|1.||Lip, Gregory Y H: 19 articles (10/2013 - 06/2003)|
|2.||Molema, Grietje: 13 articles (12/2015 - 04/2002)|
|3.||Blann, Andrew D: 13 articles (08/2013 - 06/2003)|
|4.||King, Michael R: 12 articles (10/2015 - 04/2009)|
|5.||Blann, A D: 10 articles (03/2015 - 02/2000)|
|6.||Burdick, Monica M: 10 articles (01/2015 - 04/2003)|
|7.||Sato, Shinichi: 9 articles (12/2013 - 05/2003)|
|8.||Dimitroff, Charles J: 8 articles (01/2013 - 08/2004)|
|9.||Kvasnicka, J: 8 articles (01/2013 - 09/2001)|
|10.||Sackstein, Robert: 7 articles (05/2015 - 07/2005)|
06/01/1998 - "The mean serum ELAM-1 level in patients with metastatic tumors was significantly greater than that of patients with nonmetastatic tumors. "
08/01/2013 - "Importantly, survival was improved in MCC patients whose tumors had higher vascular E-selectin expression (P<0.05). "
08/10/2014 - "Our studies revealed that the CD31(+)E-selectin(+) population accounted for 20.8%, 26.4% and 29.9% of total endothelial cells respectively inside the femur of mice bearing early, middle and late stage metastatic MDA-MB-231 tumors. "
01/01/2014 - "A literature search in PubMed, Embase, Web of Science, Science Direct, SpringerLink, EBSCO, Wanfang, and Chinese National Knowledge Infrastructure databases was carried out to identify studies of the association between E-selectin S128R polymorphism and cancer risk. "
05/15/2012 - "Finally, immunohistological study indicated high E-selectin ligand expression on cancer cells undergoing EMT in vivo, supporting their coexistence observed in vitro. "
08/04/2000 - "beta(2)-Integrin blockade driven by E-selectin promoter prevents neutrophil sequestration and lung injury in mice."
01/01/2000 - "GSR, especially lung injury, was definitely exacerbated by the administration of anti-E-selectin antibody. "
05/01/1999 - "Insofar as an elevated level of E-selectin in peripheral blood reflects vascular endothelial activation, the data are consistent with the notion that pulmonary endothelial activation is required, in addition to granulocyte activation and an expanded PGP, for granulocyte migration into lung parenchyma and, therefore, for lung injury to occur."
03/01/1994 - "Lung injury was found to be L- and E-selectin-dependent. "
10/01/1991 - "Role of endothelial-leukocyte adhesion molecule 1 (ELAM-1) in neutrophil-mediated lung injury in rats."
|3.||Brain Ischemia (Cerebral Ischemia)
11/01/2012 - "E-selectin deficiency presents protective effect on cerebral ischemia. "
11/01/2012 - "To determine whether E-selectin deficiency can attenuate brain ischemia in a mouse model of focal cerebral ischemia. "
11/01/2012 - "E-selectin deficiency attenuates brain ischemia in mice."
07/11/2008 - "Human E-selectin, an endothelial adhesion molecule, is induced in the brain arteries by cerebral ischemia and participates in the infiltration of leukocytes that cause inflammatory reaction leading to brain damage. "
07/24/1999 - "We examined brain sections from P- and E-selectin-deficient mice (-/-) and their nontransgenic littermates (+/+) after focal cerebral ischemia and reperfusion (I/R) tissue injury. "
03/01/1994 - "Survival in lung reperfusion injury is improved by an antibody that binds and inhibits L- and E-selectin."
05/01/1995 - "The aim of our study was to examine the mechanism of E-selectin production and leukocyte accumulation in myocardial ischaemia-reperfusion injury. "
03/01/2004 - "Expression of E-selectin and its transcripts during intestinal ischemia-reperfusion injury in pigs."
08/01/1999 - "Amelioration of lung reperfusion injury by L- and E- selectin blockade."
08/01/1998 - "Hepatic ischemia-reperfusion injury causes E-selectin upregulation."
|5.||Brain Injuries (Brain Injury)
12/09/2003 - "Mucosal tolerance to E-selectin provides cell-mediated protection against ischemic brain injury."
06/01/2009 - "The aim of this work was to map E-selectin expression in a traumatic brain injury model using a newly-designed MR contrast agent. "
08/01/1996 - "The late appearance of E-selectin in nonischemic cerebral tissues suggests stimulation by transferable factors generated during brain injury."
03/01/2009 - "These studies suggest that E-selectin-specific Tregs can modulate the efficacy of neurogenesis after ischemia and promote repair after brain injury."
11/01/2010 - "E-selectin-specific Tregs can modulate the efficacy of adult neurogenesis after ischemia and promote repair after brain injury."
|1.||Intercellular Adhesion Molecule-1 (Intercellular Adhesion Molecule 1)
|2.||Vascular Cell Adhesion Molecule-1 (Vascular Cell Adhesion Molecule 1)
|3.||Immunoglobulin G (IgG)
|4.||Antigen-Antibody Complex (Immune Complex)
|5.||Tumor Necrosis Factor-alpha (Tumor Necrosis Factor)
|7.||Interleukin-8 (Interleukin 8)
|10.||von Willebrand Factor
|3.||Angioplasty (Angioplasty, Transluminal)
|4.||Homologous Transplantation (Allograft)