|1.||Pasquale, Elena B: 6 articles (02/2013 - 04/2004)|
|2.||Terracciano, Luigi M: 3 articles (12/2011 - 06/2008)|
|3.||Zlobec, Inti: 3 articles (12/2011 - 06/2008)|
|4.||Wang, Lin-Fa: 3 articles (09/2011 - 07/2005)|
|5.||Broder, Christopher C: 3 articles (09/2011 - 07/2005)|
|6.||Vogt, Thomas: 3 articles (07/2007 - 07/2005)|
|7.||Landthaler, Michael: 3 articles (07/2007 - 07/2005)|
|8.||Meyer, Stefanie: 3 articles (07/2007 - 07/2005)|
|9.||Zhao, Y: 2 articles (05/2015 - 06/2013)|
|10.||Adams, Ralf H: 2 articles (12/2013 - 04/2010)|
01/01/2010 - "Combination of Dll4/Notch and Ephrin-B2/EphB4 targeted therapy is highly effective in disrupting tumor angiogenesis."
01/01/2010 - "We used the transgenic RIP1-Tag2 tumor model to study the effects of 1) inhibition of Dll4/Notch by either Dll4 allelic deletion or use of a soluble extracellular Dll4 (sDll4), 2) inhibition of Ephrin-B2/EphB4 signaling by a soluble extracellular EphB4 fused to albumin (sEphB4-Alb), and 3) inhibition of both pathways by sEphB4-Alb combined with either Dll4 allelic deletion or sDll4. "
09/01/2006 - "In this study, we assessed the expression of DLL4, Notch4, and ephrin B2 in transgenic mice developing hepatocarcinoma characterized by a strong remodeling of the tumor sinusoids. "
05/01/2015 - "Ephrin-B2 is found to be of great importance in various cell functions in both benign human tissue and tumors. "
04/10/2015 - "Proteolytic cleavage of endothelial cell ephrin-B2 has previously been suggested as one mechanism whereby the interaction between tumour cell-expressed EphB4 and endothelial cell ephrin-B2 is regulated to support both cancer promotion and angiogenesis. "
|2.||Spinal Cord Injuries (Spinal Cord Injury)
06/25/2013 - "Improved axonal regeneration after spinal cord injury in mice with conditional deletion of ephrin B2 under the GFAP promoter."
06/17/2013 - "Our data are consistent with a role for ephrin-B2 in transmission of positional information to the CNS, and thus suggest a role in synaptic plasticity of spinal cord locomotor circuits that are known to be sensitive to proprioceptive sensory input after spinal cord injury."
08/27/2003 - "In response to thoracic spinal cord transection lesions, ephrin-B2 and EphB2 protein levels exhibit an initial decrease (1 d after lesion), followed by a significant increase by day 14. "
12/01/2009 - "This article reviews the current knowledge about natural and experimental infections in different mammals, focusing on the main organ and cell tropism in vivo, and summarises some recent studies in cell culture on the role of ephrin-B2 and -B3 receptors in NiV infection of endothelial cells."
01/01/2008 - "Since expression levels of the main NiV entry receptor ephrin-B2 (EB2) are highly regulated in vivo to fulfill the physiological functions in axon guidance and angiogenesis, the goal of this study was to determine if changes in the EB2 expression influence NiV infection. "
10/01/2014 - "[Ephrin B2 is involved in Porphyromonas gingivalis infection-enhanced adhesion of THP-1 to human umbilical vein endothelial cells]."
09/01/2011 - "Mutations in the G-H loop region of ephrin-B2 can enhance Nipah virus binding and infection."
01/01/2008 - "Ephrin-B2 expression critically influences Nipah virus infection independent of its cytoplasmic tail."
06/17/2013 - "Recent studies have shown that ephrin-B2 on sensory afferent fibers from the dorsal root ganglia (DRG) controls transmission of pain sensation to the spinal cord. "
01/01/2010 - "To examine the role of ephrin-B2 in peripheral pain pathways, we deleted ephrin-B2 in Nav1.8+ nociceptive sensory neurons with the Cre-loxP system. "
01/01/2010 - "Presynaptic ephrin-B2 expression thus plays an important role in regulating inflammatory pain through the regulation of synaptic plasticity in the dorsal horn and is also involved in the pathogenesis of some types of neuropathic pain."
01/01/2010 - "Pain behavior in acute, inflammatory and neuropathic pain models was assessed in the ephrin-B2 conditional knockout (CKO) mice. "
01/01/2010 - "Although acute pain behavior and motor co-ordination were normal, inflammatory pain was attenuated in ephrin-B2 mutant mice. "
11/01/2005 - "Ephrin-B2 overexpression enhances integrin-mediated ECM-attachment and migration of B16 melanoma cells."
01/01/2011 - "Consistent with increased stability, submicromolar concentrations of PEGylated TNYL-RAW effectively impair EphB4 activation by ephrin-B2 in cultured B16 melanoma cells as well as capillary-like tube formation and capillary sprouting in co-cultures of endothelial and epicardial mesothelial cells. "
07/01/2007 - "In this study, we show that overexpressed ephrin-B2 is not only compartmentalized to classical Triton X-100 RAFTs in B16 melanoma cells, but also to the recently defined Lubrol-RAFTs. "
|1.||Vascular Endothelial Growth Factor A (Vascular Endothelial Growth Factor)
|3.||Eph Family Receptors (Eph Receptors)
|4.||ral Guanine Nucleotide Exchange Factor
|5.||Octoxynol (Triton X 100)
|9.||Messenger RNA (mRNA)
|1.||Heterologous Transplantation (Xenotransplantation)