|1.||Yeramian, Andree: 2 articles (01/2014 - 02/2008)|
|2.||Dolcet, Xavier: 2 articles (01/2014 - 02/2008)|
|3.||Matias-Guiu, Xavier: 2 articles (01/2014 - 02/2008)|
|4.||Dong, Zheng: 2 articles (08/2013 - 06/2011)|
|5.||Kondrikov, Dmitry: 2 articles (08/2013 - 06/2011)|
|6.||Su, Yunchao: 2 articles (08/2013 - 06/2011)|
|7.||Tsutsui, H: 2 articles (12/2001 - 01/2001)|
|8.||Takeshita, A: 2 articles (12/2001 - 01/2001)|
|9.||Shimokawa, H: 2 articles (12/2001 - 01/2001)|
|10.||Egashira, K: 2 articles (12/2001 - 01/2001)|
07/01/1991 - "Tiron (sodium 4,5-dihydroxybenzene-1,3-disulfonate), a chelating agent used in the treatment of experimental metal poisoning, was evaluated for developmental toxicity in pregnant Swiss mice. "
07/01/1991 - "The results of this study show that Tiron has potential beneficial effects in the treatment of repeated intraperitoneal vanadium poisoning, while the effectiveness of DFOA is very uncertain. "
08/01/1993 - "Tiron (sodium 4,5-dihydroxybenzene-1,3-disulfonate), a chelating agent widely used in analytical chemistry, is an effective antidote in the treatment of oral or parenteral vanadate poisoning. "
05/01/1991 - "Tiron (sodium 4,5-dihydroxybenzene-1,3-disulphonate) has been suggested as a possible antidote for acute uranium poisoning. "
04/01/1989 - "The results show that Tiron was consistently the most effective chelator of those tested in the treatment of uranium poisoning after repeated daily administration of the metal."
12/01/2001 - "Intracoronary infusion of a cell-permeable OFR scavenger, tiron, suppressed OFR formation and improved the vasodilating capacity to adenosine or brief ischemia in HF dogs but not in controls. "
07/05/2013 - "Tempol (10 and 100 µM) and tiron (100 and 500 µM) were perfused 15 min before ischemia and during reperfusion. "
07/05/2013 - "Cardiac levels of oxidative stress markers such as O2(-) and malondialdehyde were notably increased during ischemia and following reperfusion, which were suppressed by the administration of tempol or tiron. "
05/01/2005 - "The constriction during reperfusion after 45 min of ischemia is supposedly caused by the inhibition of Akt-mediated eNOS-Ser1177 phosphorylation, which was suppressed by a PKC inhibitor chelerythrine, or ROS scavengers N-2-mercaptopropionyl glycine (MPG) and 4,5-Dihydroxy-1, 3-benzenedisulfonic acid disodium salt (Tiron). "
09/01/2004 - "A second series of hearts was treated with the antioxidants tiron (10 mM) and N-acetylcysteine (4 mM) before 20 min of global ischemia without reperfusion. "
|4.||Diabetic Nephropathies (Diabetic Nephropathy)
01/01/2010 - "In the present study, we used streptozotocin-induced diabetic rats (STZ rats) to examine the effects of insulin, phloridzin or antioxidant, tiron on diabetic nephropathy. "
08/01/2009 - "In this study, we used streptozotocin-induced diabetic rats with diabetic nephropathy and treated them with insulin, phloridzin, or antioxidant tiron. "
08/01/1991 - "Furthermore, the increase in cerebral prostanoid synthesis in response to hypercapnia was not affected by pretreatment with SOD, Tiron, catalase, DMSO, or MPG. "
02/01/1993 - "The present study found that treatment with SOD/catalase or Tiron did not prevent loss of vasodilation to hypercapnia or the loss of hypercapnia-induced cerebral 6-keto-prostaglandin F1 alpha synthesis after cerebral ischemia. "
08/01/1991 - "Neither pretreatment with the extracellular superoxide anion radical scavenger, superoxide dismutase (SOD), the intracellular superoxide anion radical scavenger, Tiron, the H2O2 scavenger, catalase, nor hydroxyl radical scavengers, dimethyl sulfoxide (DMSO) and MPG, altered vasodilation of pial arteries or arterioles in response to hypercapnia. "
|3.||Pentetic Acid (DTPA)
|10.||6- hydroxy- 2,5,7,8- tetramethylchroman- 2- carboxylic acid (Trolox)