|1.||Urrutia, Raul: 3 articles (09/2003 - 05/2002)|
|2.||Ellenrieder, Volker: 2 articles (09/2003 - 05/2002)|
|3.||Zhang, Jin-San: 2 articles (07/2003 - 05/2002)|
|4.||Liu, Li: 1 article (08/2015)|
|5.||Li, Biaoru: 1 article (08/2015)|
|6.||Pace, Betty S: 1 article (08/2015)|
|7.||Makala, Levi H: 1 article (08/2015)|
|8.||Matsuda, Tadashi: 1 article (05/2015)|
|9.||Kon, Shigeyuki: 1 article (05/2015)|
|10.||Kitai, Yuichi: 1 article (05/2015)|
11/01/2008 - "Our studies suggest that, in the majority of cells, CMV establishes a true latent infection, defined as the lack of expression of genes associated with productive infection, and that this occurs through changes in histone modifications and recruitment of transcriptional silencing factors to the MIEP."
03/01/2010 - "Overexpression of some of these components or the inactivation of initiation repressor proteins (4E-BP1) has been implicated in cancer development including oral carcinogenesis by affecting cell survival, angiogenesis, and tumor growth and invasion. "
07/01/2004 - "In particular, the fact that human diseases, such as Rett and ICF syndromes as well as some human forms of cancer, are connected with the activities of human repressor proteins indicates that transcriptional repression and gene silencing is essential for maintenance of the cellular integrity of a multicellular organism. "
05/29/2015 - "The promyelocytic leukemia protein PML acts as a tumor suppressor by forming transcription-regulatory complexes with a variety of repressor proteins. "
09/01/2011 - "Heat-shock protein 90 (Hsp90) inhibitor downregulates c-Myc expression and upregulates the expression of tumor repressor proteins such as p53 and pRB, inhibiting the G1/S transition and causing G2/M arrest during cell cycle progression. "
07/01/2003 - "Further elucidation of the hTERT transcriptional regulation, including identification and characterization of the endogenous repressor proteins, should lead to better understanding of the complex regulation of human telomerase in normal and cancer cells and may open up new strategies for anticancer therapy."
|3.||Contagious Ecthyma (Orf)
09/01/1998 - "Upstream of the sitABC operon and divergently transcribed lies a 645-bp open reading frame (ORF), which codes for a polypeptide of approximately 25 kDa with homology to the DtxR family of metal-dependent repressor proteins. "
02/01/1997 - "The discovery of a second integrase gene (intB), a gene with similarity to bacteriophage repressor proteins (regA), and a gene similar to an ORF from a conjugative transposon (gepA), suggests that a second genetic element, either a bacteriophage or a conjugative transposon, is integrated next to vap region 3 in the D. "
04/01/1991 - "The ORF contains an amino-terminal helix-turn-helix motif, characteristic of DNA-binding proteins and has similarity to known repressor proteins. "
09/01/1989 - "In addition, trans-activator and repressor proteins encoded respectively by the full-length and carboxy-terminal E2 ORF regulate the level of expression of other BPV-1 genes. "
|4.||Sudden Infant Death (SID)
09/15/2003 - "These results demonstrate that SID-containing KLF repressor proteins can inhibit cell growth and neoplastic transformation, and outline for the first time cellular and molecular mechanisms by which these functions may be achieved."
07/31/2003 - "This finding offers insights into a mechanism of cell growth regulation by interactions of different subtypes of SID-containing repressor proteins with Sin3. "
05/15/2002 - "We recently reported that a structurally and functionally related Mad1-like SID is also present in five Sp1-like repressor proteins (TIEG1, TIEG2, BTEB1, BTEB3 and BTEB4), demonstrating that SID-mSin3A interactions have a wider functional impact on transcriptional repression. "
08/01/2006 - "Genes induced by the orc2-1 mutation, starvation, or inactivation of repressor proteins, map near ORC-binding loci significantly more frequently compared to all genes. "
09/13/2002 - "They suggest the possibility that tagged repressor molecules, produced by stalled ribosomes, can be inducers of transposition under starvation conditions."
|2.||Neisseria PorB protein (MIEP)
|3.||Transcription Factors (Transcription Factor)
|4.||Pattern Recognition Receptors
|5.||Vascular Endothelial Growth Factor A (Vascular Endothelial Growth Factor)
|8.||Telomerase (Telomerase Reverse Transcriptase)
|10.||Trans-Activators (Trans-Acting Factor)
|1.||Transplantation (Transplant Recipients)