|1.||Healy, Jasmine: 1 article (01/2015)|
|2.||Ouimet, Manon: 1 article (01/2015)|
|3.||Cassart, Pauline: 1 article (01/2015)|
|4.||Spinella, Jean-François: 1 article (01/2015)|
|5.||Sinnett, Daniel: 1 article (01/2015)|
|6.||Richer, Chantal: 1 article (01/2015)|
|7.||Saillour, Virginie: 1 article (01/2015)|
|8.||Symington, Lorraine S: 1 article (04/2014)|
|9.||Eissler, Christie L: 1 article (04/2014)|
|10.||Powers, Brendan L: 1 article (04/2014)|
09/06/2002 - "Accordingly, we show that the hypersensitivity to agents that cause replication fork stalling of mus81, eme1, and rqh1 mutants is suppressed by a Holliday junction resolvase (RusA), as is the synthetic lethality of a mus81(-) rqh1(-) double mutant. "
09/01/2011 - "Here, we examine the basis of hypersensitivity to thymine deprivation (hyper-TLD) in mutants that lack the UvrD helicase, which opposes RecA action and participates in some DNA repair mechanisms, RecBCD exonuclease, which degrades double-stranded linear DNA and works with RecA in double-strand-break repair and SOS induction, and RuvABC Holliday-junction resolvase. "
07/01/2001 - "To investigate the role of the vaccinia virus Holliday junction resolvase during an infection, we constructed two recombinant viruses: vA22-HA, which has a short C-terminal epitope tag appended to the A22R open reading frame, and vA22i, in which the original A22R gene is deleted and replaced by an inducible copy. "
|4.||Fanconi Anemia (Fanconi's Anemia)
|5.||Breast Neoplasms (Breast Cancer)
03/01/2013 - "We further revealed that the c.2515_2519delAAGTT homozygous mutation in a Holliday junction resolvase, GEN1, was overrepresented in women with bilateral breast cancer (BC) as compared to healthy controls [11/360 (3.1 %) vs. 18/1305 (1.4 %); odds ratio (OR) = 2.25 (1.02-4.75); p = 0.031], although this trend was not maintained in unilateral BC patients [23/1851 (1.2 %)]. "
|2.||DNA (Deoxyribonucleic Acid)