|1.||Moens, Ugo: 1 article (03/2014)|
|2.||Huseby, Nils-Erik: 1 article (03/2014)|
|3.||Ravuri, Chandra: 1 article (03/2014)|
|4.||Pandur, Seila: 1 article (03/2014)|
|5.||Popov, Violeta B: 1 article (11/2013)|
|6.||Spiegel, David A: 1 article (11/2013)|
|7.||Kim, Taehan: 1 article (11/2013)|
|8.||Zhang, Dongyan: 1 article (11/2013)|
|9.||Zhang, Xian-Man: 1 article (11/2013)|
|10.||Lee, Hui-Young: 1 article (11/2013)|
06/01/1961 - "Evaluation of some uncoupling agents in the chemotherapy of experimental cancer: a study of their distribution in tumour-bearing rats."
04/01/1962 - "IX. The effects of uncoupling agents on the oxidation of fatty acids by transplantable tumors."
06/01/1961 - "The effects of uncoupling agents on the uptake and incorporation of glycine by transplantable tumors."
04/01/1983 - "Results obtained demonstrate that (a) the apparent Km and Vmax values for adenosine diphosphate and adenosine triphosphate uptake are significantly lower in hepatoma compared to normal or regenerating liver mitochondria, (b) the Vmax values for adenosine diphosphate uptake correlate with tumor growth rate, and (c) the Km values for adenosine triphosphate in both hepatoma and normal mitochondria are lowered in the presence of added uncoupling agents; however, the extent of decrease is much less in fast-growing tumors than in slow-growing tumors and normal tissues. "
|2.||Myocardial Ischemia (Ischemic Heart Diseases)
04/01/2011 - "The purpose of this study was to investigate the roles of loss of oxidative phosphorylation and activation of the mitochondrial ATP-sensitive K+ channel and permeability transition pore in VF development during myocardial ischemia by using mitochondrial uncoupling agents (carbonyl cyanide m-chlorophenylhydrazone and 2,4-dinitrophenol) and channel blockers (5-hydroxydecanoate and cyclosporine A) at concentrations that have been demonstrated to block the intended targets selectively. "
09/01/1981 - "Analyses show that ATP limitations are probably not involved in the elicitation of lysis by ionophores, uncoupling agents or starvation. "
03/01/1977 - "The addition of inhibitors of protein synthesis (trichodermin, verrucarin) or uncoupling agents (2,4-dinitrophenol, sodium azide) at the beginning of starvation results in an increased rate of development of resistance. "
01/01/1990 - "No significant changes were found in animal resistance to hemic and tissue hypoxia and to oxidative phosphorylation uncoupling agents. "
01/01/1990 - "Resistance of mice to altitude, gas, hemic, tissue, mixed or hyperoxis hypoxia, to oxidative phosphorylation uncoupling agents and to X-ray irradiation during 2 days after exposure to severe acute hypobaric hypoxia was investigated. "
05/01/1992 - "Blockade of electron transport (CN-, anoxia, rotenone) and uncoupling agents (e.g. "
|1.||2,4-Dinitrophenol (2,4 Dinitrophenol)
|2.||Adenosine Triphosphate (ATP)
|6.||Reactive Oxygen Species (Oxygen Radicals)
|1.||Drug Therapy (Chemotherapy)