|1.||Dwivedi, Prabhaker: 3 articles (10/2008 - 03/2007)|
|2.||Sharma, Anil K: 3 articles (10/2008 - 03/2007)|
|3.||Kumar, Manoj: 3 articles (10/2008 - 03/2007)|
|4.||Singh, Nittin D: 3 articles (10/2008 - 03/2007)|
|5.||Patil, Rajendra D: 3 articles (10/2008 - 03/2007)|
|6.||Eddleston, Michael: 3 articles (04/2008 - 01/2003)|
|7.||Camadro, Elsa L: 2 articles (09/2014 - 06/2008)|
|8.||Menone, Mirta L: 2 articles (09/2014 - 06/2008)|
|9.||Pérez, Débora J: 2 articles (09/2014 - 06/2008)|
|10.||Bernabò, Ilaria: 2 articles (10/2013 - 01/2009)|
05/01/2013 - "Characteristics of endosulfan poisoning: a study of 23 cases. "
07/01/2010 - "Among 34 patients hospitalized during this study period for alleged organochlorine poisoning, 16 patients with endosulfan poisoning were identified. "
01/01/2008 - "One ITS study that evaluated the effect of legislation to ban Endosulfan pesticide on fatal pesticide poisonings increased the level of poisonings immediately after the introduction with an effect size of 2.20 (95% CI 0.97 to 3.43) but led to decrease in the trend of poisonings over time with an effect size of -2.15 (95% CI -2.64 to -1.66). "
12/01/2006 - "Characteristics of endosulfan poisoning: a study of 23 cases."
03/01/2000 - "The objective of the study is to identify the clinical profile of endosulfan poisoning and also to recognise any biochemical parameters which indicate clinical or subclinical dysfunction of organs so that the offending agent can be easily identified in a given case of poisoning and appropriate treatment instituted promptly. "
|2.||Body Weight (Weight, Body)
07/01/1993 - "In order to study the response of rats to repeated administration of the insecticide, endosulfan during the period of growth to maturity, food intake, body weight gain, Spontaneous Motor Activity (SMA) and Muscle Coordination (MC) were determined at regular intervals in male immature Wistar rats treated with a tolerated dose of (2 mg/kg/day) orally for 90 days. "
01/31/2009 - "Our results show that 0.05 and 0.1mg endosulfan/L caused impaired behaviour, prolonged time to metamorphosis, increased incidences of mouth and skeletal malformations as well as mortality, and reduced body weight (observed also at 0.01 mg/L) in B. "
07/01/2003 - "Endosulfan (13mg/kg/day body weight) was administered orally to mices via intragastric-during 10 days. "
11/01/2002 - "There were no significant change in body weight or the weights of the uterus, kidney, adrenal, liver, thymus, or thyroid after endosulfan treatment. "
12/01/1999 - "Male rats of pubertal age were orally administered endosulfan at a dose of 1.0 mg/kg body weight for 30 days. "
|3.||Chromosome Aberrations (Chromosome Abnormalities)
08/01/2012 - "The chromosome aberration (karyotype) was observed in endosulfan-exposed group. "
06/01/2008 - "The frequency of micronuclei (MN) and chromosome aberrations in anaphase-telophase (CAAT) was determined in root tips of the wetland macrophyte Bidens laevis exposed to environmentally relevant concentrations of endosulfan (0.01, 0.02, 0.5 and 5microg/L) for 48h. "
09/01/2014 - "Previous studies in the wetland macrophyte Bidens laevis L have demonstrated that the insecticide endosulfan induces a high frequency of somatic chromosome aberrations in anaphase-telophase (CAAT) but no DNA changes as determined by the single cell gel electrophoresis (Comet) assay. "
02/01/2015 - "The mean concentration of endosulfan in aquaria was always constant, when analyzed by dispersive liquid-liquid micro extraction (DLLME) followed by GC-MS. Autopsy was done on days 1, 2, 3, 4, 7, 14, 21, 28 and 35 of endosulfan exposure; the micronucleus formation (MN), authenticated by scanning electron microscopy, and chromosomal aberrations (CA), were induced significantly (p<0.05) in all the treated groups, including positive control cyclophosphamide (4 ppm), when compared to negative control. "
01/01/1989 - "It was shown that six pesticides induced a statistically significant increase in the number of chromosomal aberrations: Bi 58 EC, Metasystox (I) forte, Sadofos 30, Nogos 50 EC, Foschlor 25 and Thiodan 35. "
|4.||Hepatocellular Carcinoma (Hepatoma)
11/01/2015 - "Present study reports the identification of genomic and proteomic signatures of endosulfan exposure in hepatocellular carcinoma cells (HepG2). "
11/01/2015 - "An integrated genomic and proteomic approach to identify signatures of endosulfan exposure in hepatocellular carcinoma cells."
