. "
10/01/2013 - "A relatively recent investigation finds the role of c-Jun-N-terminal kinase (JNK3) in AD which also seems to bear a link with insulin resistance. "
09/01/2013 - "The stress-activated c-Jun N-terminal kinase (JNK) has been increasingly recognized as a central mediator of insulin resistance. "
04/05/2013 - "HHcy activated c-Jun N-terminal kinase (JNK), the downstream signal of ER stress in adipose tissue, and activated JNK participated in insulin resistance by inhibiting Akt activation. "
|2.||Brain Injuries (Brain Injury)
12/01/2013 - "Previous studies have strongly suggested that c-Jun N-terminal kinase (JNK) signaling pathway plays a critical role in ischemic brain injury. "
05/12/2006 - "Our previous studies and the others have strongly suggested that c-Jun N-terminal kinase (JNK) signaling pathway plays a critical role in ischemic brain injury. "
01/30/2006 - "Our previous studies and those of others have strongly suggested that c-Jun N-terminal kinase (JNK) signaling pathway plays a critical role in ischemic brain injury. "
02/01/2012 - "[Effect of expression of c-jun N-terminal kinase on neuron autophagy following diffuse brain injury in rats]."
08/01/2011 - "It is demonstrated that the c-Jun N-terminal kinase (JNK) signaling pathway plays a critical role in ischemic brain injury. "
01/01/2014 - "c-Jun N-terminal kinase (JNK), a stress-activated MAPK, is activated during cardiac ischemia-reperfusion (IR). "
10/01/2009 - "Using a simulated ischemia-reperfusion (I/Re) model with primary cultured rat neonatal cardiomyocytes, I/Re induced a rapid, time-dependent phosphorylation of c-Jun N-terminal kinase (JNK), with significant elevation at 0.25 h and a peak at 0.5h during reperfusion. "
12/01/2008 - "To investigate the effect and mechanism of JNK mitogen-activated protein kinases (JNK MAPKs) inhibitor SP 600125 on hemodynamics after ischemia/reperfusion (I/R) injury to heart in anesthetized rats. "
06/01/2008 - "Whether the response of the fetal heart to ischemia-reperfusion is associated with activation of the c-Jun N-terminal kinase (JNK) pathway is not known. "
11/30/2007 - "The d-retro-inverso form of c-Jun N-terminal kinase-inhibitor (D-JNKI1), a cell-permeable inhibitor of JNK, powerfully reduces neuronal death induced by permanent and transient ischemia, even when administered 6 h after the ischemic insult, offering a clinically relevant window. "
05/01/2012 - "To investigate the protective effects and mechanism of SP600125-specificity inhibitor of c-Jun N-terminal kinase (JNK)on lung ischemia /reperfusion injury in rats. "
05/08/2007 - "Accumulating evidence suggests that c-Jun N-terminal kinase (JNK) signaling pathway plays a critical role in renal ischemia/reperfusion injury. "
05/15/2006 - "In small intestinal ischemia reperfusion injury, we investigated the pathophysiological role of c-Jun NH2 terminal kinase (JNK) and p38 in order to determine whether the dual inhibition of JNK and p38 was beneficial. "
05/15/2006 - "JNK (c-Jun NH2 terminal kinase) and p38 during ischemia reperfusion injury in the small intestine."
08/01/2003 - "Apoptotic cell death and c-Jun N-terminal kinase (JNK) activation occur after hepatic ischemia/reperfusion injury. "
04/01/2012 - "The objective of this study was to investigate the effects of the c-Jun N-terminal kinase (JNK) signaling pathway on rats' acute pancreatitis-associated lung injury (APALI). "
04/01/2012 - "Effects of c-Jun N-terminal kinase signaling pathway on severe acute pancreatitis-associated lung injury."
07/25/2008 - "In vitro studies using freshly prepared pancreatic acini show that genetic deletion of PAR2 reduces bile salt-induced pathological calcium transients, acinar cell injury, and activation of c-Jun N-terminal kinase, whereas genetic deletion of PAR2 has the opposite or no effect on these pancreatitis-related events when they are elicited, in vitro, by caerulein stimulation. "
05/01/2015 - "Additionally, GS treatment suppressed the activation of extracellular signal-regulated protein kinase (ERK) and c-Jun N-terminal kinase (JNK) in the pancreas in cerulein-induced pancreatitis. "
09/24/2000 - "Mitogen-activated protein kinase (MAPK) family members such as c-jun N-terminal kinase (JNK) may act as signal transducers early during pancreatitis development and evidence indicates that MAPK phosphatases (MKP) downregulate MAPK. "
|3.||Protein Kinases (Protein Kinase)
|5.||Reactive Oxygen Species (Oxygen Radicals)
|7.||Mitogen-Activated Protein Kinase 3
|9.||Messenger RNA (mRNA)
|3.||Drug Therapy (Chemotherapy)