|1.||Worgall, Stefan: 2 articles (12/2015 - 05/2013)|
|2.||Worgall, Tilla S: 2 articles (12/2015 - 05/2013)|
|3.||Holloway, Graham P: 2 articles (08/2015 - 06/2014)|
|4.||Spiekerkoetter, Ute: 2 articles (12/2014 - 01/2014)|
|5.||Herebian, Diran: 2 articles (12/2014 - 01/2014)|
|6.||Jiang, Xian-Cheng: 2 articles (05/2013 - 03/2005)|
|7.||Krab, Klaas: 2 articles (01/2013 - 04/2005)|
|8.||Ono, Jennie G: 1 article (12/2015)|
|9.||Allison, Mary K: 1 article (08/2015)|
|10.||Herbst, Eric A F: 1 article (08/2015)|
10/15/1985 - "Peroxisomal palmitoyl-CoA oxidation (nmol/min per mg of peroxisomal protein) was also increased in livers of adult obese mice and did not change with starvation. "
06/15/1983 - "The effects of 3 or 6 days of starvation on hepatic peroxisomal palmitoyl-CoA oxidation were examined in adult lean and obese Zucker rats. "
07/01/2008 - "After 48 h starvation, there is: (a) a significant increase in K(i) and decrease in hepatic malonyl-CoA content; (b) a decreased K(m) for palmitoyl-CoA; and (c) increased catalytic activity (V(max)) and CPT-I protein abundance that is significantly greater in contact sites compared with outer membranes. "
02/01/1988 - "Moreover, starvation and diabetes had the same effects on enzyme activity, affinity for palmitoyl-CoA and malonyl-CoA sensitivity of CPT I in isolated cells as found in isolated mitochondria. "
10/01/1986 - "Starvation of young rats did not change mitochondrial oxidation rates, although riboflavin-deficient starved rats exhibited increased rates of palmitoyl-CoA oxidation as well as increased CPT-A Vmax. "
04/15/1983 - "Peroxisomal and mitochondrial palmitoyl coenzyme A beta-oxidation activities in rat white adipose tissue and their regulation in hypothyroidism."
10/01/1983 - "Dixonplot studies indicated that the AdNT in hypothyroidism was two- to threefold more sensitive to atractylate and palmitoyl-CoA inhibitions. "
|3.||Heart Diseases (Heart Disease)
01/01/2014 - "This study demonstrates significant toxicity of especially those fatty acid species in vitro that significantly accumulate in fatty acid oxidation defects presenting with cardiac disease such as very long-chain acyl-CoA dehydrogenase, carnitine acylcarnitine translocase and carnitine palmitoyl-CoA transferase deficiencies. "
01/01/2013 - "Subsequently, we simulated the condition of obesity by increasing the palmitoyl-CoA concentration. "
03/01/2008 - "We postulate that covalent modification and perhaps inhibition of various mitochondrial enzymes by palmitoyl-CoA could lead to the metabolic impairments found in obesity-related diseases."
04/01/2005 - "The effects of palmitoyl-CoA in our model system support the proposed mechanism linking obesity and type 2 diabetes through an effect on adenine nucleotide translocator."
01/01/1980 - "In this latter, the palmitoyl-CoA monooleinacyltransferase enzyme probalby plays a key role and appears a major target of the overall anti-obesity of fenfluramine."
|1.||Malonyl Coenzyme A (Malonyl CoA)
|5.||Long-Chain Acyl-CoA Dehydrogenase (Long-Chain-Acyl-Coenzyme A Dehydrogenase)
|8.||Carnitine Acyltransferases (Carnitine-Acylcarnitine Translocase)