10/09/2012 - "Effects of endosulfan on hepatoma cell adhesion: Epithelial-mesenchymal transition and anoikis resistance."
11/01/2015 - "Furthermore, downregulation of PHLDA gene, upregulation of ACIN1 protein and caspase-3 activation in exposed cells indicated that endosulfan can trigger apoptotic cascade in hepatocellular carcinoma cells. "
05/15/2001 - "In human hepatoma HepG2 cells, which lack functional ER, alpha-endosulfan, but not 17beta-estradiol, displays a repressive effect on CYP1A1 through a different mechanism. "
|5.||Breast Neoplasms (Breast Cancer)
04/01/2011 - "Moreover, extracts of CN treated with endosulfan, dieldrin, or lindane induced cell proliferation in MCF-7 human breast cancer-derived cells, whereas only extracts of CGC treated with dieldrin induced MCF-7 cell proliferation. "
11/01/1997 - "Endosulfan and dieldrin showed no synergism in displacing 3H-E2 from rat uterine ER or in inducing the proliferation of MCF-7 breast cancer cells, an estrogen-dependent response. "
11/01/2001 - "The effects of simultaneous administration of some binary mixtures of seven natural and synthetic oestrogenic substances (17beta-estradiol, estrone, bisphenol A, butylbenzyl phthalate, endosulfan, methoxychlor and pentachlorophenol) on the cellular proliferation of human breast cancer MCF-7 cells in-vitro (a modified E-screen assay) have been measured. "
03/01/2014 - "Activity of the sinusoidal influx transporter OCT1 (organic cation transporter 1) was thus inhibited by endosulfan, chlordane, heptachlor, lindane, and dieldrine, but not by dichlorodiphenyltrichloroethane isomers, whereas those of the canalicular efflux pumps MRP2 (multidrug resistance-associated protein 2) and BCRP (breast cancer resistance protein) were blocked by endosulfan, chlordane, heptachlor, and chlordecone; this latter OC additionally inhibited the multidrug resistance gene 1 (MDR1)/P-glycoprotein (P-gp) activity. "
05/01/2008 - "This study investigated the effects of E2, diethylstilbestrol (DES), antiestrogens, the phytoestrogen resveratrol, and the xenoestrogens octylphenol (OP), nonylphenol (NP), endosulfan, kepone, 2,3,4,5-tetrachlorobiphenyl-4-ol (HO-PCB-Cl(4)), bisphenol-A (BPA), and 2,2-bis-(p-hydroxyphenyl)-1,1,1-trichloroethane (HPTE) on induction of luciferase activity in breast cancer cells transfected with a construct (pSp1(3)) containing three tandem GC-rich Sp binding sites linked to luciferase and wild-type or variant ERalpha. "
|2.||serum P-component (CIt)
|4.||DNA (Deoxyribonucleic Acid)
|7.||Estrogen Receptor alpha
|8.||Estrogen Receptor Modulators (Antiestrogen)
|2.||Artificial Respiration (Mechanical Ventilation)
|3.||Renal Replacement Therapy (Therapies, Renal Replacement)
|4.||Renal Dialysis (Hemodialysis)