|1.||Nakano, Hiroyasu: 4 articles (05/2014 - 04/2004)|
|2.||Zhou, Yan: 3 articles (01/2014 - 01/2013)|
|3.||Croft, Michael: 2 articles (05/2014 - 04/2004)|
|4.||So, Takanori: 2 articles (05/2014 - 04/2004)|
|5.||Guan, Hongjing: 2 articles (02/2014 - 08/2013)|
|6.||Li, Hongliang: 2 articles (02/2014 - 08/2013)|
|7.||Zhang, Yan: 2 articles (02/2014 - 08/2013)|
|8.||Fang, Jing: 2 articles (01/2014 - 01/2013)|
|9.||Hou, Shengping: 2 articles (01/2014 - 01/2013)|
|10.||Bai, Lin: 2 articles (01/2014 - 01/2013)|
12/01/2008 - "Collectively, these studies identify TRAF5 as an important positive signaling element that enhances T cell expansion and pathogen containment by providing a survival advantage to responding Ag-specific CD8(+) T cells during infection."
12/01/2008 - "We report in this study that TRAF5 was required for optimal CD8(+) T cell responses following infection with Listeria monocytogenes expressing OVA (LM-OVA). "
01/01/2010 - "TRAF5 may be a key molecule in the innate response against viral infection."
12/01/2008 - "TRAF5 was necessary for optimal T cell expansion following primary infection with LM-OVA, and its absence resulted in fewer memory CD8(+) T cells following LM-OVA infection, together with higher bacterial loads in the liver. "
12/01/2008 - "However, the potential importance of TRAF5 in cellular immune responses to infection or in T cell expansion and memory have not been studied. "
09/17/2010 - "This study tested the hypothesis that TRAF5 modulates atherogenesis in vivo. "
09/17/2010 - "TRAF5 deficiency accelerates atherogenesis in mice by increasing inflammatory cell recruitment and foam cell formation."
09/17/2010 - "Unexpectedly, TRAF5 deficiency accelerates atherogenesis in mice, an effect likely mediated by increased inflammatory cell recruitment to the vessel wall and enhanced foam cell formation."
|3.||Inflammatory Bowel Diseases (Inflammatory Bowel Disease)
01/01/2013 - "The aim of the present study is to explore the potential roles of TRAF3 and TRAF5 in patients with inflammatory bowel disease. "
01/01/2013 - "TRAF3 and TRAF5 are overexpressed in inflammatory bowel disease. "
01/01/2013 - "However, the expressions of TRAF3 and TRAF5 in patients with inflammatory bowel disease have not been elucidated. "
01/01/2013 - "Up-regulation and pre-activation of TRAF3 and TRAF5 in inflammatory bowel disease."
|4.||Uveomeningoencephalitic Syndrome (Vogt Koyanagi Harada Syndrome)
01/01/2014 - "This study provides evidence that TRAF5 and TRAF3IP2 genes are involved in the development of BD and VKH syndrome. "
01/01/2014 - "Three SNPs (rs6540679, rs12569232, rs10863888) of TRAF5 and rs13210247 of TRAF3IP2 were significantly associated with Behçet's disease and VKH syndrome (corrected P values ranging from 9.45×10(-12) to 0.027). "
01/01/2014 - "TRAF5 and TRAF3IP2 gene polymorphisms are associated with Behçet's disease and Vogt-Koyanagi-Harada syndrome: a case-control study."
|1.||TNF Receptor-Associated Factor 3
|2.||TNF Receptor-Associated Factor 5
|3.||TNF Receptor-Associated Factor 2 (TRAF2)
|4.||NF-kappa B (NF-kB)
|5.||Tumor Necrosis Factor-alpha (Tumor Necrosis Factor)
|6.||Messenger RNA (mRNA)
|7.||TNF Receptor-Associated Factor 4
|8.||Lymphotoxin beta Receptor
|9.||TNF Receptor-Associated Factor 6 (TRAF6)
|10.||Interleukin-17 (Interleukin 27)