Arterivirus Infections

Infections caused by viruses of the genus ARTERIVIRUS.

Also Known As:

Infections, Arterivirus; Arterivirus Infection; Infection, Arterivirus

Networked: 10 relevant articles (0 outcomes, 1 trials/studies)

Disease Context: Research Results

Infections: 526564
- Virus Diseases: 41777
  - RNA Virus Infections: 303
    - Nidovirales Infections
      - Arterivirus Infections: 10
        Porcine Reproductive and Respiratory Syndrome: 3395

Related Diseases

1.	Persistent Infection

Experts

1.	Snijder, Eric J: 4 articles (10/2013 - 05/2008)
2.	Kikkert, Marjolein: 2 articles (10/2013 - 01/2012)
3.	Balasuriya, Udeni B R: 2 articles (05/2011 - 12/2004)
4.	Posthuma, Clara C: 2 articles (05/2011 - 05/2008)
5.	Zevenhoven-Dobbe, Jessika C: 2 articles (05/2011 - 05/2008)
6.	Cai, Xuehui: 1 article (12/2015)
7.	Hu, Liang: 1 article (12/2015)
8.	Huang, Li: 1 article (12/2015)
9.	Li, Jiangnan: 1 article (12/2015)
10.	Liu, Yuanyuan: 1 article (12/2015)

Drugs and Biologics

Drugs and Important Biological Agents (IBA) related to Arterivirus Infections:

1.	Proteins (Proteins, Gene)FDA Link 05/01/2011 - "These defects, which could be partially complemented in ORF5a-expressing cells, indicate that the novel protein, which may be the eighth structural protein of arteriviruses, is expressed and important for arterivirus infection." 05/01/2008 - "Previously, coexpression of two putative transmembrane nonstructural proteins (nsp2 and nsp3) was found to suffice to induce these remarkable membrane structures, which are typical of arterivirus infection. " 12/08/2004 - "Although arterivirus infections are strictly species-specific, the causative agents share many biological and molecular properties, including their virion morphology, replication strategy, unique properties of their structural proteins, and their ability to establish distinctive persistent infections in their natural hosts. " 12/15/2015 - "In this study, DDX19A, a member of the DEAD/H-box protein family, was identified as a novel component of NLRP3 inflammasome using arterivirus infection as a model. "
2.	InflammasomesIBA 12/15/2015 - "In this study, DDX19A, a member of the DEAD/H-box protein family, was identified as a novel component of NLRP3 inflammasome using arterivirus infection as a model. "
3.	Sialic Acid Binding Ig-like Lectin 1IBA 09/01/2007 - "IFN-alpha treatment enhances porcine Arterivirus infection of monocytes via upregulation of the porcine Arterivirus receptor sialoadhesin."
4.	Neutralizing AntibodiesIBA 12/19/2014 - "Further, we depict the location and characteristics of epitopes in the membrane proteins since the late appearance of neutralizing antibodies may lead to persistence, a characteristic hallmark of arterivirus infection. "
5.	Toll-Like Receptor 3IBA 09/01/2008 - "Toll-like receptor 3 activation decreases porcine arterivirus infection."
6.	Pattern Recognition ReceptorsIBA 01/01/2012 - "The potential relevance of RIG-I-like receptor (RLR) signaling for the innate immune response against arterivirus infection is supported by our finding that in mouse embryonic fibroblasts, the production of beta interferon primarily depends on the recognition of arterivirus RNA by the pattern-recognition receptor MDA5. "
7.	N-Acetylneuraminic Acid (Sialic Acid)IBA 08/01/2004 - "Porcine arterivirus infection of alveolar macrophages is mediated by sialic acid on the virus."
8.	Interferon-betaIBA 01/01/2012 - "The potential relevance of RIG-I-like receptor (RLR) signaling for the innate immune response against arterivirus infection is supported by our finding that in mouse embryonic fibroblasts, the production of beta interferon primarily depends on the recognition of arterivirus RNA by the pattern-recognition receptor MDA5. "
9.	VaccinesIBA 10/01/2013 - "We have also briefly reviewed the impact of these advances on disease management, the engineering of novel candidate live vaccines and the diagnosis of arterivirus infection. "
10.	RNA (Ribonucleic Acid)IBA 01/01/2012 - "The potential relevance of RIG-I-like receptor (RLR) signaling for the innate immune response against arterivirus infection is supported by our finding that in mouse embryonic fibroblasts, the production of beta interferon primarily depends on the recognition of arterivirus RNA by the pattern-recognition receptor MDA5. "