|1.||Lardelli, Michael: 3 articles (10/2014 - 07/2011)|
|2.||Moussavi Nik, Seyyed Hani: 3 articles (10/2014 - 07/2011)|
|3.||Siniscalchi, Anna: 3 articles (08/2013 - 12/2006)|
|4.||Marino, Silvia: 3 articles (08/2013 - 12/2006)|
|5.||Voĭnikov, V K: 3 articles (04/2004 - 05/2001)|
|6.||Rusaleva, T M: 3 articles (04/2004 - 05/2001)|
|7.||Rachenko, E I: 3 articles (04/2004 - 05/2001)|
|8.||Varakina, N N: 3 articles (04/2004 - 05/2001)|
|9.||Rikhvanov, E G: 3 articles (04/2004 - 05/2001)|
|10.||Seo, Jung-Woo: 2 articles (01/2015 - 08/2013)|
|1.||Chromosome Aberrations (Chromosome Abnormalities)
07/01/2007 - "Sodium azide alone induces chromosomal aberrations significantly with increasing concentrations. "
11/01/1992 - "With this method sodium azide is not diluted in culture medium, but it is 'focused' into cells, producing chromosomal aberrations and other major genetic damages. "
08/01/1977 - "The low ability of NaN3 (compared to EMS) to induce mutant sectors may be related to the cells not being treated at the optimum time during the cell cycle, but it is more likely due to its low effectiveness for inducing chromosome aberrations."
07/01/2007 - "The study reveals that curcumin has antimutagenic potential against sodium azide induced chromosomal aberrations in Allium cepa root meristem cells. "
01/01/1988 - "Pretreatment of Chinese hamster ovary cells with proteases or with NaN3 leads to less chromosomal aberrations when the cells are posttreated with Alu I compared to the treatment of cells with Alu I alone. "
|3.||Bovine Brucellosis (Bang's Disease)
10/01/1995 - "Improved enzyme immunoassay (EIA) procedures achieved by incorporating sodium azide during predilution of serum samples in a solid-phase EIA for the detection of anti-Toxoplasma antibody in swine using a peroxidase conjugate and in all washes of a bovine brucellosis rapid card test EIA using alkaline phosphatase conjugate are reported. "
10/01/1995 - "Whereas sodium azide could be used only in sample predilution in the swine toxoplasmosis peroxidase-conjugate test, in the bovine brucellosis alkaline phosphatase-conjugate card test it could be used in all wash cycles. "
05/01/2006 - "Here we characterise a p44ERK-like protein in the rainbow trout cell line RTHDF and study the effect of (i) serum stimulation, (ii) sodium azide (chemical anoxia) and removal of azide (recovery) and (iii) anoxia (P(O)2<0.1%) and recovery. "
06/01/2014 - "In this article, we examine the utility of various assays of lipid peroxidation and more general assays of intracellular oxidative stress to detect the changes in oxidative stress in the brains of adult zebrafish exposed to hypoxia or explanted into a sodium azide solution for chemical mimicry of hypoxia. "
01/15/2013 - "Methane biogenesis during sodium azide-induced chemical hypoxia in rats."
07/01/2011 - "We report the identification by phylogenetic and conserved synteny analyses of an HMGA1 orthologue in zebrafish, hmga1 (hmg-i/y) and analysis of sodium azide as a chemical agent for inducing hypoxia-like responses in zebrafish embryos including temporary suspension of development ("suspended animation"). "
05/01/2009 - "Treatment with the respiratory inhibitor sodium azide produced an identical response to the hypoxia stress. "
|5.||Neurodegenerative Diseases (Neurodegenerative Disease)
05/11/2001 - "Therefore, chronic nonlethal sodium azide administration may provide a potential rat model for the study of mitochondrial dysfunction and the role of environmental pollutants in brain and muscle tissues affected in certain neurodegenerative diseases."
10/01/2000 - "To study the role of mitochondrial deficiency in the pathogenesis of neurodegenerative disease by investigating the energy metabolism in a sodium azide inhibited cytochrome-c oxidase SH-SY5Y Cell model. "
01/01/1999 - "Since energy-dependent secondary excitotoxic mechanisms also account for the pathogenesis of neurodegenerative diseases, a study was made of the effects of sodium azide on the immunohistochemical localization of kynurenine aminotransferase-I. After in vivo administration of sodium azide for five days, a markedly decreased glial kynurenine aminotransferase-I immunoreactivity was found by immunohistochemical techniques in the glial cells of the striatum, hippocampus, dentate gyrus and temporal cortex; at the same time, kynurenine aminotransferase-I started to be expressed by nerve cells which had not been immunoreactive previously. "
08/01/2013 - "Primary cortical neurons exposed to the mitochondrial toxin NaN3 (0.1-3 mM) were submitted to oxidative stress with H2O2 (30-150 μM), to mimic conditions observed in neurodegenerative disorders. "
05/11/2001 - "Effects of chronic sodium azide on brain and muscle cytochrome oxidase activity: a potential model to investigate environmental contributions to neurodegenerative diseases."
|5.||Adenosine Triphosphate (ATP)
|7.||Electron Transport Complex IV (Cytochrome c Oxidase)
|8.||Etoposide (VP 16)
|2.||Air Bags (Airbags)
|3.||Photochemotherapy (Photodynamic Therapy)