|1.||Königsrainer, Ingmar: 5 articles (08/2013 - 01/2008)|
|2.||Zieker, Derek: 5 articles (08/2013 - 01/2008)|
|3.||Northoff, Hinnak: 5 articles (08/2013 - 01/2008)|
|4.||Königsrainer, Alfred: 5 articles (08/2013 - 01/2008)|
|5.||Bühler, Sarah: 4 articles (08/2013 - 01/2010)|
|6.||Mishra, Suresh: 3 articles (02/2015 - 10/2003)|
|7.||Glatzle, Jörg: 3 articles (08/2013 - 01/2010)|
|8.||Zhang, Wei: 3 articles (01/2013 - 12/2007)|
|9.||Brücher, Björn L D M: 3 articles (03/2010 - 01/2008)|
|10.||Nieselt, Kay: 3 articles (03/2010 - 01/2008)|
03/15/2011 - "Recently, we reported a new mutation of phosphoglycerate kinase (PGK), called PGK-Barcelona, which causes chronic hemolytic anemia associated with progressive neurological impairment. "
03/15/2011 - "Enzymatic and metabolic characterization of the phosphoglycerate kinase deficiency associated with chronic hemolytic anemia caused by the PGK-Barcelona mutation."
01/01/1998 - "A phosphoglycerate kinase mutant (PGK Herlev; D285V) in a Danish patient with isolated chronic hemolytic anemia: mechanism of mutation and structure-function relationships."
12/01/1980 - "A phosphoglycerate kinase variant, PGK Uppsala, associated with hemolytic anemia."
09/01/1980 - "Molecular abnormality of phosphoglycerate kinase-Uppsala associated with chronic nonspherocytic hemolytic anemia."
06/01/1993 - "We have studied methylation patterns at the DXS255 locus and the phosphoglycerate kinase (PGK) gene in 25 females in remission from lymphoma and compared them to 35 normal females. "
11/01/1984 - "The clonal origin of radiation-induced thymic lymphoma was studied in mice with cellular mosaicism for phosphoglycerate kinase (PGK). "
01/01/2013 - "The clinicopathologic characteristics, immunophenotype, EBV in situ hybridization, and T cell receptor (TCR) gene rearrangement of primary pulmonary NK cell lymphoma from a 73-year-old Chinese woman were investigated and the clonal status was determined using female X-chromosomal inactivation mosaicism and polymorphisms at the phosphoglycerate kinase (PGK) gene. "
09/28/2005 - "Overexpression of PrPC by injection of rAd (replication-defective recombinant adenoviral)-PGK (phosphoglycerate kinase)-PrPC-Flag into ischemic rat brain improved neurological behavior and reduced the volume of cerebral infarction, which is supportive of a role for PrPC in the neuroprotective adaptive cellular response to ischemic lesions. "
11/15/1980 - "Results of studies with the X-chromosome-linked enzyme phosphoglycerate kinase strongly suggest that these tumors had a clonal origin. "
12/01/2012 - "Subsequent investigations indicated that MVIH could activate tumor-inducing angiogenesis by inhibiting the secretion of phosphoglycerate kinase 1 (PGK1). "
11/01/2010 - "Nucleotide-binding domain of phosphoglycerate kinase 1 reduces tumor growth by suppressing COX-2 expression."
01/01/2010 - "Phosphoglycerate kinase 1 (PGK1) elicited greater reactivity in 20.9% (10 in 48) of the samples in the cancer group, while no outlier was present in the non-cancer groups."
04/01/2004 - "Blood level of phosphoglycerate kinase does not correlate with presence or extent of tumor."
|5.||Myeloproliferative Disorders (Myeloproliferative Disorder)
02/01/1994 - "The restriction fragment length polymorphisms (RFLP) of the X-chromosome phosphoglycerate kinase (PGK) and hypoxanthine phosphoribosyltransferase (HPRT) genes were used to study the clonal basis of the chronic myeloproliferative disorders (CMPD). "
04/01/1990 - "Restriction fragment length polymorphisms of the X-chromosome genes phosphoglycerate kinase and hypoxanthine phosphoribosyl transferase were used to study clonality in peripheral blood leukocytes from 48 women with chronic myeloproliferative disorders (c-MPD). "
06/01/1989 - "Using hypoxanthine phosphoribosyl transferase and phosphoglycerate kinase probes, we have demonstrated monoclonality of peripheral blood leukocytes in three females with myeloproliferative disorders who had uninformative chromosomal analysis. "
01/01/1991 - "Three Down's syndrome patients with transient myeloproliferative disorder were studied for clonality of the proliferating blast cells using the X chromosome-linked polymorphic gene phosphoglycerate kinase, immunoglobulin heavy chain (IgH) gene and T-cell antigen receptor (TCR) (beta, gamma, delta) genes. "
|2.||Triose-Phosphate Isomerase (Triosephosphate Isomerase)
|3.||Glucose-6-Phosphate Isomerase (Phosphoglucose Isomerase)
|6.||Hypoxanthine Phosphoribosyltransferase (Hypoxanthine Guanine Phosphoribosyltransferase)
|8.||Androgen Receptors (Androgen Receptor)
|9.||Vascular Endothelial Growth Factor A (Vascular Endothelial Growth Factor)
|10.||Messenger RNA (mRNA)