|1.||Yamaguchi, Takeo: 1 article (01/2012)|
|2.||Iwata, Yohei: 1 article (01/2012)|
|3.||Kawada, Kazumasa: 1 article (01/2012)|
|4.||Miura, Shingo: 1 article (01/2012)|
|5.||Kitano, Hiromi: 1 article (04/2011)|
|6.||Saruwatari, Yoshiyuki: 1 article (04/2011)|
|7.||Ohno, Kohji: 1 article (04/2011)|
|8.||Iwanaga, Shintaroh: 1 article (04/2011)|
|9.||Kondo, Takuya: 1 article (04/2011)|
|10.||Suzuki, Hisatomo: 1 article (04/2011)|
|2.||Hepatocellular Carcinoma (Hepatoma)
04/08/2011 - "Upon ion beam irradiation, furthermore, the PCMB brush was ablated and a hollow space with a designed shape could be made to which HEK293 cells (from human embryonic kidney) and Hep G2 (from human hepatoma) cells non-specifically adhered, while no adhesion of these cells to the non-treated area on the brush was observed. "
02/01/1995 - "In contrast, NADH oxidase activity of rat liver plasma membrane was largely unaffected over the same range of PCMB concentrations that either stimulated or inhibited with rat hepatoma or HeLa cell plasma membranes. "
02/01/1995 - "With some hepatoma preparations, the NADH oxidase activity of hepatoma plasma membranes was stimulated rather than inhibited by PCMB, whereas with all preparations of hepatoma plasma membranes, NEM and DTNB stimulated the activity. "
01/01/2012 - "Recently, we have found that pressure-induced hemolysis is enhanced by inhibiting water transport via aquaporin-1 (AQP1), as seen in p-chloromercuribenzoate (pCMB)-treated erythrocytes. "
03/01/1956 - "Hemolysis of human erythrocytes by a sulfhydryl inhibitor, p-chloromercuribenzoic acid."
01/01/2012 - "The enhancement effect of pCMB on pressure-induced hemolysis was unaffected by TEA(+) but attenuated by DMSO. "
06/01/2000 - "Sulfhydryl alkylating agents, N-ethylmaleimide and p-chloromercuribenzoic acid, which have been found to presynaptically increase GABA release and have been suggested to have effects on proteins involved in transmitter release processes occurring in nerve terminals, occlude tetanus-induced potentiation of evoked and spontaneous IPSCs. "
|1.||Peptide Hydrolases (Proteases)
|2.||Edetic Acid (EDTA)
|4.||p-Chloromercuribenzoic Acid (PCMB)
|5.||Mercuric Chloride (Sublimate)
|7.||Dithionitrobenzoic Acid (Ellman's Reagent)
|8.||Adenosine Triphosphatases (ATPase)
|9.||Vasoactive Intestinal Peptide Receptors (Vasoactive Intestinal Peptide Receptor)
|10.||Vasoactive Intestinal Peptide (VIP (Vasoactive Intestinal Peptide))