|1.||Fujii, N: 2 articles (03/2004 - 11/2000)|
|2.||Liu, Ping: 1 article (01/2011)|
|3.||Zhang, Yi: 1 article (01/2011)|
|4.||Su, Sheng: 1 article (01/2011)|
|5.||Cao, Wenping: 1 article (01/2011)|
|6.||Zhang, Lu: 1 article (01/2011)|
|7.||Tang, Xianling: 1 article (01/2011)|
|8.||Nguyen, Angeline M: 1 article (07/2010)|
|9.||Saraswathy, Sindhu: 1 article (07/2010)|
|10.||Rao, Narsing A: 1 article (07/2010)|
08/15/2005 - "Pb2+ exposure alters the lens alpha A-crystallin protein profile in vivo and induces cataract formation in lens organ culture."
03/01/2004 - "Comparison of post-translational modifications of alpha A-crystallin from normal and hereditary cataract rats."
12/26/2000 - "Structural and functional changes in the alpha A-crystallin R116C mutant in hereditary cataracts."
02/15/2000 - "These findings suggest that the molecular basis for the congenital cataract with the alpha A-R116C mutation is due to the generation of a highly oligomerized alpha A-crystallin having a modified structure and decreased chaperone-like function."
02/15/2000 - "An autosomal dominant congenital cataract in human is associated with mutation of Arg-116 to Cys (R116C) in alpha A-crystallin. "
02/01/1996 - "Mice with the alpha A-crystallin promoter exhibit bilateral lens tumors and PNET of the midbrain. "
11/01/1989 - "We report on two lines of transgenic mice that express a murine alpha A-crystallin/SV40 tumor antigen fusion gene in the eye lens. "
11/01/1987 - "A fusion gene consisting of this alpha A-crystallin promoter sequence and the T-antigen gene of SV40 produced a lens tumor in transgenic mice. "
03/27/1987 - "Transgenic mice carrying a hybrid gene comprising the murine alpha A-crystallin promoter (-366 to +46) fused to the coding sequence of the SV40 T antigens developed lens tumors, which obliterated the eye cavity and even invaded neighboring tissue, thus establishing that the lens is not refractive to oncogenesis. "
07/15/1992 - "Seven transgenic mouse founder lines were produced carrying the murine lens-specific alpha A-crystallin promoter and the simian virus 40 large tumor-antigen gene sequence, separated by a 1.3-kilobase-pair Stop sequence that contains elements preventing expression of the large tumor-antigen gene and Cre recombinase recognition sites. "
|3.||Primitive Neuroectodermal Tumors (PNET)
|4.||T-Cell Leukemia (Leukemia, T Cell)
02/01/1995 - "Stimulation of alpha A-crystallin promoter activity via 8-bromo-cAMP, forskolin, or human T-cell leukemia virus type I Tax1 in transfections and reduction of activity of this site in cell-free transcription tests by competition with the somatostatin CRE supported the idea that DE1 is a functional CRE. "
|4.||Tumor Viral Antigens (Large T Antigen)
|6.||Aldehyde Reductase (Aldose Reductase)
|7.||Human immunodeficiency virus 1 p16 protease
|8.||Facilitative Glucose Transport Proteins (Glucose Transporter)
|10.||Molecular Chaperones (Chaperone, Molecular)