Insulin Antagonists

09/01/1956 - "Studies on humoral insulin antagonists in diabetic acidosis."

05/01/1992 - "In vivo insulin resistance may result from an increased level of circulating insulin antagonists."
04/01/2007 - "The development of insulin resistance may be associated not only with insulin production disorders or presence of insulin antagonists but also with modification of the number of receptors and sensitivity of peripheral tissues. "
01/01/1981 - "In general, the causes of insulin resistance can be placed into three categories: (1) abnormal beta cell secretory products, (2) circulating insulin antagonists, and (3) target tissue defects in insulin action. "
08/01/1958 - "Insulin antagonists, insulin antibodies and insulin resistance."
11/01/1995 - "In male type 1 diabetic patients: (1) acute hyperinsulinaemia decreases IGFBP-1, DHEAS and SHBG concentrations with the greatest decline in IGFBP-1, (2) SHBG concentration is positively associated with factors indicating good insulin sensitivity, (3) association between fuel homeostasis and SHBG, DHEAS and insulin antagonists suggests a network of these factors in the regulation of insulin action in type 1 diabetic patients."

01/10/1963 - "[Diabetes mellitus and insulin antagonists]."
09/01/1958 - "Circulating insulin antagonists in diabetes mellitus. "
11/01/1959 - "Circulating insulin antagonists in diabetes mellitus: untreated diabetics, diabetics during infections and acromegalics with diabetes."
01/01/1983 - "[Role of insulin antagonists in the pathogenesis of insulin--sensitive and insulin-resistant diabetes mellitus]."
09/01/1978 - "[Changes of insulin antagonists in juvenile diabetes mellitus--with special reference to growth hormone and glucagon]."

01/01/2005 - "Other treatments, such as renin-angiotensin-aldosterone system antagonists, insulin sensitisers and lifestyle-based interventions, have shown beneficial effects on vascular function in type 2 diabetes. "
09/20/1985 - "Characteristic of both obesity and non-insulin-dependent diabetes mellitus, insulin resistance is triggered at the level of the target tissue and can be induced by three general categories of causes: (1) an abnormal beta cell secretory product, (2) circulating insulin antagonists, or (3) a target tissue defect in insulin action. "

02/01/1994 - "The glucose intolerance is not the result of reduced insulin secretion, or circulating insulin antagonists, and does not correlate with the coexisting metabolic acidosis. "