|1.||Ki, Chang-Seok: 2 articles (03/2006 - 04/2005)|
|2.||Kang, Hee Jung: 2 articles (03/2006 - 04/2005)|
|3.||Kim, Eui-Chong: 1 article (03/2013)|
|4.||Heo, Jung Yeon: 1 article (03/2013)|
|5.||Sim, Sung Hoon: 1 article (03/2013)|
|6.||Choe, Kang-Won: 1 article (03/2013)|
|7.||Welsh, Kerry J: 1 article (01/2012)|
|8.||Actor, Jeffrey K: 1 article (01/2012)|
|9.||Lewis, Cole T: 1 article (01/2012)|
|10.||Braun, Michael C: 1 article (01/2012)|
08/15/2006 - "[Combined-heterozygous deficiency of complement C7 in a patient with recurrent meningitis]."
01/01/1992 - "[Recurrent Neisseria meningitidis meningitis associated with homozygote complement C7 fraction deficiency]."
08/01/1989 - "We describe an 11-year-old girl suffering from recurrent meningitis with a complete absence of the seventh component of complement (C7). "
06/01/1988 - "Three recent cases of inherited deficiency of the seventh component of complement (C7) associated with recurrent infectious meningitis are described. "
03/01/2013 - "A case of meningococcal sepsis and meningitis with complement 7 deficiency in a military trainee."
03/01/2013 - "Complement component 7 (C7) deficiency leads to the loss of complement lytic function, and affected patients show increased susceptibility to encapsulated organisms infection, especially Neisseria meningitidis. "
01/01/2012 - "Mice deficient in complement C7 and wild-type C57BL/6 mice were aerosol challenged with MTB Erdman and assessed for bacterial burden, histopathology, and lung cytokine responses at days 30 and 60 post-infection. "
01/01/1991 - "64 members of a large kindred with inherited deficiency of the seventh component of complement, C7, were studied for plasma levels of antigenetic and functional components of complement as well as for clinical manifestations of infections and autoimmune diseases. "
03/01/2006 - "Complement C7 deficiency is associated with increased susceptibility to meningococcal infection. "
04/01/2005 - "Complement C7 deficiency is an autosomal recessive disorder well known to be associated with increased susceptibility to meningococcal infection and has mostly been reported in Caucasians. "
|5.||Focal Epithelial Hyperplasia
06/01/1979 - "Support for its specificity to fibropapilloma derived bovine papilloma virus comes from (I) the absence of transformation associated changes following inoculations of normal bovine skin, bovine teat papilloma, bovine teat focal epithelial hyperplasia lesion and canine papilloma derived suspensions; (2) in vitro transformation occurs when CsCl purified fibropapilloma-derived BPV is used; (3) in vitro transformation experiments reported here parallel in vivo transmission of fibropapillomas described elsewhere using the same extracts; (4) the time-temperature inactivation of BPV using in vitro transformation parallels in vivo results and is similar to that reported for other papovaviruses; (5) BPV in vitro transformed cells are tumorigenic in nude mice and increase in vivo resistance in calves to subsequent challenge with fibropapilloma derived virus; (6) inhibition of in vitro transformation by bovine papilloma virus occurs when sera from calves bearing regressing fibropapillomas are used in conjunction with complement; (7) transformation inhibition activity may be adsorbed from high titre sera using BPV-induced tumour cells or in vitro transformed cells but not using various virus suspensions. "
|1.||Complement System Proteins (Complement)
|3.||butyl 3-(1H-tetrazol-5-yl)oxanilate (MTB)
|4.||cesium chloride (CsCl)