|1.||De Meyer, Guido R Y: 4 articles (06/2009 - 08/2005)|
|2.||Herman, Arnold G: 4 articles (06/2009 - 08/2005)|
|3.||Martinet, Wim: 4 articles (06/2009 - 08/2005)|
|4.||Nikitin, V P: 3 articles (08/2014 - 03/2010)|
|5.||Sung, Dong Kyung: 3 articles (07/2013 - 04/2006)|
|6.||Park, Won Soon: 3 articles (07/2013 - 04/2006)|
|7.||Chang, Yun Sil: 3 articles (07/2013 - 04/2006)|
|8.||Lee, Jang Hoon: 3 articles (07/2013 - 04/2006)|
|9.||Anokhin, K V: 3 articles (03/2013 - 11/2000)|
|10.||Croons, Valerie: 3 articles (06/2009 - 03/2007)|
|1.||Amnesia (Dissociative Amnesia)
04/01/1984 - "Amnesia for a single trial discriminated passive avoidance learning task in day-old chicks, resulting from the antibiotic cycloheximide, is shown to begin after 30 min following learning when cycloheximide is given immediately after or before learning. "
01/01/1978 - "Studies on memory: spontaneous return of memory in 6-hydroxydopamine-treated mice and its relation to cycloheximide-induced transient amnesia."
03/01/2013 - "Mice were injected with protein synthesis inhibitor cycloheximide twenty minutes prior learning to induce amnesia. "
03/01/2013 - "Injection of cycloheximide (an inhibitor of protein synthesis) to the snails in 48 h after training combined with subsequent reminder and presentation of the conditional stimulus resulted in the development of persistent amnesia and depression of the responses of the defensive behavior command neurons LPl1 and RPl1 to the conditional stimulus. "
09/01/2012 - "In mice, amnesia was induced by intraperitoneal cycloheximide (100 mg/kg) administration 20 min before exposure to tone signal combined with footshock (2 sec, 0.5 mA). "
06/01/1989 - "Optimal conditions of heat shock for enhanced mutagenesis were determined in 2.5 X 10(7) conidia/ml 0.067 M KH2PO4-Na2HPO4 (pH 7.0) buffer to be treatment at 43 degrees C for 60 min. When protein synthesis during heat stock was eliminated by cycloheximide or by use of the temperature-sensitive mutation psi-1, induction of thermotolerance was inhibited while induction of the enhanced state of mutability was not. "
07/01/1986 - "The presence of the protein synthesis inhibitor cycloheximide during spore exposure to 45 degrees C did not abolish the protection against freezing injury induced by heat shock. "
01/01/1994 - "However, changes of heat shock immunoreactivity in the postischemic hippocampus were not seen in the present study following cycloheximide injection."
11/01/1977 - "Utilizing a dosage of cycloheximide which was found to inhibit cerebral protein synthesis by almost 90% after injection, separate groups of 13-day-old mice received either cycloheximide or saline followed by 0 (control), 15, or 25 training trials in a discriminated shock-escape T-maze. "
01/01/2015 - "pombe, SGA methods use either cycloheximide (C) or heat shock (HS) to select double mutants before measuring colony size as a surrogate for fitness. "
11/30/2007 - "In our previous study, we found that cycloheximide (CHX) induces hepatocellular necrosis as well as hepatocellular apoptosis. "
11/01/2015 - "The results showed that the numbers of microparticles were increased by tumour necrosis factor (TNF) or the combination of TNF and cycloheximide (CHX). "
01/01/2013 - "However, in the presence of cycloheximide or SMAC mimetics, programmed necrosis was only moderately reduced in CYLD(-/-) cells. "
02/01/2012 - "In cultured human proximal tubule (HK-2) cells, the S1P(2)R antagonist selectively upregulated SK1 and attenuated both H(2)O(2)-induced necrosis and TNF-α/cycloheximide-induced apoptosis; the S1P(2)R agonist had the opposite effects. "
06/01/2009 - "Cycloheximide (CHX)-induced liver injury in rats has been characterized by hepatocellular apoptosis and necrosis. "
01/01/2012 - "RT-PCR and cycloheximide inhibition experiments indicated that PMS-induced HIF-1α decrease is involved in post-translational degradation during hypoxia. "
10/01/2007 - "Cycloheximide did not block the effect of hypoxia on D2 activity and D2 half-life was enhanced under hypoxia demonstrating a posttranslational action of hypoxia. "
02/01/2004 - "We have shown that the specific inhibition of hypoxia-induced relaxation by organ culture in porcine coronary arteries can be mimicked by treatment of control vessels with the protein synthesis inhibitor, cycloheximide. "
11/09/2000 - "Similiar results were obtained upon application of cycloheximide (CHX; 1 microM) simultaneously with hypoxia (34.75 +/- 5.6% vs 56.75 +/- 7.3% with and without CHX, respectively, n = 4, p < 0.035), indicating that hypoxia-induced neuronal death is an active, protein synthesis-dependent process. "
06/01/2000 - "Cycloheximide pretreatment suggested that hypoxia-induced downregulation of IL-1beta expression did not require de novo protein synthesis."
|5.||Brain Injuries (Brain Injury)
09/01/1998 - "Cycloheximide eliminated ischemic PC-induced IT effects on both brain injury and neurological deficits if administered before PC (P<0.05) but not if administered long after PC but before PMCAO. "
04/01/2006 - "This study was done to determine the neuroprotective effect of cycloheximide on neonatal hypoxic-ischemic brain injury. "
01/01/2013 - "Cycloheximide also protected neurons from hypoxic cell death, indicating that translational inhibitors may generally reduce secondary brain injury. "
06/01/2006 - "Therapeutic window for cycloheximide treatment after hypoxic-ischemic brain injury in neonatal rats."
04/01/2006 - "Neuroprotective effect of cycloheximide on hypoxic-ischemic brain injury in neonatal rats."
|2.||Tumor Necrosis Factor-alpha (Tumor Necrosis Factor)
|4.||DNA (Deoxyribonucleic Acid)
|5.||Staphylococcal Protein A (A, Protein)
|8.||RNA (Ribonucleic Acid)
|10.||Protein Synthesis Inhibitors
|4.||Drug Therapy (Chemotherapy)