|1.||Wang, Zhengguang: 2 articles (07/2015 - 01/2013)|
|2.||Liu, Zhendong: 1 article (07/2015)|
|3.||Wu, Song: 1 article (07/2015)|
|4.||Liu, Gengyan: 1 article (07/2015)|
|5.||Liu, Bo: 1 article (07/2015)|
|6.||Roche, O: 1 article (05/2015)|
|7.||Volpert, O: 1 article (05/2015)|
|8.||Fernandez-Barral, A: 1 article (05/2015)|
|9.||del Peso, L: 1 article (05/2015)|
|10.||Jensen, L: 1 article (05/2015)|
|1.||Nerve Sheath Neoplasms (Nerve Sheath Neoplasm)
07/01/2015 - "Our previous study indicated that miR-210-mediated Ephrin-A3 (EFNA3) promotion of proliferation and invasion of MPNST cells plays an important role in MPNST tumorigenesis and progression. "
01/01/2013 - "In this study, miR-210 was identified as downregulated in MPNST cells, and its potential target ephrin-A3 (EFNA3) was upregulated in them compared with neurofibroma cells using quantitative real-time (qRT)-PCR. "
11/01/2014 - "In the present study, we found that the EphA4 receptor and its ephrin-A3 ligand, which were distributed in neurons and astrocytes, respectively, in the hippocampus showed a coincident up-regulation of protein expression in the early stage of ischemia. "
11/01/2014 - "Ephrin-A3 reverse signaling regulates hippocampal neuronal damage and astrocytic glutamate transport after transient global ischemia."
07/01/2015 - "Dissecting the roles of Ephrin-A3 in malignant peripheral nerve sheath tumor by TALENs."
05/14/2015 - "We demonstrate that sustained expression of both Ephrin-A3 and novel EFNA3 lncRNAs increased the metastatic potential of human breast cancer cells, possibly by increasing the ability of tumor cells to extravasate from the blood vessels into surrounding tissue. "
01/01/2013 - "Here we show that in cancer cells, coexpressed ephrin-A3 can inhibit the ability of EphA2 and EphA3 to bind ephrins in trans and become activated, while ephrin-B2 can inhibit not only EphB4 but also EphA3. "
08/15/2002 - "A second mRNA profile that included ephrin-A3, ephrin-A5, and ephrin-B1 was expressed by a subset of tumors. "
05/14/2015 - "Here, we show that hypoxia induces the expression of Ephrin-A3 through a novel hypoxia-inducible factor (HIF)-mediated mechanism. "
06/06/2008 - "We determined that one relevant target of miR-210 in hypoxia was Ephrin-A3 since miR-210 was necessary and sufficient to down-modulate its expression. "
05/14/2015 - "Taken together, our results suggest that hypoxia could contribute to metastatic spread of breast cancer via HIF-mediated induction of EFNA3 lncRNAs and subsequent Ephrin-A3 protein accumulation. "
05/14/2015 - "In response to hypoxia, the coding EFNA3 mRNA levels remained relatively stable, but HIFs drove the expression of previously unknown long noncoding (lnc) RNAs from EFNA3 locus and these lncRNA caused Ephrin-A3 protein accumulation. "
07/09/2008 - "Hypoxia induces microRNA miR-210 in vitro and in vivo ephrin-A3 and neuronal pentraxin 1 are potentially regulated by miR-210."
|2.||Ephrin-B2 (Ephrin B2)
|3.||Messenger RNA (mRNA)
|7.||Vascular Endothelial Growth Factor C
|8.||Vascular Endothelial Growth Factor A (Vascular Endothelial Growth Factor)
|10.||Ephrin-B3 (Ephrin B3)