|1.||de Carvalho, José Gilberto Barbosa: 1 article (07/2012)|
|2.||Venditti, Marco Antonio Campana: 1 article (07/2012)|
|3.||Contó, Marcos Brandão: 1 article (07/2012)|
|4.||Boldyrev, A: 1 article (11/2000)|
|5.||Dobrota, D: 1 article (11/2000)|
|6.||Stvolinsky, S: 1 article (11/2000)|
|7.||Mezesova, V: 1 article (11/2000)|
|8.||Kukley, M: 1 article (11/2000)|
|1.||Hypertension (High Blood Pressure)
11/30/1983 - "4-nitrophenyl phosphatase activity of the red blood cell membrane in essential hypertension."
01/01/1990 - "This ultracytochemical study was undertaken to determine whether increased arteriolar permeability in acute hypertension is accompanied by altered localisation of the ouabain-sensitive, K(+)-dependent p-nitrophenyl-phosphatase (K(+)-NPPase), a component of the Na+, K(+)-ATPase system. "
07/01/2012 - "Considering the putative participation of N-methyl-D-aspartate (NMDA) receptors and the Na(+), K(+)-ATPase enzymes in the susceptibility to convulsions induced by the benzodiazepine inverse agonist methyl 6,7-dimethoxy-4-ethyl-β-carboline-3-carboxylate (DMCM), the present study sought to determine if rats with high (HTR) and low (LTR) thresholds to clonic convulsions induced by DMCM differed in the following aspects: the binding of NMDA receptors by [(3)H]-MK-801, Na(+), K(+)-ATPase activity (K(+)-stimulated p-nitrophenylphosphatase) and high-affinity [(3)H]-ouabain binding to membranes from discrete brain regions. "
11/01/2000 - "Rat brain subjected to 45-min global ischemia is characterized by decreased activity of K-p-nitrophenyl phosphatase and monoamine oxidase B and a disordering of the membrane bilayer by reactive oxygen species attack, the latter being monitored by the fluorescence of the membrane fluorescent probe, 1-anilino, 8-naphtalene sulphonate (ANS). "
06/01/1989 - "Rabbit ventricle was made ischemic by incubation in hypoxic, glucose-free Tyrode solution at 37 degrees C for 60-120 min. Ischemia inhibited Na+-K+-ATPase and K+-p-nitrophenylphosphatase (PNPPase) activity in the adult myocardium more than in the newborn. "
|4.||Ehrlich Tumor Carcinoma
07/01/1984 - "The presence of a soluble, Mg2+- or Mn2+-dependent p-nitrophenylphosphatase activity in Ehrlich ascites tumor cell homogenates is reported. "
07/01/1984 - "Mg2+- or Mn2+-dependent p-nitrophenylphosphatase activity is present in Ehrlich ascites tumor cells."
03/01/1978 - "Further investigations on the p-nitrophenylphosphatase activity of intact Ehrlich ascites tumor cells."
03/01/1978 - "The substrate specificity and the effects of nucleotides and SH-blocking agents on the p-nitrophenylphosphatase activity of intact Ehrlich ascites tumor cells (EAT) cells were studied. "
07/01/1984 - "Ehrlich ascites tumor cell p-nitrophenylphosphatase is shown to be sensitive to inactivation by trypsin, N-ethylmaleimide, or heat treatments."
|5.||Limb-Girdle Muscular Dystrophies (Limb-Girdle Muscular Dystrophy)
12/01/1996 - "The allosteric behaviour of the p-nitrophenyl-phosphatase (E.C.126.96.36.199.) from membrane erythrocytes was investigated in the following multisystemic diseases: myotonic dystrophy, limb-girdle muscular dystrophy, Charcot-Marie-Tooth and juvenile spinal muscular atrophy; in myotonia congenita, which is not a multisystemic disease, and in healthy controls. "
|1.||Adenosine Triphosphatases (ATPase)
|3.||Ouabain (G Strophanthin)
|5.||Dizocilpine Maleate (Dizocilpine)
|7.||N-Methyl-D-Aspartate Receptors (NMDA Receptors)
|9.||methyl 6,7- dimethoxy- 4- ethyl- beta- carboline- 3- carboxylate