|1.||Ward, Peter A: 7 articles (02/2015 - 01/2008)|
|2.||Zetoune, Firas S: 4 articles (02/2015 - 05/2008)|
|3.||Sarma, J Vidya: 4 articles (12/2013 - 01/2008)|
|4.||Grailer, Jamison J: 2 articles (02/2015 - 12/2013)|
|5.||Fairlie, David P: 2 articles (07/2014 - 06/2004)|
|6.||Gerard, Craig: 2 articles (01/2014 - 05/2008)|
|7.||Bosmann, Markus: 2 articles (12/2013 - 07/2011)|
|8.||Lambris, John D: 2 articles (10/2013 - 11/2009)|
|9.||Yeh, Chiu-Li: 2 articles (06/2013 - 08/2012)|
|10.||Yeh, Sung-Ling: 2 articles (06/2013 - 08/2012)|
11/17/2009 - "The results of this study indicate that simultaneous blockade of the C3a and C5a receptors represents a promising neuroprotective strategy in hemorrhagic stroke."
02/01/2014 - "The results of this study indicated that simultaneous blockade of the thrombin and C5a receptors represent a promising neuroprotective strategy in haemorrhagic stroke. "
03/15/2001 - "Thus, normal rodent hippocampal neurons as well as undifferentiated and differentiated human neuroblastoma cells express functional C5a receptors. "
03/15/2001 - "Additional studies with human neuroblastoma cells conducted to address the functional role of C5a receptors revealed that C5a triggered rapid activation of protein kinase C and activation and nuclear translocation of the NF-kappa B transcription factor. "
|3.||Folic Acid Deficiency (Folic Acid Deficiencies)
10/01/1991 - "The maximum decrease in C5a receptors was measured at the 15-min sampling time, when the number of C5a receptor decreased from 189,240 +/- 24,500 predialysis to 160,740 +/- 19,380 receptors (P was not significant) at the nadir of granulocytopenia. "
10/01/1991 - "These studies suggest that down-regulation of C5a receptors, which is seen maximally at 15 min after initiation of dialysis, does not sufficiently account for the reversal of granulocytopenia during hemodialysis."
10/01/1991 - "The rapid reversal of granulocytopenia during hemodialysis has previously been ascribed to down-regulation of granulocyte C5a receptors. "
01/01/2012 - "New insights for C5a and C5a receptors in sepsis."
01/01/2010 - "Neutralization of C5a in vivo or absence/blockade of C5a receptors greatly reduces the adverse events in the setting of sepsis, markedly attenuates MOF, and greatly improves survival. "
12/01/2008 - "This communication reviews the evidence for the adverse roles of C5a and C5aR in the setting of experimental sepsis and linkages to the various complications of sepsis, especially apoptosis as well as the roles of the two C5a receptors (C5aR and C5L2) in experimental sepsis."
05/01/2008 - "Functional roles for C5a receptors in sepsis."
03/01/2008 - "During sepsis, there is up-regulation of C5a receptors (C5aR, C5L2) on blood polymorphonuclear neutrophils (PMNs) and in lungs, liver, kidneys, and heart. "
|1.||NF-kappa B (NF-kB)
|2.||Transcription Factors (Transcription Factor)
|4.||Protein Kinase C
|5.||Complement System Proteins (Complement)
|6.||Interleukin-17 (Interleukin 27)
|7.||CD55 Antigens (Decay Accelerating Factor)
|8.||Complement C3a (Complement 3a)
|9.||Complement Receptors (Complement Receptor)
|1.||Renal Dialysis (Hemodialysis)