|1.||Villicaña, Claudia: 1 article (01/2014)|
|2.||Zurita, Mario: 1 article (01/2014)|
|3.||Cruz, Grisel: 1 article (01/2014)|
|4.||Petrosko, Patricia: 1 article (06/2012)|
|5.||Parwani, Anil V: 1 article (06/2012)|
|6.||LaFramboise, William A: 1 article (06/2012)|
|7.||Dhir, Rajiv: 1 article (06/2012)|
|8.||Kubal, Michael D: 1 article (06/2012)|
|9.||Kelly, Lori A: 1 article (06/2012)|
|10.||Chandran, Uma R: 1 article (06/2012)|
02/15/2010 - "While searching for sequence patterns that characterize DNA methylation states, we discovered several nonredundant sequences in CpG islands that were resistant to aberrant de novo methylation in cancer and that resembled consensus binding sites for general transcription factors (TF). "
01/01/2014 - "For example, drugs such as triptolide that targets the general transcription factors TFIIH and JQ1 to inhibit BRD4 are administered to target the high proliferative rate of cancer cells. "
11/01/2007 - "This very close relationship between E2F binding sites and binding sites for general transcription factors in both normal and tumor cells suggests that a chromatin-bound E2F may be a signpost for active transcription initiation complexes. "
12/01/2000 - "The retinoblastoma tumor suppressor protein targets distinct general transcription factors to regulate RNA polymerase III gene expression."
01/01/1996 - "Outside of the cell cycle, cyclin homologs play important roles in regulating the assembly of transcription complexes; distant structural relatives of the conserved cyclin core or "box" can also function as general transcription factors (like TFIIB) or survive embedded in the chain of the tumor suppressor, retinoblastoma protein. "
10/01/2001 - "We find that the RNAP II general transcription factors (GTFs) remain abundant after infection and are recruited into viral replication compartments, suggesting that they continue to be involved in viral gene transcription. "
07/26/2002 - "Transcriptional activation of the IFN-beta gene in response to virus infection requires the assembly of an enhanceosome, which instructs a recruitment program of chromatin modifiers/remodelers and general transcription factors to the promoter. "
09/01/2004 - "In the present work, chemical cross-linking and immunoprecipitation assays were used to probe the presence of activators, general transcription factors, and chromatin-modifying coactivators at IE gene promoters during infection of HeLa cells by wild-type HSV-1 and by RP5, a viral strain lacking the VP16 transcriptional activation domain. "
|3.||Oxyphilic Adenoma (Oncocytoma)
|5.||Retinoblastoma (Glioblastoma, Retinal)
|4.||Transcription Factor TFIIB (TFIIB)
|6.||Trans-Activators (Trans-Acting Factor)
|7.||Messenger RNA (mRNA)
|8.||RNA Polymerase III
|9.||RNA Polymerase II (RNA Polymerase B)