|1.||Song, Wen-Chao: 10 articles (01/2014 - 09/2002)|
|2.||Fujita, Teizo: 10 articles (11/2006 - 01/2002)|
|3.||Lin, Feng: 9 articles (04/2011 - 05/2002)|
|4.||Yamamoto, Kazuhide: 9 articles (01/2005 - 01/2002)|
|5.||Okada, Hiroyuki: 9 articles (01/2005 - 01/2002)|
|6.||Mizuno, Motowo: 9 articles (01/2005 - 01/2002)|
|7.||Medof, M Edward: 8 articles (06/2010 - 05/2002)|
|8.||Nasu, Junichirou: 8 articles (01/2005 - 08/2002)|
|9.||Shiratori, Yasushi: 8 articles (01/2005 - 01/2002)|
|10.||Miwa, Takashi: 7 articles (04/2013 - 09/2002)|
07/28/2011 - "Decay-accelerating factor regulates T-cell immunity in the context of inflammation by influencing costimulatory molecule expression on antigen-presenting cells."
05/12/2006 - "The role of decay-accelerating factor as a receptor for Helicobacter pylori and a mediator of gastric inflammation."
02/15/2005 - "Decay-accelerating factor (CD55) is expressed by neurons in response to chronic but not acute autoimmune central nervous system inflammation associated with complement activation."
08/01/2005 - "This study was undertaken to characterize the expression and regulation of decay-accelerating factor (DAF), a complement regulator, as a potential mediator of ovarian tissue protection from ovulatory inflammation. "
01/21/2000 - "Its regulation is needed to protect tissues from inflammation, but mice with a disrupted gene for the complement regulator decay accelerating factor were normal. "
|2.||Autoimmune Diseases (Autoimmune Disease)
01/01/2014 - "The role of decay accelerating factor in environmentally induced and idiopathic systemic autoimmune disease."
06/01/2007 - "Reduced expression of decay-accelerating factor 1 on CD4+ T cells in murine systemic autoimmune disease."
09/01/2002 - "Deletion of decay-accelerating factor (CD55) exacerbates autoimmune disease development in MRL/lpr mice."
04/01/2007 - "Decay-accelerating factor ameliorates systemic autoimmune disease in MRL/lpr mice via both complement-dependent and -independent mechanisms."
01/01/2006 - "Decay accelerating factor (DAF, CD55), a glycosylphosphatidylinositol-anchored membrane protein, is a candidate for autoimmune disease susceptibility based on its role in restricting complement activation and evidence that DAF expression modulates the phenotype of mice models for autoimmune disease. "
02/15/2004 - "Respective roles of decay-accelerating factor and CD59 in circumventing glomerular injury in acute nephrotoxic serum nephritis."
01/01/2014 - "Decay-accelerating factor 1 deficiency exacerbates leptospiral-induced murine chronic nephritis and renal fibrosis."
05/01/2002 - "Decay-accelerating factor confers protection against complement-mediated podocyte injury in acute nephrotoxic nephritis."
11/01/2010 - "The present studies were performed to examine the effects of murine cytomegalovirus (MCMV) infection on decay accelerating factor (DAF) and MCMVs role in exacerbating morbidity and mortality post-allo-BMT. "
04/01/2009 - "This study showed that coxsackievirus type B3 (CVB3) associated with decay-accelerating factor in non-polarized HeLa cells can be redirected into non-productive compartments by Arf6-dependent internalization, thus restricting infection. "
04/01/2015 - "In vitro, infection of polarized human intestinal epithelial cells by coxsackievirus B3 (CVB3) depends on virus interaction with decay-accelerating factor (DAF), a receptor expressed on the apical cell surface. "
04/01/2015 - "Expression of human decay-accelerating factor on intestinal epithelium of transgenic mice does not facilitate infection by the enteral route."
01/01/2012 - "A number of enteroviruses, including echoviruses (EV) and group B coxsackieviruses (CVB), initiate infection by attaching to decay-accelerating factor (DAF), a molecule that is highly expressed on the apical surface of polarized epithelial cells. "
|1.||Complement System Proteins (Complement)
|3.||Antigen-Antibody Complex (Immune Complex)
|7.||Proteins (Proteins, Gene)
|8.||Complement Receptors (Complement Receptor)
|9.||Complement C3 (C3 Complement)
|2.||Heterologous Transplantation (Xenotransplantation)
|4.||Blood Component Removal (Apheresis)