|1.||Guan, Yun-Feng: 2 articles (07/2015 - 02/2011)|
|2.||Miao, Chao-Yu: 2 articles (07/2015 - 02/2011)|
|3.||Wang, Pei: 2 articles (07/2015 - 02/2011)|
|4.||Zhai, R Grace: 2 articles (04/2013 - 01/2012)|
|5.||Li, Zhi-Yong: 1 article (07/2015)|
|6.||Li, Guo-Qiang: 1 article (07/2015)|
|7.||Zhou, Can-Can: 1 article (07/2015)|
|8.||Zhao, Yan: 1 article (07/2015)|
|9.||Zhou, Xiao-Ming: 1 article (07/2015)|
|10.||Lu, Y-B: 1 article (04/2015)|
07/01/2015 - "A delayed nicotinamide mononucleotide administration for 7 days with the first dose at 12 hours post middle cerebral artery occlusion did not protect acute brain infarction and neuronal deficit; however, it still improved postischemic regenerative neurogenesis. "
02/01/2011 - "Nampt inhibition by a highly-specific Nampt inhibitor, FK866, aggravated brain infarction in experimentally cerebral ischemia rats, whereas Nampt overexpression in local brain and Nampt enzymatic product nicotinamide mononucleotide (NMN) reduced ischemia-induced cerebral injuries. "
|2.||Middle Cerebral Artery Infarction (Middle Cerebral Artery Syndrome)
01/15/2012 - "Here, we use a Drosophila model of frontotemporal dementia with parkinsonism linked to chromosome 17 (FTDP-17), in order to study the neuroprotective capacity of a recently identified neuronal maintenance factor, nicotinamide mononucleotide (NAD) adenylyl transferase (NMNAT), a protein that has both NAD synthase and chaperone function. "
08/01/2014 - "Expressing the slow Wallerian degeneration protein (Wld(S)) or related nicotinamide mononucleotide adenyltransferases (NmNATs) protects axons against damage from a broad range of insults, but the ability of these proteins to protect against inhibition of axonal transport has received little attention. "
02/23/2009 - "Slow Wallerian degeneration (Wld(S)) encodes a chimeric Ube4b/nicotinamide mononucleotide adenylyl transferase 1 (Nmnat1) fusion protein that potently suppresses Wallerian degeneration, but the mechanistic action of Wld(S) remains controversial. "
02/08/2007 - "The chimeric Wld(s) protein consisting of the N-terminal 70 amino acids of Ufd2 and the complete sequence of nicotinamide mononucleotide adenylyltransferase1 (Nmnat1), delays Wallerian degeneration in Wld(s) mice. "
02/15/2006 - "Wallerian degeneration of injured neuronal axons and synapses is blocked in Wld(S) mutant mice by expression of an nicotinamide mononucleotide adenylyl transferase 1 (Nmnat-1)/truncated-Ube4b chimeric gene. "
|3.||Staphylococcal Protein A (A, Protein)
|6.||Proteins (Proteins, Gene)
|9.||Niacin (Nicotinic Acid)