|1.||Oksal, Ayşegül: 1 article (08/2008)|
|2.||Bek, Kenan: 1 article (08/2008)|
|3.||Demircin, Gülay: 1 article (08/2008)|
|4.||Ezgü, Fatih Süheyl: 1 article (08/2008)|
|5.||Oner, Ayşe: 1 article (08/2008)|
|6.||Delibaş, Ali: 1 article (08/2008)|
|7.||Greenberg, Cynthia C: 1 article (05/2006)|
|8.||Aiston, Susan: 1 article (05/2006)|
|9.||Hampson, Laura J: 1 article (05/2006)|
|10.||Brady, Matthew J: 1 article (05/2006)|
03/01/2000 - "During the last 14 min of exercise, glycogenolysis was greater in hypoxia despite a lower mole fraction of phosphorylase a. "
12/01/1992 - "Hypoxia causes sustained glycogenolysis in skeletal muscle without an increase in percentage of phosphorylase a. "
12/01/1992 - "Hypoxia causes glycogenolysis without an increase in percent phosphorylase a in rat skeletal muscle."
02/15/1990 - "Hence the Km of phosphorylase a for its substrate Pi must be considerably higher than the concentrations that occur in the cytosol, even during hypoxia."
12/01/1988 - "3. We conclude that the hepatic glycogenolysis induced by anoxia and by KCN is solely mediated by phosphorylase a. "
08/15/2002 - "PP2A activity as measured with phosphorylase a as substrate was slightly inhibited after 30 min ischemia followed by 30 min reperfusion, and this inhibition correlated with an increased S6K1 and ERK1/2 phosphorylation. "
12/01/2000 - "Protein phosphatase activity, as measured with [(32)P]phosphorylase a as a substrate, diminished during ischemia and returned to control levels upon 30-min recovery. "
07/01/1996 - "However, the ratio phosphorylase a/a + b was not altered, whatever the conditions, (5) the concentration of inorganic phosphate (Pi) increased sharply during the first minutes of ischemia, to values above 8-10 mM, under all conditions studied. "
01/01/1989 - "To investigate the role of the intracellular calcium ion in the development of acute gastric mucosal lesions, phosphorylase a activity was measured as an index of the intracellular calcium ion concentration ([Ca2+]i), using Lowry's method, in the rat ischemia-reperfusion model. "
01/01/1983 - "In contrast to epinephrine infusion prolonged ischemia resulted in a decrease in the mole fraction of phosphorylase a and simultaneously in an increase of the glycogenolytic rate. "
|5.||Glycogen Storage Disease (Glycogenosis)
08/01/2008 - "Enzymatic analysis of the muscle biopsy showed a phosphorylase A level of 129 nmol/s/mg protein (normal: 200-600) and a phosphorylase A+B level of 385 nmol/s/mg protein (normal: 500-1500), which was compatible with glycogenosis type V. As McArdle disease rarely becomes symptomatic and ARF secondary to this condition is very rare, our case represents a rare clinical presentation."
01/01/1988 - "We determined glycogen concentration and phosphorylase 'a+b' and phosphorylase a activities in platelets, mononuclear and polymorphonuclear cells from control subjects and patients with phosphorylase kinase deficiency (glycogen storage disease IX) and liver phosphorylase deficiency (glycogen storage disease VI). "
08/01/1986 - "The behavior of hepatic phosphorylase b kinase, phosphorylase a and b after administration of glucagon to patients with glycogen storage disease type VIa."
08/01/1986 - "In the patients with glycogen storage disease (GSD) type VIa and different serum glucose response to glucagon, the activities of hepatic phosphorylase b kinase, phosphorylase a and b were estimated before and after the intravenous administration of glucagon. "
05/01/1990 - "The activities of enzyme in the pathway of glycogen and glycogenosis (alpha-glucosidase, amylo-1,6-glucosidase, phosphorylase a, phosphorylase kinase, phosphofructokinase, etc.) were within normal limits. "
|3.||Glycogen Synthase (Synthase I)
|5.||Glycogen Phosphorylase (Phosphorylase, Glycogen)
|6.||Phosphorylase Kinase (Phosphorylase Kinase, Glycogen)
|8.||Adenosine Triphosphate (ATP)