|1.||Vanderhyden, Barbara C: 2 articles (01/2014 - 11/2013)|
|2.||Freiman, Richard N: 2 articles (01/2014 - 11/2013)|
|3.||Pugh, B Franklin: 2 articles (02/2011 - 01/2011)|
|4.||Wei, Haixiang: 1 article (07/2014)|
|5.||Wu, Hao: 1 article (07/2014)|
|6.||Wen, Hai: 1 article (07/2014)|
|7.||Zhao, Jian: 1 article (07/2014)|
|8.||Chen, Fangyi: 1 article (07/2014)|
|9.||Zhao, Jinlong: 1 article (07/2014)|
|10.||Ding, Tao: 1 article (07/2014)|
01/01/2011 - "Our data suggest that MMS-induced genes undergo transcription complex assembly sequentially, first involving SAGA and then involving a slower TFIID recruitment, whereas heat shock genes utilize the SAGA and TFIID pathways rapidly and in parallel. "
01/01/2003 - "Furthermore, the TFIID binding site, cap signal for transcription initiation, PEA3 motif, heat shock factor binding motif, and many other motifs were found in the TAUT promoter region, and require characterization."
01/01/1998 - "The heat shock genes provide an example for transcriptional regulation at a later step: in nuclei TFIID can be detected at the TATA box prior to heat induction. "
06/03/1994 - "Northern analysis indicated that weak, inducible expression of the mutant promoter occurred within the same period of heat shock as for the normal promoter, suggesting that TFIID was associated with the mutant promoter prior to heat shock. "
09/15/1993 - "Transcription factor TFIID recognizes DNA sequences downstream of the TATA element in the Hsp70 heat shock gene."
06/01/2011 - "Taken together, the data suggest that ICP4 interacts with components of TFIID and Mediator in the context of viral infection, and this may explain the broad transactivation properties of ICP4."
11/25/1998 - "These results indicated that VSV infection leads to an inhibition of host transcription by inactivation of TFIID rather than reduction in the level of TFIID."
09/01/1990 - "These studies further support the idea that for maximal response to the HCMV infection, the 2.7-kb RNA promoter requires multiple cis-acting sequences, two of which include the binding sites for USF/MLTF and TFIID."
06/01/2001 - "We propose that such interactions could be favored upon viral DNA replication since TFIID concentrates to viral transcription foci that form during the later stages of infection."
02/01/2006 - "Our results show that HSV-1 infection has no influence on the protein levels of TFIID components and leads to a redistribution of TBP and hsTAFs to prereplicative sites that enlarge to viral DNA replication compartments."
01/01/2014 - "At the biochemical level, TAF2 directs TFIID to TATA-less promoters by contact with an Initiator element, which may lead to the deregulation of the transcriptional output of these tumor cells. "
04/11/1995 - "These results suggest that RB can confer transcriptional regulation and possibly cell cycle control and tumor suppression through an interaction with TFIID, in particular with TAFII250."
11/01/2013 - "Estrogen responsiveness of the TFIID subunit TAF4B in the normal mouse ovary and in ovarian tumors."
10/01/1995 - "Likewise, GST-E2TA bound TFIID and TFIIB present in a nuclear extract from the human cervical cancer-derived cell line, HeLa. "
07/01/2009 - "As a first step toward structurally dissecting activator/TFIID interactions, we determined the three-dimensional structures of TFIID bound to three distinct activators (i.e., the tumor suppressor p53 protein, glutamine-rich Sp1 and the oncoprotein c-Jun) and compared their structures as determined by electron microscopy and single-particle reconstruction. "
|4.||Retinoblastoma (Glioblastoma, Retinal)
07/24/1997 - "The retinoblastoma tumor suppressor gene product (Rb) binds directly to the largest TFIID subunit, TATA-binding protein associated factor TAF(II)250, first identified as the cell cycle regulatory protein CCG1. "
02/15/1993 - "The retinoblastoma (RB) tumor suppressor protein and the TATA-box-binding protein TFIID form contacts with a number of viral transactivator proteins. "
02/15/1993 - "The activation domain of transcription factor PU.1 binds the retinoblastoma (RB) protein and the transcription factor TFIID in vitro: RB shows sequence similarity to TFIID and TFIIB."
01/15/1992 - "A quantitative polymerase chain reaction procedure was developed and the human transcription factor IID (hTFIID) transcript was measured in normal human tissues, lung carcinomas, lung carcinoma cell lines, and breast carcinomas. "
06/01/2008 - "To investigate potential links between the regulation of TFIID function and the RAS-induced carcinogenesis, we monitored the expression of the TATA box-binding protein and its associated factors (TAF) in human colon carcinoma cells. "
01/15/1992 - "A general transcription initiation factor, human transcription factor IID, overexpressed in human lung and breast carcinoma and rapidly induced with serum stimulation."
|1.||RNA (Ribonucleic Acid)
|2.||Transfer RNA (tRNA)
|4.||TATA-Box Binding Protein (TATA-Binding Protein)
|5.||Transcription Factor TFIID (TFIID)
|6.||RNA Polymerase II (RNA Polymerase B)
|8.||tributyl phosphate (TBP)
|9.||Transcription Factor TFIIB (TFIIB)
|10.||Transcription Factors (Transcription Factor)