|1.||Rajini, Padmanabhan Sharda: 3 articles (01/2015 - 03/2012)|
|2.||Ru, Shaoguo: 3 articles (01/2013 - 12/2003)|
|3.||Oommen, Anna: 3 articles (10/2012 - 01/2006)|
|4.||Kiran, Ravi: 3 articles (01/2011 - 03/2004)|
|5.||Sandhir, Rajat: 3 articles (01/2011 - 03/2004)|
|6.||Joshi, Apurva Kumar Ramesh: 2 articles (01/2015 - 03/2012)|
|7.||Nagaraju, Raju: 2 articles (01/2015 - 03/2012)|
|8.||Wang, Wei: 2 articles (01/2013 - 09/2012)|
|9.||Tian, Hua: 2 articles (01/2013 - 09/2012)|
|10.||Kazi, Amajad Iqbal: 2 articles (10/2012 - 03/2012)|
01/01/2012 - "Our results clearly demonstrate the involvement of AChE inhibition in hyperglycemia and stressogenic effects of monocrotophos in rats following acute exposure. "
01/01/2012 - "Oral administration of monocrotophos (1.8 mg/kg b.w., 1/10 LD(50)) caused reversible hyperglycemia in rats with peak increase occurring at 2 h following administration. "
03/29/2012 - "Further, hydrazine sulfate (a gluconeogenesis inhibitor) abolished hyperglycemia in monocrotophos-treated rats. "
03/29/2012 - "Hyperglycemia and hyperlactacidemia induced by monocrotophos were abolished by pre-treatment with atropine. "
03/29/2012 - "Pattern of changes in plasma lactate suggests that β-adrenergic activation mediates monocrotophos-induced hyperlactacidemia, while α-adrenergic receptor mediates lactate utilization, leading to hyperglycemia. "
01/01/1994 - "However, the fact remains, that only a small number of reliably reported poisoning cases are attributable to monocrotophos products, and the reports confirm the efficacy of basic medical attention to poisoning. "
01/01/2006 - "In this study rats were subjected to increasing doses (0.25 to 0.8 LD(50)) of monocrotophos, and cell membrane lipid profiles, particularly those of myofibril membranes, were examined in relation to neuromuscular weakness occurring in poisoning. "
01/01/2015 - "Monocrotophos, implicated in about 1/4th of organophosphate poisonings in our centre, is associated with the highest mortality (24%). "
10/01/2012 - "Acute monocrotophos administration resulted in significant AChE inhibition (50-82%) in the rat brain regions 2.5 h after poisoning. "
09/01/2012 - "This report describes an episode of mass monocrotophos poisoning that occurred due to accidental ingestion of monocrotophos-contaminated millet (so-called bavta) flour involving eight severely poisoned persons. "
|3.||Body Weight (Weight, Body)
10/01/2010 - "A significant reduction (p < .05) was observed in the weight gained by rats while the liver-to-body weight ratio increased significantly (p < .05) as the level of exposure to monocrotophos increased. "
06/01/2012 - "Exposure of rats to monocrotophos (0.50 or 1.0 mg/kg body weight, p.o.) decreased body weight (10% and 30%) and impaired grip strength (28% and 32%) and learning ability (65% and 68%) at both the doses, respectively in comparison to controls. "
07/01/2002 - "There was no change organs weight except for a significant decrease in weight of the ovary with 3.3, 6.6, 10 and 13 mg/kg body weight/day and uterus and body weight with 10 and 13 mg/kg body weight/day monocrotophos treatment. "
07/01/2002 - "There were significant decrease in the small, medium, large and total number of healthy follicles and increase in the medium, large and total number of atretic follicles with 6.6, 10 and 13 mg/kg body weight/day monocrotophos treatment. "
07/01/2002 - "Estrous cycle was affected by showing a significant decrease in the number of estrous cycle and duration of proestrus, estrus and metestrus with concomitant significant increase in the duration of diestrus in all the treated groups, except with 1.6 mg/kg body weight/day monocrotophos treated group. "
11/09/2010 - "Monocrotophos toxicity and bioenergetics of muscle weakness in the rat."
03/01/2009 - "This study examined mitochondrial function and its modulation by nitric oxide in muscle weakness of rats exposed to an acute, oral (0.8LD(50)) dose of monocrotophos. "
11/09/2010 - "This study examined whether muscle weakness in rats subject to monocrotophos toxicity (0.8LD₅₀) was caused by inhibition of ATP synthesis, by oxidative phosphorylation and glycolysis, in addition to inhibition of muscle acetylcholinesterase. "
11/09/2010 - "Severe muscle weakness in rats following monocrotophos administration was associated with inhibition of muscle acetylcholinesterase (30-60%) but not with reduced ATP production. "
03/01/2009 - "Mitochondrial ATP synthase inhibition and nitric oxide are involved in muscle weakness that occurs in acute exposure of rats to monocrotophos."
|5.||Parkinson Disease (Parkinson's Disease)
12/01/2012 - "In the present study, Caenorhabditis elegans was selected as the model organism to evaluate the potential of monocrotophos (MCP), an OPI, to elicit dopaminergic features of Parkinson's disease in terms of dopamine content, basic movement and integrity of dopaminergic neurons along with its effect on acetylcholinesterase (AChE) activity and life span. "
|2.||Adrenergic Receptors (Adrenergic Receptor)
|5.||Acetylcholine (Acetylcholine Chloride)
|8.||Adenosine Triphosphate (ATP)
|9.||Messenger RNA (mRNA)
|10.||Nitric Oxide (Nitrogen Monoxide)