|1.||Zheng, Yixian: 3 articles (01/2015 - 01/2014)|
|2.||Chen, Haiyang: 3 articles (01/2015 - 01/2014)|
|3.||Shimi, Takeshi: 2 articles (01/2015 - 04/2012)|
|4.||Taimen, Pekka: 2 articles (01/2015 - 04/2012)|
|5.||Zheng, Xiaobin: 2 articles (01/2015 - 11/2014)|
|6.||Kondo, Tetsuo: 1 article (10/2015)|
|7.||Nakazawa, Tadao: 1 article (10/2015)|
|8.||Oish, Naoki: 1 article (10/2015)|
|9.||Katoh, Ryohei: 1 article (10/2015)|
|10.||Yamane, Tetsu: 1 article (10/2015)|
01/01/2002 - "Previous studies have shown that heat-shock may significantly modulate lamin B content. "
01/01/1999 - "While increased levels of lamin B during 45.5 degrees C heating do not appear to confer a survival advantage directly, a possible role for lamin B in cellular recovery after heat shock cannot be discounted."
07/01/1993 - "These heat-shock-induced changes were also accompanied by a dephosphorylation of lamins A and C but not of lamin B."
11/01/2000 - "Using Western blotting, the temporal patterns of cleavage of three nuclear matrix proteins (lamin B, NUMA and the nucleoporin TPR) were compared in HL60 cells induced to undergo apoptosis after irradiation, heat shock or treatment with etoposide. "
04/10/1998 - "In this report, the p53-null human leukemia HL60 cells were used to investigate phosphorylation and degradation of lamin B during apoptosis. "
04/05/1993 - "Using highly purified human beta II PKC and isolated nuclear envelopes from the human promyelocytic (HL60) leukemia cell line, we have now determined the major sites for beta II PKC-mediated lamin B phosphorylation. "
04/10/1998 - "Lamin B phosphorylation by protein kinase calpha and proteolysis during apoptosis in human leukemia HL60 cells."
12/01/1989 - "We have reported previously that the human promyelocytic leukemia cell line HL-60, in its undifferentiated state, is devoid of cytoplasmic intermediate filament proteins and nuclear lamins A and C, but does express lamin B. "
08/01/2000 - "In this study, we show that caspases 2, 3, 6, and 7 were activated during peroxynitrite-induced apoptosis in human leukemia HL-60 cells and that processing of these caspases was accompanied by cleavage of poly(ADP-ribose) polymerase and lamin B. "
|3.||Astrocytoma (Pilocytic Astrocytoma)
02/01/2005 - "To understand the function of APC in normal epithelium and how its loss leads to tumor formation, we tested whether APC is a mediator of apoptosis using an in vitro assay that monitors caspase-3-mediated cleavage of lamin B protein or a colorimetric substrate in a cell-free Xenopus egg extract. "
03/01/2006 - "In prostate cancer, lamin B undergoes a significant increase; interestingly, its nuclear content strongly correlates with tumor differentiation. "
01/01/1984 - "In proliferating cells (tumors, regenerating liver, log phase cell culture) a polypeptide band of 150 kD and lamin B were conspicuous at the expense of lamins A and C."
01/01/2002 - "The farnesyl transferase inhibitor RPR-130401 does not alter radiation susceptibility in human tumor cells with a K-Ras mutation in spite of large changes in ploidy and lamin B distribution."
11/01/2001 - "Tumor response correlated with an increase in the appearance of prelamin A, but no changes in the prenylation of lamin B, heat shock protein 40, or N-Ras were detectable. "
|5.||Systemic Lupus Erythematosus (Libman-Sacks Disease)
03/01/1987 - "In addition, autoantibodies to the nuclear lamina in sera of certain patients with systemic lupus erythematosus may be useful for defining the molecular structure and biological functions of lamin B, as well as for studying mechanisms of autoimmunity."
03/01/1987 - "Lamin B autoantibodies in sera of certain patients with systemic lupus erythematosus."
09/01/1992 - "Sera from certain patients with systemic lupus erythematosus (SLE) and autoimmune liver disease contain high titers of autoantibodies against lamin B. "
01/01/1991 - "Anti-lamin B autoantibodies are associated with both systemic lupus erythematosus (SLE) and autoimmune liver disease. "
05/01/1992 - "Autoantibodies to lamins, the major polypeptide components of the nuclear lamina, have been reported in selected sera from patients with autoimmune diseases, including anti-lamin B in systemic lupus erythematosus (SLE) and anti-lamins AC in autoimmune chronic active hepatitis (CAH). "
|1.||Peroxynitrous Acid (Peroxynitrite)
|4.||Poly(ADP-ribose) Polymerases (Poly ADP Ribose Polymerase)
|7.||Lamin Type A (Lamin A)
|8.||Caspase 3 (Caspase-3)
|9.||Proliferating Cell Nuclear Antigen (PCNA)
|10.||Protein Kinase C