|1.||Malim, Michael H: 5 articles (11/2006 - 08/2002)|
|2.||Sheehy, Ann M: 4 articles (11/2004 - 08/2002)|
|3.||Gaddis, Nathan C: 3 articles (11/2004 - 08/2002)|
|4.||Friedler, Assaf: 2 articles (08/2010 - 09/2002)|
|5.||Li, Peng: 2 articles (10/2006 - 10/2004)|
|6.||Bishop, Kate N: 2 articles (11/2004 - 08/2004)|
|7.||Law, Emily K: 1 article (11/2014)|
|8.||Harris, Reuben S: 1 article (11/2014)|
|9.||Brown, William L: 1 article (11/2014)|
|10.||Reilly, Cavan: 1 article (11/2014)|
11/01/2004 - "Additional virus replication studies also revealed that the Vif protein of SIVtan is, in fact, active in human T cells, as it substantially enhanced the replication of its cognate virus and human immunodeficiency virus type 1. In sum, we now consider it improbable that species-specific restrictions to SIV Vif function can explain the lack of human infection with certain SIVs. "
08/01/2010 - "The HIV-1 Vif protein (192 residues) is required for HIV-1 infection of many target cells. "
11/01/2006 - "Wild type viral infections are largely spared from APOBEC function through the action of the viral Vif protein. "
01/01/2006 - "Probably, the deamination reaction that is inhibited in the wild type HIV-1 by the Vif protein, occurs rarely during infection and thus increases viral diversity and drug's resistance. "
10/25/2004 - "In cells infected by HIV-1 mutants lacking a functional Vif protein, APOBEC3G is specifically packaged into progeny virions and then interferes with the process of virus infection. "
09/01/2002 - "This study demonstrates that (i) purified recombinant Vif protein and HIV-1 but not avian sarcoma leukaemia virus protease specifically bind each other and (ii) the interaction between these two proteins takes place at the N terminus of the protease (aa 1-9) and the central part of Vif (aa 78-98). "
|3.||Acquired Immunodeficiency Syndrome (AIDS)
06/01/2012 - "In this work, we analyzed the APOBEC3-mediated editing in proviral HIV-1 from perinatally infected children (n=93) in order to explore its association with polymorphisms of APOBEC3G and CUL5 genes (APOBEC3G H186R, APOBEC3G C40693T, and CUL5 SNP6), the Vif protein variability, and also the time to AIDS development. "
07/01/2006 - "We suggest that an artificial dHIV construct carrying a mutated vif gene (coding for a Vif protein unable to block APOBEC3G/F) could have a therapeutic effect as well in HIV infected individuals and AIDS patients."
|4.||HIV Infections (HIV Infection)
10/01/2006 - "Although these host factors are present in diverse mammalian species and have been shown to act against various organisms, their importance in HIV infection has been highlighted because of their suggested activities against HIV in vivo and the strong conservation of the HIV vif gene encoding the Vif protein capable of countering this innate activity. "
|1.||Proteins (Proteins, Gene)
|2.||RNA-Directed DNA Polymerase (Reverse Transcriptase)
|3.||DNA (Deoxyribonucleic Acid)