|1.||Kurohane, Kohta: 8 articles (11/2015 - 01/2007)|
|2.||Imai, Yasuyuki: 8 articles (11/2015 - 01/2007)|
|3.||Low, Philip S: 6 articles (08/2014 - 01/2006)|
|4.||Baker, James R: 5 articles (04/2011 - 01/2005)|
|5.||Li, Ying: 4 articles (04/2014 - 01/2010)|
|6.||Zhang, Sheng-Hua: 4 articles (02/2014 - 07/2009)|
|7.||Zhang, Lin: 4 articles (12/2010 - 07/2003)|
|8.||Matsuda, Tomoko: 4 articles (06/2010 - 01/2007)|
|9.||Bacon, Kevin B: 4 articles (01/2009 - 05/2004)|
|10.||Moore, Bethany B: 4 articles (12/2007 - 04/2004)|
09/01/1989 - "After determination of baseline data, the muscle was made ischemic for 2 hr by clamping its vascular pedicle, and subsequently reperfused for 2 hr. Ischemia-reperfusion produced a marked increase in clearance of FITC-Dx 150. "
07/01/1995 - "In saline-treated animals, ischemia-reperfusion increased the clearance of FITC-Dx 150 from 1.8 +/- 0.3 to 9.7 +/- 1.0 microliters/15 min/g by the end of the reperfusion period. "
07/01/1995 - "After determination of baseline data, the muscle was made ischemic for 4 hr by clamping the vascular pedicle and subsequently reperfused for 2 hr. In control animals not subjected to ischemia, clearance of FITC-Dx 150 remained constant throughout the experimental 7-hr period. "
05/01/1987 - "Clearance of FITC-Dx 150 increased from a control value (mean +/- SE) of 8.3 +/- 2.7 to 29.9 +/- 8.1 microliters/min/g after reperfusion following a 30-min period of ischemia, and from a control value of 36.2 +/- 13.6 to 274 +/- 94.5 after 2 hr of ischemia. "
02/20/2013 - "Transcardial perfusion of FITC-labeled albumin suggested that local ischemia was at least in part the cause of this myelination failure. "
09/01/2006 - "The cleared tumors were imaged in three modes (i) optical-CT to image the 3D distribution of microvasculature as indicated by absorbing dye, (ii) optical-ECT using the FITC excitation and emission filter set, to determine microvasculature as indicated by lectin-endothelial binding, and (iii) optical-ECT using the DSRed2 filter set to determine the 3D distribution of viable tumor as indicated by RFP emission. "
01/01/2015 - "Fluorescence microscopic studies indicated that the FITC-labeled K4R2-Nal2-S1 preferentially binds cancer cells and causes apoptotic cell death. "
04/01/2010 - "In vivo imaging study showed that FITC-labeled Ec-LDP-Hr was targeted and accumulated in the tumors. "
11/03/2015 - "Meta analyses revealed free intraperitoneal tumor cells (FITC) to be associated with poor overall survival in univariate (HR 3.27; 95% CI 2.82 - 3.78]) and multivariate (HR 2.45; 95% CI 2.04 - 2.94) analysis and poor peritoneal recurrence free survival in univariate (4.15; 95% CI 3.10 - 5.57) and multivariate (3.09; 95% CI 2.02 - 4.71) analysis. "
04/01/2015 - "Altogether, the FITC-D tumor to normal tissue ratio was significantly higher in the 10 J/cm(2) group compared others. "
01/01/2016 - "Synthetic BK or mBK labeled with FITC at the N-terminus was employed for discriminating between inflamed and normal cells; this method was found to be effective for detection of inflammation in live cells. "
01/01/2015 - "The present study evaluated different methods to induce gut inflammation in poultry based on FITC-d leakage. "
06/01/2015 - "In conclusion, FR, DSS, and RBD affected GIT tight junction integrity, suggesting their value for enteric inflammation models, and FITC-d may be a good indicator of permeability."
06/01/2015 - "All inflammation treatments significantly increased serum FITC-d levels at both doses. "
06/01/2015 - "Dose titration of FITC-D for optimal measurement of enteric inflammation in broiler chicks."
07/01/2015 - "Intact cells (Annexin-5-FITC(-)/7-AAD(-)), cells with early signs of apoptosis (Annexin-5-FITC(+)/7-AAD(-)), cells with late signs of apoptosis and partially necrotic cells (Annexin-5-FITC(+)/7-AAD(+)), as well as cells with early signs of necrosis (Annexin-5-FITC(-)/7-AAD(+)) were differentiated by fl ow cytometry. "
02/01/2006 - "Flow cytometric analysis of annexinV-FITC/PI-stained cells showed time-related increase in apoptosis and post-apoptotic necrosis. "
03/01/2004 - "The quantity of FITC+/PI- cells corresponded to early apoptosis, while that of FITC+/PI+ cells corresponded to late apoptosis or secondary necrosis. "
02/01/2002 - "Both apoptosis and necrosis of human fetal bone marrow CD34(+) hematopoietic stem cells cultured under MH were significantly decreased as detected by MTT and flow cytometry, with three-color labeled by PE-CD(34) (+)/ FITC-AnnexinV /7AAD. "
06/24/2015 - "Annexin-V FITC and PI staining experiments confirmed that the cell inhibition was primarily due to apoptosis and not by necrosis, which was also supported by LDH release assay experiment. "
|5.||Breast Neoplasms (Breast Cancer)
03/01/2013 - "Labeling signals of QD525 in both fixed and living breast cancer cells and tissue preparations were found to be significantly higher than those of FITC at 460-495 nm excitation wavelengths. "
03/01/2011 - "Furthermore, we verified the uptake of FITC-loaded OBs by the MCF-7 breast cancer cell line. "
09/01/2009 - "Purified DNA repair enzyme, OGG1, and nuclear lysates from MCF-7 breast cancer cells were incubated with double-stranded FITC-labeled wt and 8-oxo-G oligonucleotide to demonstrate the OGG1 incision assay. "
03/01/2006 - "We show that there is a partial correlation between autofluorescence and total Her-2/neu/FITC fluorescence in individual cells in the three breast cancer cell lines. "
08/15/2004 - "In contrast, transfection of DU145 cells or the T47D and MCF-7 breast cancer cell lines to express CD44s increased cell surface binding of FITC-HA and cell adherence to hBMECs. "
|7.||Fibrinogen (Factor I)
|10.||Inosine Triphosphate (ITP)
|2.||Heterologous Transplantation (Xenotransplantation)
|3.||Drug Therapy (Chemotherapy)
|4.||Quantum Dots (Quantum Dot)
|5.||Photochemotherapy (Photodynamic Therapy)