|1.||McGivern, Joseph G: 1 article (12/2004)|
|2.||McDonough, Stefan I: 1 article (12/2004)|
|3.||Toru, S: 1 article (04/2000)|
|4.||Nagayama, S: 1 article (04/2000)|
|5.||Ishikawa, K: 1 article (04/2000)|
|6.||Osanai, M: 1 article (04/2000)|
|7.||Saegusa, H: 1 article (04/2000)|
|8.||Mizusawa, H: 1 article (04/2000)|
|9.||Murakoshi, T: 1 article (04/2000)|
|10.||Uchihara, T: 1 article (04/2000)|
10/01/1997 - "Thus P-type calcium channels seem to acquire a predominant importance in the excitation of spinal cord neurons by mechanosensory input from inflamed tissue and hence in the generation of inflammatory pain. "
12/01/2004 - "We describe the different families of voltage-gated calcium channels that are expressed in pain pathway neurons, how the expression levels of calcium channel currents change in chronic pain conditions, and the validation of N-type, T-type, and P-type calcium channels as targets for the treatment of pain. "
|2.||Small Cell Lung Carcinoma (Small Cell Lung Cancer)
|3.||Spinocerebellar Ataxias (Spinocerebellar Ataxia)
06/01/1999 - "The shift in the effect of omega-agatoxin, from slight facilitation or no change of the responses before inflammation to inhibition in the state of hyperexcitability, indicates that P-type calcium channels are important for excitatory synaptic transmission involved in the maintenance of inflammation-evoked hyperexcitability."
06/01/1999 - "Thus the development of inflammation-evoked hyperexcitability was attenuated by omega-agatoxin, and this suggests that P-type calcium channels in the spinal cord are involved in the generation of inflammation-evoked hyperexcitability of spinal cord neurons. "
06/01/1999 - "To investigate the importance of P-type calcium channels in the generation of inflammation-evoked hyperexcitability of spinal cord neurons, electrophysiological recordings were made from wide-dynamic-range neurons with input from the knee joint in the anesthetized rat. "
06/01/1999 - "Effects of omega-agatoxin IVA, a P-type calcium channel antagonist, on the development of spinal neuronal hyperexcitability caused by knee inflammation in rats."
|2.||N-Type Calcium Channels (N-Type Calcium Channel)
|3.||Calcium Channels (Calcium Channel)
|4.||Lipid Bilayers (Lipid Bilayer)
|5.||Immunoglobulin G (IgG)