Excitatory Amino Acids

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1106 relevant articles (23 outcomes, 110 trials/studies) found for this Bio-Agent

Description: Endogenous amino acids released by neurons as excitatory neurotransmitters. Glutamic acid is the most common excitatory neurotransmitter in the brain. Aspartic acid has been regarded as an excitatory transmitter for many years, but the extent of its role as a transmitter is unclear.

Also Known As:

Excitatory Amino Acid; Acid, Excitatory Amino; Acids, Excitatory Amino; Amino Acid, Excitatory; Amino Acids, Excitatory

Relationship Network

Bio-Agent Context: Research Results

Amino Acids, Peptides, and Proteins

Amino Acids: 8786

Excitatory Amino Acids: 1106

Glutamic Acid: 7815

Aspartic Acid: 1892

AHN 649: 1

Experts

1.	Marion, Donald W: 3 articles (09/2003 - 05/2002)
2.	Wen, Z-H: 2 articles (06/2007 - 06/2007)
3.	Jean, Y-H: 2 articles (06/2007 - 06/2007)
4.	Ruitenbeek, Wim: 2 articles (07/2006 - 01/2006)
5.	Hopman, Jeroen: 2 articles (07/2006 - 01/2006)
6.	van de Bor, Margot: 2 articles (07/2006 - 01/2006)
7.	van Os, Sandra: 2 articles (07/2006 - 01/2006)
8.	Meador-Woodruff, James H: 2 articles (06/2006 - 03/2002)
9.	McCullumsmith, Robert E: 2 articles (06/2006 - 03/2002)
10.	Wong, Chih-Shung: 2 articles (05/2006 - 06/2002)

Related Diseases

1.	Ischemia 03/01/2005 - "This methodology provides the possibility of evaluating the protection against the outflow of excitatory amino acids and against an acute motor disturbance, i.e.contralateral turning to the ischemic side in the first hours after ischemia in awake rats. " 01/06/1992 - "GM1 ganglioside reduces ischemia-induced excitatory amino acid output: a microdialysis study in the gerbil hippocampus." 10/01/1991 - "Although considerable evidence supports a role for excitatory amino acids in the pathogenesis of ischemic neuronal injury, few in vivo studies have examined the effect of increasing durations of ischemia on the extracellular concentrations of these agents. " 03/01/2008 - "Treatment with RWPC completely prevented the burst of excitatory amino acids that occurred in response to ischemia in untreated rats and significantly reduced brain infarct volumes. " 04/01/2007 - "Transient ischemia-induced changes of excitatory amino acid carrier 1 in the ventral horn of the lumbar spinal cord in rabbits." Order ALL the reference details at left...
2.	Brain Ischemia (Cerebral Ischemia) 09/01/1991 - "Monosialoganglioside GM1 prevents excitatory amino acid (EAA)-related neuronal death in cultured central nervous system (CNS) neurons and reduces the severity of acute brain damage in different experimental models of cerebral ischemia. " 10/19/2001 - "This study suggested that altered brain energy metabolites and excitatory amino acids occurred during cerebral ischemia and and HBO regulated these striatal metabolites, which might contribute to the protective effect of HBO in cerebral ischemia." 04/01/1996 - "OBJECTIVE: To study the changes of excitatory amino acid (EAA) in patients with cerebral ischemia. " 09/01/1995 - "Recently, studies have demonstrated in vitro, that D-2 agonist, at the level of striatum and retina, may represent a powerful signal to inhibit release of excitatory amino acids implicated in cerebral ischemia. " 08/15/1994 - "Since ischemia brain damage is ascribed to an elevated level of extracellular excitatory amino acids (EAAs), this study was undertaken to investigate the effect of GCs on EAA homeostasis in hippocampal cell cultures during the insult of cyanide exposure. " Order ALL the reference details at left...
3.	Anoxia (Hypoxia) 07/01/2006 - "Excitatory amino acid release and electrocortical brain activity after hypoxemia in near-term lambs." 05/01/2004 - "Activation of ionotropic excitatory amino acid (EAA) receptors in pre-Bötzinger complex (pre-BötC) not only influences the eupneic pattern of phrenic motor output but also modifies hypoxia-induced gasping in vivo by increasing gasp frequency. " 08/01/2000 - "Taken together, these data suggest that EtOH enhances DA release in vivo during short-term hypoxia, possibly through mechanisms other than excitatory amino acid pathways." 01/01/1997 - "A novel in vitro cell culture model has been developed to investigate the mechanisms of delayed neuronal cell death following exposure to excitatory amino acids and hypoxia. " 07/01/1996 - "The cytotoxic effects of hypoxia and excitatory amino acids on cultured retinal ganglion cells were studied. " Order ALL the reference details at left...
4.	Brain Injuries (Brain Injury) 01/01/2004 - "OBJECTIVES: To assess systematically the efficacy of excitatory amino acid inhibitors on improving patient outcome following traumatic brain injury. " 01/01/2004 - "SELECTION CRITERIA: Trials were included if they were randomised, double-blind, controlled trials where excitatory amino acid inhibitors were administered to patients with traumatic brain injury, within 24 hours of sustaining that injury, and compared to a control group. " 03/01/2001 - "The aim of this study is to monitor excitatory amino acids (EAAs) in the extracellular fluids of the brain and to characterize regional neuronal damage in a new experimental model for brain injury, in which rabbits were exposed to 180-260 krad/s2 rotational head acceleration. " 01/01/1998 - "In previous studies, Katayama and our group have documented a massive increase in excitatory amino acid release following traumatic brain injury, in both rat fluid percussion, and humans [2,5]. " 01/01/1992 - "Recent studies suggest that excitatory amino acids (EAAs) may be important for the development of hypoxic-ischemic brain injury in the newborn. " Order ALL the reference details at left...
5.	Hypothermia 07/01/1996 - "By contrast, hypothermia reduced the release of all excitatory amino acids. " 05/01/1996 - "Effects of hypothermia on the rate of excitatory amino acid release after ischemic depolarization." 02/01/2002 - "BACKGROUND AND PURPOSE: The objective of this study was to assess the effect of therapeutic moderate hypothermia on excitatory amino acids and metabolism by applying cerebral microdialysis in patients suffering from space-occupying middle cerebral artery infarction. " 02/01/2002 - "Effects of hypothermia on excitatory amino acids and metabolism in stroke patients: a microdialysis study." 05/01/1996 - "BACKGROUND AND PURPOSE: Hypothermia slows the increase in extracellular excitatory amino acid (EAA) concentrations during temporary cerebral ischemia. " Order ALL the reference details at left...

Related Drugs and Biologics

1.	Glutamic Acid (Glutamate)
2.	Free Radicals
3.	N-Methylaspartate (NMDA)
4.	Excitatory Amino Acids
5.	AMPA Receptors (AMPA Receptor)
6.	Valproic Acid (Valproate, Semisodium)
7.	Aspartic Acid (Aspartate)
8.	Nitric Oxide (Nitrogen Monoxide)
9.	Dizocilpine Maleate (Dizocilpine)
10.	Glutamate Receptors (Glutamate Receptor)

Related Therapies and Procedures

1.	Analgesia
2.	Anesthesia
3.	Sutures (Suture)
4.	Cardiopulmonary Bypass (Heart-Lung Bypass)
5.	Injections

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