|1.||Samoilov, M O: 2 articles (07/2003 - 06/2001)|
|2.||Głuch-Lutwin, Monika: 1 article (03/2014)|
|3.||Więckowski, Krzysztof: 1 article (03/2014)|
|4.||Solnica, Bogdan: 1 article (03/2014)|
|5.||Gawlik, Katarzyna: 1 article (03/2014)|
|6.||Sałat, Kinga: 1 article (03/2014)|
|7.||Librowski, Tadeusz: 1 article (03/2014)|
|8.||Witalis, Jadwiga: 1 article (03/2014)|
|9.||Pawlica-Gosiewska, Dorota: 1 article (03/2014)|
|10.||Nawieśniak, Barbara: 1 article (03/2014)|
07/01/2003 - "These studies showed that DTNB (200 microM) efficiently prevented the suppression of focal EPSP generation due to long-term anoxia in most slices. "
04/09/2008 - "SH-oxidation by DTNB (5,5'-dithiobis 2-nitrobenzoic acid) or H2O2 postponed the onset of hypoxia-induced spreading depression (HSD), thereby delaying the loss of neuronal function, whereas SH-reduction by DTT (1,4-dithio-dl-threitol) hastened HSD onset. "
08/01/2005 - "While the targets of DTT are less clear, the postponement of HSD by DTNB indicates that sulfhydryl oxidation increases the tolerance of hippocampal tissue slices against hypoxia. "
02/01/2004 - "In the presence of hypoxia, the K(0.5) for activation of I(Ks) by Iso was significantly decreased from 18.3 to 1.9 nm. DTT mimicked the effect of hypoxia on the sensitivity of I(Ks) to Iso while DTNB had no effect. "
07/01/2003 - "In addition, DTNB partially reversed the protective effect of preconditioning with short-term anoxia on the impairment of focal EPSP generation induced by long-term anoxia. "
05/01/2009 - "TrxR activity in plasma and tumor tissues was detected by dithio-bis-nitrobenzoic acid (DTNB) method. "
05/22/2012 - "RNAi-mediated knockdown in a liver progenitor cell line further validate three of these genes, Ncoa2/Src-2, Zfx, and Dtnb, as tumor suppressors in liver cancer. "
01/01/1981 - "Aliquots from preparations of rat hepatocytes, Yoshida ascites tumor cells, Ehrlich ascites tumor cells, isolated nuclei of Ehrlich-cells and chicken thymocytes were investigated as well for the protein thiol content of the cells macroscopically with DTNB as microspectrophotometrically for the extinctions of the cells after staining with MT4- or MCN-method. "
08/01/2013 - "In order to identify the mechanism of OA-mediated radiosensitization of tumor cells, the levels of glutathione (GSH) in irradiated cells following various pretreatments were determined using glutathione reductase/5,5'-dithiobis-(2-nitrobenzoic acid) (DTNB) recycling assay. "
09/01/1992 - "The results revealed differences in accessibility and titration of histone H3 SH-groups in DNP of normal and tumor cells with DTNB, as well as in molecular dynamics of the mercury-containing spin label introduced to these SH-groups."
01/01/2011 - "Lastly, we also found evidence of long-lasting host cell protection in vitro by DTNB, an important pharmacodynamic parameter for a topical microbicide against virus infection, hours after the extracellular drug was removed; this protection was not rendered by Bacitracin. "
02/01/2002 - "Interestingly, exposure of EB to the membrane-impermeable thiol-alkylating reagent 5,5'-dithiobis(2-nitrobenzoic acid) enhanced attachment but compromised infectivity, suggesting that EB outer membrane proteins must be reduced for entry and productive infection. "
09/01/1998 - "SFV and SIN infection were partially inhibited by the thiol blocker DTNB, but not by pCMBS or Thiolyte MQ. "
06/01/2013 - "Rotavirus infection of villi proved to be sensitive to membrane-impermeant thiol/disulfide inhibitors such as DTNB and bacitracin, suggesting the involvement of a redox reaction in infection. "
01/01/2012 - "We found that the nonspecific thiol/disulfide exchange inhibitor, 5,5'-dithiobis(2-nitrobenzoic acid) (DTNB), significantly reduced HIV-1 entry and infection in cell lines, human monocyte-derived macrophages (MDM), and also phytohemagglutinin (PHA)-stimulated peripheral blood mononuclear cells (PBMC). "
06/01/2004 - "DTNB completely blocked L-cysteine-induced thermal hyperalgesia in both animal groups. "
07/19/2001 - "Likewise, L-cysteine induces mechanical DTNB-sensitive hyperalgesia in peripheral receptive fields. "
07/19/2001 - "This hyperalgesia is blocked by the oxidizing agent 5,5'-dithio-bis-(2-nitrobenzoic acid) (DTNB) and the T channel antagonist mibefradil. "
03/01/1999 - "administration of dynorphin, whereas post-treatment with DTNB reduced the permanent allodynia induced by dynorphin for 5 days. "
06/01/2004 - "DTNB, an exogenous oxidizing agent, induced dose-dependent alleviation of thermal hyperalgesia in rats with CCI of the sciatic nerve and caused analgesia in sham-operated rats. "
|1.||Dithionitrobenzoic Acid (Ellman's Reagent)
|3.||Proteins (Proteins, Gene)
|7.||p-Chloromercuribenzoic Acid (PCMB)
|8.||Indicators and Reagents (Reagents)
|9.||R-SNARE Proteins (Vesicle-Associated Membrane Protein)
|10.||Ryanodine Receptor Calcium Release Channel (Ryanodine Receptor)