|1.||Moursi, Mohammed M: 2 articles (01/2005 - 12/2004)|
|2.||Brown, Aliza T: 2 articles (01/2005 - 12/2004)|
|3.||Davis, Joseph A: 2 articles (01/2005 - 12/2004)|
|4.||Eidt, John F: 2 articles (01/2005 - 12/2004)|
|5.||Nogueira, Célia: 1 article (01/2015)|
|6.||Ferreira, Filipa: 1 article (01/2015)|
|7.||Couce, María L: 1 article (01/2015)|
|8.||Marcão, Ana: 1 article (01/2015)|
|9.||Vilarinho, Laura: 1 article (01/2015)|
|10.||Bóveda, M Dolores: 1 article (01/2015)|
|1.||Vascular Diseases (Vascular Disease)
03/01/2002 - "Homocystine solubility and vascular disease."
01/01/2003 - "For screening of Cardio-Vascular diseases by this method, 0.5mg of L-Homocystine as a control marker was found to be the most sensitive and reliable, compared with most effective amount of CRP, 0.5ng, for detecting early Cardio-Vascular problems due to various localized infections. "
01/01/2003 - "2 minute non-invasive screening for cardio-vascular diseases: relative limitation of C-Reactive Protein compared with more sensitive L-Homocystine as cardio-vascular risk factors; safe and effective treatment using the selective drug uptake enhancement method."
04/01/1995 - "Homocyst(e)ine [H(e)], the sum of homocysteine, homocystine, and the homocysteine-cysteine mixed disulfide, free and protein-bound, has been shown to be associated in retrospective case control studies, and in one prospective study, with vascular disease, including coronary artery disease (CAD), cerebrovascular disease, and peripheral vascular disease. "
07/01/2010 - "Homocystinuria is an inborn error of amino acid metabolism in which homocystine accumulates in the blood and produces a slowly evolving clinical syndrome. "
09/05/1997 - "The diagnosis of homocystinuria was delayed in each case due to difficulties with detection of small amounts of homocystine in physiologic samples. "
06/01/1982 - "These data extend the knowledge of homocystine metabolism and provide a rational basis for new approaches to the treatment of homocystinuria."
12/01/1972 - "Variability of the urinary excretion of homocystine and inorganic sulfate in a patient with homocystinuria."
04/25/1997 - "Therefore, methionine determination is much more effective than homocystine determination for newborn screening for homocystinuria. "
03/01/2009 - "Homocystine levels, polymorphisms and the risk of ischemic stroke in young Asian Indians."
10/01/2000 - "We found 21 studies searching Medline from 1966-July 1999 using the key words homocysteine, homocystine and cerebrovascular disease or stroke combined with a search of Embase, Science Citation Index and Biological Abstract. "
12/01/1997 - "When an ischemic stroke was identified, the following tests were performed: an ultrasound examination of the cervical arteries, a cerebral arteriogram, a lumbar puncture, an investigation of the coagulation factors and lipid status, a measurement of homocystine in the plasma and the urine, an electrocardiogram, a Holter procedure and a cardiac echography. "
10/01/1997 - "Total homocysteine (tHcy) (free homocysteine, protein-bound homocysteine, the disulfide homocystine and the mixed disulfide homocysteine-cysteine) concentration was 10.91 (4.95-23.05) mumol/l in the stroke patients and 8.73 (3.95-15.10) mumol/l in controls (p = 0.031). "
12/01/2004 - "Intimal hyperplasia group received either (1) control or (2) 4.5 g/kg DL-homocystine diets for two weeks followed by CEA and treated with diluent or 5.0 microg Saratin. "
09/01/2001 - "Increasing dietary homocystine produced a proportionate increase in plasma homocysteine and an increase in intimal hyperplasia. "
09/01/2001 - "Increasing levels of dietary homocystine with carotid endarterectomy produced proportionate increases in plasma homocysteine and intimal hyperplasia."
09/01/2001 - "Increasing dietary homocystine increased plasma homocysteine and was followed by increases in the replicative index thus producing increased intimal hyperplasia and lumenal stenosis. "
01/01/2005 - "Male Sprague-Dawley rats underwent carotid endarterectomy (CEA) and treatment with either placebo or varying regimens of clopidogrel, including chronic, pre-CEA bolus, chronic plus pre-CEA bolus, and chronic plus post-CEA bolus; a homocystine diet was used to elevate both plasma homocysteine and the degree of intimal hyperplasia. "
11/01/1986 - "High-affinity GABA uptake into astrocytes was notably inhibited by L-homocystine, none of the other compounds tested causing appreciable inhibition below a concentration of 5 mM. Neuronal and synaptosomal high-affinity uptake of GABA was not significantly affected by any of the test compounds at concentrations below 5 mM. The implication of these results to the study of the mechanism of homocysteine-induced seizures and their relevance to the genetic disorder homocystinuria is discussed."
03/01/2006 - "Homocystinuria is due to enzymatic deficiencies resulting in elevated blood levels of homocysteine (Hcy), homocystine (Hci), and/or methionine (Met) and the clinical presentation of mental retardation, seizures, and cardiovascular disease. "
|9.||gamma-Aminobutyric Acid (GABA)