|1.||Tiwari, Vaibhav: 3 articles (01/2013 - 08/2008)|
|2.||Shukla, Deepak: 3 articles (01/2013 - 08/2008)|
|3.||Tan, Guang-Hong: 2 articles (07/2013 - 03/2012)|
|4.||Wang, Hua: 2 articles (07/2013 - 09/2012)|
|5.||Mei, Wen-Li: 2 articles (07/2013 - 03/2012)|
|6.||Li, Yue-Nan: 2 articles (07/2013 - 03/2012)|
|7.||Dai, Hao-Fu: 2 articles (07/2013 - 03/2012)|
|8.||Huang, Feng-Ying: 2 articles (07/2013 - 03/2012)|
|9.||Debeir, Olivier: 2 articles (01/2010 - 02/2006)|
|10.||Kiss, Robert: 2 articles (01/2010 - 02/2006)|
01/01/2015 - "aureus by suppressing the phagocytic activity of macrophages with cytochalasin D before infection was investigated. "
03/01/2014 - "Disruption of actin filaments by cell treatment with cytochalasin D resulted in a successful second infection. "
01/01/2013 - "The levels of viral infection and filopodia induction were reduced with the actin polymerization inhibitors, Cytochalasin-D and Lantriculin B, suggesting an important role for actin reorganization during ZF-3-OST-3 mediated HSV-1 entry. "
02/01/2010 - "Here, we challenge this hypothesis by quantifying Encephalitozoon cuniculi infection rates of J774 cells that were incubated with the phagocytosis inhibitor cytochalasin D. "
05/01/2009 - "Cytochalasin D treatment significantly reduced the rate of nuclear infection. "
|2.||Anaphylaxis (Anaphylactic Shock)
08/01/1997 - "Our studies provide evidence that cytochalasin D will be beneficial in the treatment of anaphylaxis."
08/01/1997 - "We investigated the effect of cytochalasin D on anaphylaxis. "
08/01/1997 - "Effect of cytochalasin D on systemic and local anaphylaxis in a murine model."
07/01/1995 - "Experimental data not only showed that anaphylactic reaction can be suppressed in vivo by blocking cytoskeletal activity but also demonstrated different pharmacodynamic characteristics of cytochalasin D on clinical course, gravity and outcome of anaphylactic shock in sensitized guinea pigs, depending on the interval between administration of the drug and allergen."
07/01/1995 - "Suppression of anaphylactic shock in sensitized guinea pigs by cytochalasin D."
10/01/2001 - "Although perivascular infusion of cytochalasin D inhibited collar-induced SMC migration from media to intima in vivo as well, the intimal hyperplasia was not reduced due to concomitant development of an inflammatory response."
10/01/2001 - "Therefore, we investigated whether cytochalasin D, a fungal metabolite that inhibits actin filament formation, suppressed SMC migration and collar-induced intimal hyperplasia in the rabbit carotid artery. "
10/01/2001 - "Effects of local cytochalasin D delivery on smooth muscle cell migration and on collar-induced intimal hyperplasia in the rabbit carotid artery."
02/01/2006 - "Effects of cytochalasin D-eluting stents on intimal hyperplasia in a porcine coronary artery model."
12/01/2003 - "Local cytochalasin D delivery from this stent-platform significantly inhibits neointimal hyperplasia in a porcine coronary model."
|4.||Pemphigus (Pemphigus Vulgaris)
09/01/2010 - "Therefore, we investigated whether pharmacological manipulation of actin polymerization modulates pathogenic effects of PV-IgG. Pharmacological stabilization of actin filaments via jasplakinolide significantly blocked cell dissociation and Dsg3 fragmentation, whereas cytochalasin D-induced actin depolymerization strongly enhanced pathogenic effects of PV-IgG. To substantiate these findings, we studied whether the protective effects of Rho GTPases, which are potent regulators of the actin cytoskeleton and were shown to be involved in pemphigus pathogenesis, were dependent on modulation of actin dynamics. "
12/01/2014 - "Our data shows that cytochalasin D compromises the spreading area of single glioma cells, eventually decreasing their 'inside-out' contractile forces, and 'outside-in' force response to external strain. "
12/01/2014 - "Using a highly aggressive glioma model (DBTRG-05MG), we present a platform technology to quantify single cell force modulation (both inside-out and outside-in) with and without the presence of a cytoskeleton altering drug (cytochalasin D) using suspended and aligned fiber networks (nanonets) beginning to represent the aligned glioma environment. "
07/14/2004 - "At the distal end of C6 glioma cell processes, GLT-1 clusters undergo rapid morphological changes in both shape and size, and these changes are inhibited by cytochalasin D treatment, suggesting that the morphogenesis of GLT-1 clusters is highly dependent on the actin network. "
08/16/2002 - "Cytochalasin D and latrunculin A also diminished extracellular Ca2+ influx in unstimulated glioma C6 cells previously incubated in Ca2+ free buffer. "
04/24/1997 - "In the present study, cytochalasin D (CD) and calmodulin antagonists were used to examine the influence of F-actin and calmodulin on membrane interaction of MARCKS in C6 glioma cells. "
|3.||rho GTP-Binding Proteins (rho GTP-Binding Protein)
|5.||Immunoglobulin G (IgG)
|7.||Actins (F Actin)
|10.||latrunculin A (LAT-A)