|1.||Lefkowitz, Robert J: 5 articles (03/2011 - 01/2004)|
|2.||Wei, Wei: 4 articles (11/2015 - 01/2011)|
|3.||Gurevich, E V: 4 articles (11/2011 - 03/2005)|
|4.||Gurevich, V V: 4 articles (11/2011 - 03/2005)|
|5.||Walker, Julia K L: 3 articles (01/2015 - 07/2008)|
|6.||Benovic, Jeffrey L: 3 articles (12/2014 - 03/2008)|
|7.||Pei, Gang: 3 articles (12/2013 - 02/2008)|
|8.||Benovic, J L: 3 articles (11/2011 - 03/2005)|
|9.||Theccanat, Tiju: 2 articles (12/2015 - 11/2014)|
|10.||Li, Jinju: 2 articles (12/2015 - 11/2014)|
01/01/2014 - "Interestingly, several recent studies have documented changes in expression and localisation of arrestins that occur during cancer progression and that correlate with clinical outcome. "
01/01/2014 - "Here, we discuss these advances and how changes in expression/localisation may affect functional outputs of arrestins in cancer biology. "
01/01/2014 - "Arrestins as regulatory hubs in cancer signalling pathways."
02/01/2013 - "In this article, we reviewed a comprehensive summary of the β-arrestins-mediated functions in human cancers."
02/01/2013 - "Because of the regulation and biological output of β-arrestins is so complex, the role of β-arrestins in cancer development still remains enigmatic. "
05/01/2013 - "In this study, the possible relationship between the expression of β-arrestins with the experimental hepatic fibrosis and the proliferation of hepatic stellate cells (HSCs) were investigated. "
12/01/2015 - "Targeted inhibition of β-arrestins in CFs might decrease oxidative stress as well as pathological cardiac fibrosis. "
12/01/2015 - "We hypothesize that β-arrestins regulate oxidative stress in a Nox4-dependent manner and increase fibrosis in HF. "
11/01/2015 - "In this review, we survey the involvement of β-arrestins in various signaling pathways and highlight different aspects of their regulation of fibrosis. "
11/01/2014 - "Targeted inhibition of β-arrestins in CF may represent a therapeutic strategy to prevent maladaptive myocardial fibrosis. "
|3.||Asthma (Bronchial Asthma)
01/01/2015 - "To assess the role of β-arrestins in regulating β2AR function in asthma, we and others have utilized β-arrestin-1 and -2 knockout mice. "
01/01/2014 - "This review will focus on the potential role of arrestins in those GPCR-mediated airway cell functions that are dysregulated in asthma. "
01/01/2014 - "GPCRs and arrestins in airways: implications for asthma."
01/01/2015 - "As β-arrestins are known to modulate β2AR function, our analysis of βAR subtype expression in β-arrestin knockout mice airways sets a reference point for future studies exploiting these knockout mice in various disease models including asthma. "
12/26/2014 - "A biased agonist that can selectively stimulate Gs signaling without promoting receptor interaction with G protein-coupled receptor kinases and β-arrestins should serve as an advantageous asthma therapeutic. "
01/01/2015 - "This study suggests that in the CLP-induced lung injury model, PHC could reduce pulmonary microvascular permeability by upregulating expressions of beta arrestins."
01/01/2015 - "This study is to investigate the effects of PHC on pulmonary microvascular permeability and on expressions of beta arrestins in lung injury induced by the cecal ligation and puncture (CLP) procedure. "
|2.||gamma-Aminobutyric Acid (GABA)
|3.||GTP-Binding Proteins (G-Protein)
|4.||Proteins (Proteins, Gene)
|6.||G-Protein-Coupled Receptor Kinases
|7.||G-Protein-Coupled Receptors (Receptors, G Protein Coupled)
|8.||Morphine (MS Contin)
|9.||Facilitative Glucose Transport Proteins (Glucose Transporter)