|1.||Krautgartner, W D: 2 articles (10/2005 - 02/2002)|
|2.||Yamauchi, Yoshiko: 2 articles (10/2005 - 11/2003)|
|3.||Hannig, M: 2 articles (10/2005 - 02/2002)|
|4.||Kawahara, Koichi: 2 articles (10/2005 - 11/2003)|
|5.||Vitkov, L: 2 articles (10/2005 - 02/2002)|
|6.||Iwata, Yuko: 1 article (09/2015)|
|7.||Dufour, Inès: 1 article (09/2015)|
|8.||Schakman, Olivier: 1 article (09/2015)|
|9.||Mondin, Ludivine: 1 article (09/2015)|
|10.||Zanou, Nadège: 1 article (09/2015)|
01/01/1978 - "Use of 1 mM ruthenium red selectively inhibited the tonic component of K-contracture and caused a marked decrease in cellular 45Ca uptake in that component of K-contracture. "
01/01/1989 - "High concentrations of ruthenium red (12.5 to 25 microM) resulted in the development of contracture. "
12/01/1988 - "The contracture induced by 12.5 microM ruthenium red was markedly inhibited when Ca2+ in the medium was lowered. "
12/01/1988 - "High concentrations (12.5-25.0 microM) of ruthenium red caused a sustained contracture. "
05/01/1985 - "Ruthenium red (10(-4) M) does not inhibit the Ca readmission contracture but reduces the release of cellular material and the gain of Ca and Na. "
01/01/2010 - "Ruthenium red, an inhibitor of intracellular Ca(2+) release, repressed induction of OsHREF1-4 under hypoxia. "
05/01/1990 - "When present throughout hypoxia and reoxygenation, 1.24 microM ruthenium red prevented the decrease in coronary flow normally seen and allowed recovery of heart rate, +dP/dT, -dP/dT and work (defined as the product of developed pressure and heart rate) to normal levels. "
01/01/1984 - "Illumination of white-eyed Musca photoreceptors following hypoxia or the application of ruthenium red (RR, a known blocker of Ca2+ uptake into intracellular organelles) induced a transient after depolarization (TA). "
05/01/2006 - "Compared with the hypoxia/reoxygenation (H/R) group, ruthenium red (RR, 5 micromol/L), given at the on set of reoxygenation, significantly improved the contractile function of left ventricle, decreased the myocardial infarct size, alleviated the production of ROS in myocardial mitochondria and LDH release in coronary effluent. "
08/01/1998 - "We investigated the pathways involved in the hypoxia induced changes in [Ca2+]m by using known inhibitors of mitochondrial Ca2+ transport, namely ruthenium red, an inhibitor of the Ca2+ uniporter (the normal influx route) and clonazepam, an inhibitor of Na+/Ca2+ exchange, (the normal efflux route). "
04/01/1991 - "Postischaemic reperfusion injury in the isolated rat heart: effect of ruthenium red."
01/01/1989 - "Reperfusion injury in ischemic myocardium: protective effects of ruthenium red and of nitroprusside."
04/01/1991 - "To examine the role of mitochondrial calcium uptake in mediating ischaemic and reperfusion injury, isolated rat hearts were perfused with ruthenium red (n = 6), a polysaccharide dye which inhibits calcium uptake by mitochondria, and were compared to control perfused hearts (n = 7). "
01/01/1989 - "Ruthenium red is an inorganic dye with calcium flux-inhibiting properties which protects ischemic myocardium against reperfusion damage, as judged by biochemical indices of mitochondrial function. "
|5.||Hypotension (Low Blood Pressure)
04/17/2007 - "Both antagonists ruthenium red and AM281 eliminated post-anandamide apnoea and hypotension but had no effect on post-apnoeic depression of V(T). "
12/01/1993 - "In addition, the afferent function of the sensory nerves are not totally blocked by ruthenium red as capsaicin elicits the reflex hypotension in the presence of this blocker of sensory nerve efferent function."
|2.||L-Lactate Dehydrogenase (Lactate Dehydrogenase)
|3.||Calcitonin Gene-Related Peptide
|4.||Nitroprusside (Sodium Nitroprusside)
|7.||Coloring Agents (Dyes)
|1.||Mechanical Ventilators (Ventilator)
|3.||Transplantation (Transplant Recipients)