|1.||Gokarakonda, Amulya: 1 article (11/2013)|
|2.||Czene, Stefan: 1 article (11/2013)|
|3.||Osterman Golkar, Siv: 1 article (11/2013)|
|4.||Haghdoost, Siamak: 1 article (11/2013)|
|5.||Zomer, Aldert: 1 article (08/2013)|
|6.||van Sinderen, Douwe: 1 article (08/2013)|
|7.||Ainsworth, Stuart: 1 article (08/2013)|
|8.||Mahony, Jennifer: 1 article (08/2013)|
|9.||Dumontet, Charles: 1 article (07/2011)|
|10.||Nanni, Patrizia: 1 article (07/2011)|
11/11/1994 - "Comparing the anaerobic burst size of 55.9- infections to that of the parental phage indicates that anaerobic de novo synthesis of deoxyribonucleotides is nearly abolished in phage lacking the 55.9 product. "
08/01/2013 - "Despite the apparent lack of a coordinated general phage response, three themes common to both infections were noted: alternative transcription of genes involved in catabolic flux and energy production, differential transcription of genes involved in cell wall modification, and differential transcription of genes involved in the conversion of ribonucleotides to deoxyribonucleotides. "
09/01/1995 - "To clarify the role of the ribonucleotide reductase in virus infection and to evaluate it as an antiviral target, the metabolism of deoxyribonucleotides in infected cells was examined. "
05/05/1988 - "The accompanying paper describes the decreased rate of synthesis of deoxyribonucleotides after infection by the T4 mutant, nrdB93, and the suppression of this defect by a second mutation in gene 39, coding for one of the three protein chains of T4 DNA topoisomerase. "
05/10/1977 - "The kinetics of the de novo formation of pyrimidine deoxyribonucleotides is the same after infection by wild type bacteriophage T4, which generate very low steady state levels of deoxytibonucleotides, and by T4 DNA synthesis-negative mutatants (Dna-), which accumulate high levels, suggesting that the control is not by a feedback mechanism. "
07/01/2011 - "Tumor specificity resided in low PKR activity, and high deoxyribonucleotides content of cancer cells. "
01/01/2005 - "Taking in account that DNA replication and repair are essential mechanisms for cell integrity and are dependent on the availability of deoxyribonucleotides, many researchers are giving special attention to this enzyme, since it is an attractive target to treat several diseases of our time specially cancer. "
05/01/1957 - "Purine deoxyribonucleotides in rat tumor."
|3.||Precursor T-Cell Lymphoblastic Leukemia-Lymphoma (T-ALL)
12/10/2005 - "When the pH of the DNA solution buffer increased from 3 to 11, the A260/A280 ratio changed significantly from 1.5 to 2.2 in mixtures of DNA bases [A:T:C:G = 28.5:28.5: 21.5:21.5, i.e., (A + T)/(all four bases) = 57%, expressed as mole percent], of deoxyribonucleosides (adenosine:thymidine:cytidine:guanosine= 28.5:28.5:21.5:21.5, as mole percent), or of deoxyribonucleotides (dAMP:dTMP:dGMP:dCMP = 28.5:28.5:21.5:21.5, as mole percent) examined. "
|5.||HIV Infections (HIV Infection)
|1.||Ribonucleotide Reductases (Ribonucleotide Reductase)
|2.||DNA (Deoxyribonucleic Acid)
|6.||triphosphoric acid (triphosphate)
|7.||Vascular Endothelial Growth Factor A (Vascular Endothelial Growth Factor)
|10.||Thymidine Monophosphate (TMP)