|1.||Martinez, Laurent O: 3 articles (01/2015 - 01/2005)|
|2.||Perret, Bertrand: 3 articles (01/2015 - 01/2005)|
|3.||Ishmukhametov, Robert: 3 articles (03/2014 - 07/2008)|
|4.||Hornung, Tassilo: 3 articles (03/2014 - 07/2008)|
|5.||Frasch, Wayne D: 3 articles (03/2014 - 07/2008)|
|6.||Wada, Yoh: 3 articles (07/2013 - 01/2010)|
|7.||Sun-Wada, Ge-Hong: 3 articles (07/2013 - 01/2010)|
|8.||Kirk, Kiaran: 3 articles (05/2013 - 02/2003)|
|9.||Cuezva, José M: 3 articles (01/2011 - 07/2004)|
|10.||Tresguerres, Martin: 3 articles (07/2010 - 05/2007)|
03/01/2011 - "We have previously shown that treatment of cancer cells with EF results in disruption of the Hsp90:F(1)F(0)-ATPase complex and inhibition of Hsp90 chaperone activity. "
01/01/2011 - "Metabolic reprogramming resulting in enhanced glycolysis is a phenotypic trait of cancer cells, which is imposed by the tumor microenvironment and is linked to the down-regulation of the catalytic subunit of the mitochondrial H+-ATPase (β-F1-ATPase). "
01/01/2005 - "Here, we show that an entity related to the mitochondrial F1-ATPase is expressed on tumor cell surface and promotes tumor recognition by Vgamma9Vdelta2 T cells. "
07/01/2004 - "The beta-F1-ATPase/Hsp 60 ratio is significantly higher in well differentiated and bronchioloalveolar tumors than in moderate or poorly differentiated and in bronchial-derived tumors. "
01/01/2004 - "Plasmalemmal vacuolar-type H+-ATPase in cancer biology."
04/01/2012 - "The subsequent increased expression of NHE3, H(+)-ATPase in the kidney may play a role in promoting acid excretion in the later stage of acid loading, which counteract the development of metabolic acidosis."
12/01/2007 - "Granulosa cells were also found to possess a V-type H(+)-ATPase that drives partial acidosis recovery when Na(+)/H(+) exchange is inactivated. "
03/01/2007 - "Vacuolar-type H+-ATPase-mediated acidosis promotes in vitro osteoclastogenesis via modulation of cell migration."
09/20/2005 - "Apical expression of the alternative B-subunit isoform, B2, is increased in Atp6v1b1(-/-) medulla and colocalizes with the H(+)ATPase E-subunit; however, the greater severity of metabolic acidosis in Atp6v1b1(-/-) mice after oral acid challenge indicates that the B2-subunit cannot fully functionally compensate for the loss of B1. "
12/01/1998 - "Metabolic acidosis in vivo, as well as in vitro (1 h at pH 6.8 followed by 2 h at pH 7.4) stimulates H+-ATPase-dependent H+ secretion in outer medullary collecting ducts from the inner stripe (OMCDi) (S. "
10/01/2015 - "Gene expression analyses highlighted for the first time that at least two members of the multidrug and toxic compound extrusion (MATE) transporter family and one member of the plasma membrane H(+)-ATPase family are involved in the response to both P and Fe starvation in strawberry plants. "
01/01/2006 - "IAA might be involved in signal transduction of P starvation by activating the plasma membrane H+-ATPase in soybean roots."
01/01/2006 - "Taken together, these results suggested an involvement of root plasma membrane H+-ATPase in the adaptation of soybean to P starvation. "
01/01/2006 - "Results from real-time RT-PCR, western-blotting and immunolocalization analysis indicated that the increase in activity of the plasma membrane H+-ATPase by P starvation was caused by its transcriptional and translational regulation. "
01/01/2006 - "These results suggested that P uptake might be regulated via the modulation of the activity of plasma membrane H+-ATPase under P starvation. "
09/01/2015 - "Similarly, the bone resorption marker vacuolar type H+-ATPase was increased in the NR group compared with that in the control group (P = 0.041). "
05/01/1997 - "Acidification of the bone surface, leading to bone resorption, is accomplished by a vacuolar-type H+-ATPase present in a specialized domain of the plasma membrane of the osteoclast known as the ruffled membrane. "
03/09/2005 - "In mice and man, loss of ClC-7 or the H(+)-ATPase a3 subunit causes osteopetrosis, a disease characterized by defective bone resorption. "
03/01/2007 - "Localized acidification of the osteoclast-bone interface is driven by a vacuolar-type H+-ATPase (V-ATPase) in the plasma membrane in a process thought to be associated with bone resorption. "
09/20/2002 - "The present experiments were carried out to further test the hypothesis that arterial calcification is linked to bone resorption by determining whether the selective inhibition of bone resorption with SB 242784, a specific inhibitor of the osteoclastic V-H+-ATPase, will inhibit arterial calcification. "
|5.||Renal Tubular Acidosis (Distal Renal Tubular Acidosis)
01/01/2015 - "We present a case of distal renal tubular acidosis-associated Sjögren's syndrome with a defect in H(+)-ATPase and pendrin in the renal collecting duct. "
01/01/2015 - "Decreased Renal Expression of H(+)-ATPase and Pendrin in a Patient with Distal Renal Tubular Acidosis Associated with Sjögren's Syndrome."
08/01/2004 - "The urine-blood PCO gradient as a diagnostic index of H(+)-ATPase defect distal renal tubular acidosis."
06/01/2002 - "Secretory-defect distal renal tubular acidosis is associated with transporter defect in H(+)-ATPase and anion exchanger-1."
12/01/1998 - "H+-ATPase in hyperkalemic distal renal tubular acidosis."
|1.||Adenosine Triphosphatases (ATPase)
|2.||dirhodium tetraacetate (DRTA)
|7.||Carbonic Anhydrases (Carbonic Anhydrase)
|8.||Protein Isoforms (Isoforms)
|10.||Heat-Shock Proteins (Heat-Shock Protein)
|1.||Drug Therapy (Chemotherapy)
|2.||Proximal Gastric Vagotomy