|1.||Edwards, Robert A: 3 articles (08/2009 - 06/2007)|
|2.||Birnbaumer, Lutz: 3 articles (08/2009 - 06/2007)|
|3.||Kilic, E: 2 articles (07/2013 - 09/2009)|
|4.||Vaarwater, J: 2 articles (07/2013 - 09/2009)|
|5.||de Klein, A: 2 articles (07/2013 - 09/2009)|
|6.||Wang, Kehui: 2 articles (08/2009 - 07/2008)|
|7.||Weinstein, Lee S: 2 articles (03/2007 - 09/2003)|
|8.||Özsu, Elif: 1 article (06/2015)|
|9.||Çizmecioğlu, Filiz Mine: 1 article (06/2015)|
|10.||Mutlu, Gül Yeşiltepe: 1 article (06/2015)|
06/01/1996 - "Low incidence of the stimulatory G protein alpha-subunit mutations in autonomously functioning thyroid adenomas in Japan."
03/01/2008 - "Some studies have found better responses to somatostatin analogues in G-protein alpha subunit (Gsalpha) mutation (gsp oncogene)-positive adenomas. "
02/15/1993 - "Stimulation of human thyroid growth via the inhibitory guanine nucleotide binding (G) protein Gi: constitutive expression of the G-protein alpha subunit Gi alpha-1 in autonomous adenoma."
12/01/2006 - "Moreover, alteration in genes encoding for second messengers may cause forms of resistance other than those due to receptor mutations, the most important one being that of an inactivating mutation in the G-protein alpha-subunit leading to TSH resistance in the setting of pseudohypoparathyroidism type 1a. "
05/01/2006 - "Most loss of function mutations of GNAS identified in different forms of pseudohypoparathyroidism disrupt not only the stimulatory G protein alpha-subunit (Gsalpha), but also its paternally expressed variant, XLalphas. "
09/01/2003 - "The stimulatory G protein alpha-subunit Gs alpha is imprinted in human thyroid glands: implications for thyroid function in pseudohypoparathyroidism types 1A and 1B."
03/01/2002 - "Pseudohypoparathyroidism: a multitude of mutations in the stimulatory G protein alpha subunit (Gsalpha)."
07/01/2001 - "Pseudohypoparathyroidism Ia (PHP Ia) is characterized by resistance to PTH and many other stimuli because of deficiency of stimulatory G protein alpha-subunit. "
05/01/2009 - "A missense mutation in the non-neural G-protein alpha-subunit isoforms modulates susceptibility to obesity."
03/01/2007 - "Obesity is a prominent feature of Albright hereditary osteodystrophy (AHO), a disorder caused by heterozygous GNAS mutations that disrupt the stimulatory G protein alpha-subunit Galpha(s). "
07/21/1998 - "Albright hereditary osteodystrophy (AHO), an autosomal dominant disorder characterized by short stature, obesity, and skeletal defects, is associated with heterozygous inactivating mutations of GNAS1, the gene encoding the heterotrimeric G protein alpha-subunit (Gsalpha) that couples multiple receptors to the stimulation of adenylyl cyclase. "
|1.||Somatostatin (Somatotropin Release-Inhibiting Factor)
|2.||GTP-Binding Proteins (G-Protein)
|3.||Adenylate Cyclase (Adenylyl Cyclase)
|4.||Messenger RNA (mRNA)
|5.||Human Growth Hormone (Saizen)
|7.||Albright's hereditary osteodystrophy
|9.||Type 4 Fibroblast Growth Factor Receptor