|1.||Traber, Daniel L: 72 articles (04/2014 - 09/2002)|
|2.||Enkhbaatar, Perenlei: 66 articles (05/2014 - 03/2003)|
|3.||Traber, Lillian D: 56 articles (04/2014 - 09/2002)|
|4.||Cox, Robert A: 52 articles (05/2014 - 09/2002)|
|5.||Rahman, Irfan: 45 articles (11/2015 - 08/2002)|
|6.||Herndon, David N: 44 articles (05/2014 - 09/2002)|
|7.||Hawkins, Hal K: 42 articles (05/2014 - 09/2002)|
|8.||Pinkerton, Kent E: 41 articles (01/2015 - 01/2002)|
|9.||Churg, Andrew: 33 articles (12/2014 - 05/2002)|
|10.||Wright, Joanne L: 32 articles (12/2014 - 05/2002)|
|1.||Chronic Obstructive Pulmonary Disease (COPD)
06/01/2014 - "Strong smoke-free public policies may provide protection against COPD hospitalizations, particularly after 12 months, with the potential to save lives and decrease health care costs."
01/01/2013 - "Because of a significant reduction of SIRT1 in rodent lungs exposed to cigarette smoke and in lungs of patients with chronic obstructive pulmonary disease (COPD), activation of SIRT1 may be a potential target for chronic obstructive pulmonary disease therapy. "
09/28/2009 - "To conclude, a more adequate sidestream cigarette smoke rat COPD model was established, which will be beneficial for understanding the pathogenesis of the disease and for evaluation of drug effectiveness."
07/15/2014 - "Tobacco smoke exposure, the major cause of chronic obstructive pulmonary disease (COPD), instigates a dysfunctional clearance of thick obstructive mucus. "
03/01/2008 - "Public health measures, such as effective smoking prevention and cessation programs, reduction of air pollution and exposure to biomass smoke, and improved access to health care, would help reduce the burden of COPD among Hispanics in the United States and Latin America."
01/01/2008 - "Direct comparison between animals injured by 40% TBSA burn and 2 x 30 sec smoke exposure and sacrificed after 48 h showed significantly greater abnormality in the C57BL/6 mice. "
07/01/2006 - "PaCO2 was significantly greater in VCMV group at 36, 48, and 72 hours after smoke and burn injury. "
07/01/2006 - "Survival 84 hours after smoke and burn injury was significantly greater in the HFPV (7/7, 100%) compared with the VCMV group (3/6, 50%, P < .05). "
09/01/2003 - "The mean degree of bronchial, bronchiolar, and terminal bronchiolar obstruction was significantly greater in animals with smoke injury alone or combined smoke inhalation and burn (S+B) injury, compared with animals with burn injury alone or uninjured animals (P < 0.05). "
01/01/1989 - "Burn patients exhibited a significant reduction in serum Cu, although concomitant smoke inhalation increased serum Cu back to low-normal levels. "
06/01/2010 - "To determine whether DAP12 plays a role in cigarette smoke-induced pulmonary inflammation, we exposed wild-type and DAP12-deficient mice to chronic cigarette smoke and found significant reduction in recruitment of alveolar macrophages in DAP12-deficient mice. "
08/15/2015 - "The aim of this study was to investigate the protective effects of Sal B on cigarette smoke (CS)-induced acute lung inflammation. "
05/01/2014 - "We recently demonstrated the protective effects of NF-E2-related factor (Nrf) 2 against influenza virus (FluV)-induced pulmonary inflammation in mice exposed to cigarette smoke (CS). "
10/01/2005 - "This study shows for the first time that a PDE4 inhibitor partially ameliorates lung inflammation and fully prevents parenchymal destruction induced by cigarette smoke."
05/01/2011 - "In the present study, we investigated the role of Nrf2 in protection against influenza virus-induced pulmonary inflammation after cigarette smoke exposure with both in vitro and in vivo approaches. "
|4.||Asthma (Bronchial Asthma)
01/01/2014 - "One study did not aim to reduce children's tobacco smoke exposure, but rather aimed to reduce symptoms of asthma, and found a significant reduction in symptoms in the group exposed to motivational interviewing. "
09/01/2010 - "ETS exposure was associated with a significant increase in nasal wash neutrophils (beta coefficient = 0.73 [95% confidence interval, CI: 0.11 to 1.35]; p = .023) and a significant decrease in ACT score (-5.17 [-8.42 to -1.93]; p = .003) within Cluster 1 and a significant reduction in the bronchodilator-induced % change in forced expiratory volume in one second (FEV(1)) (-36.32 [-62.18 to -10.46]; p = .009) within Cluster 3. Clustering techniques defined more homogeneous subgroups, allowing for the detection of otherwise undetectable associations between environmental tobacco smoke exposure and asthma characteristics."
04/01/2003 - "To reduce the burden of asthma in Georgia, exposure of people with asthma to tobacco smoke in the home should be eliminated and training in asthma management should be more widely available."
08/01/2015 - "Comprehensive statewide smoke-free indoor air laws appear to be effective in reducing SHS exposure and improving asthma outcomes. "
10/01/2013 - "These findings suggest that avoiding or limiting exposure to traffic pollution and wood smoke may help to reduce asthma. "
01/01/2016 - "In this study, we investigated protective effects of EC-18 (a synthetic copy of PLAG) on inflammatory responses using a cigarette smoke with lipopolysaccharide (LPS)-induced airway inflammation model. "
01/01/2013 - "Colonic inflammation in mice is improved by cigarette smoke through iNKT cells recruitment."
11/01/2010 - "Accelerated clearance of live NTHi from cigarette smoke-exposed mice was independent of the establishment of chronic inflammation or direct toxic effects of cigarette smoke components on bacteria. "
07/15/2015 - "The aim of the study was to investigate the time-dependent effects of cigarette smoke exposure on exercise capacity, markers of systemic inflammation, and skeletal muscle structure. "
06/01/2015 - "The present study aimed to observe the level of inflammation and the number of lesions in the airways and parenchyma of mouse lungs subsequent to smoking cessation following 4 weeks exposure to cigarette smoke. "
|3.||Pentetic Acid (DTPA)
|4.||Biological Markers (Surrogate Marker)
|5.||Tumor Necrosis Factor-alpha (Tumor Necrosis Factor)
|6.||Adrenal Cortex Hormones (Corticosteroids)
|9.||Technetium (Technetium 99m)
|3.||Tissue Therapy (Cell Therapy)
|4.||Drug Therapy (Chemotherapy)
|5.||Transplantation (Transplant Recipients)