|1.||Haeggström, Jesper Z: 4 articles (02/2014 - 07/2008)|
|2.||Niegowski, Damian: 2 articles (02/2014 - 01/2014)|
|3.||Kleinschmidt, Thea: 2 articles (02/2014 - 01/2014)|
|4.||Ahmad, Shabbir: 2 articles (02/2014 - 01/2014)|
|5.||Rinaldo-Matthis, Agnes: 2 articles (02/2014 - 01/2014)|
|6.||Cho, Yong-Yeon: 2 articles (12/2010 - 07/2009)|
|7.||Pugliese, Angelo: 2 articles (12/2010 - 07/2009)|
|8.||Bode, Ann M: 2 articles (12/2010 - 07/2009)|
|9.||Jeong, Chul-Ho: 2 articles (12/2010 - 07/2009)|
|10.||Dong, Zigang: 2 articles (12/2010 - 07/2009)|
|1.||Asthma (Bronchial Asthma)
05/01/1998 - "Anti-leukotrienes: a new treatment for asthma."
03/01/1999 - "[The anti-leukotrienes: a new class of drugs to integrate into the therapeutic strategy of asthma]."
09/01/1997 - "[Anti-leukotrienes: a new perspective in treatment of bronchial asthma]."
09/01/1997 - "The leukotrienes, a group of important inflammatory mediators, cause vascular leakage and tissue edema; they also promote mucus secretion and a potent bronchoconstriction in patients with asthma. "
02/21/2014 - "Leukotriene (LT) C4 synthase (LTC4S) catalyzes the conjugation of the fatty acid LTA4 with the tripeptide GSH to produce LTC4, the parent compound of the cysteinyl leukotrienes, important mediators of asthma. "
03/01/1990 - "In the model of pulmonary leukostasis, no nonenzymatic LTA4-derivatives were detected, coincident with a predominance of cysteinyl-LT. "
03/01/1990 - "Moreover, a threefold increase in the total amount of LTA4-derived products was noted in the model of leukostasis, paralleled by a marked decrease in 5-HETE liberation. "
|3.||Nephrotic Syndrome (Syndrome, Nephrotic)
05/01/2010 - "In this study, we used a leukotriene A(4) hydrolase (LTA(4)H) inhibitor as a pharmacological tool to directly examine the role of LTB(4) in a mast cell-dependent murine model of allergic airway inflammation. "
04/19/2012 - "Because platelet adherence to leukocytes allows for both firm adhesion to endothelial cells and augmented transcellular conversion of leukotrienes, a disturbance in platelet-leukocyte interactions may be partly responsible for the respiratory tissue inflammation and the overproduction of cysLTs that characterize AERD."
05/01/2010 - "Leukotriene A(4) hydrolase inhibition attenuates allergic airway inflammation and hyperresponsiveness."
05/25/2001 - "Neutrophils, endothelial cells, and cysteinyl leukotrienes: a new approach to neutrophil-dependent inflammation?"
02/01/2001 - "Crystal structure of human leukotriene A(4) hydrolase, a bifunctional enzyme in inflammation."
|1.||Arachidonate 5-Lipoxygenase (5 Lipoxygenase)
|2.||Hydroxyeicosatetraenoic Acids (HETE)
|3.||leukotriene A4 hydrolase
|10.||Glutathione (Reduced Glutathione)