|1.||Chen, Wei: 2 articles (01/2015 - 04/2011)|
|2.||Baeg, Gyeong Hun: 1 article (07/2015)|
|3.||Salim, Agus: 1 article (07/2015)|
|4.||Tan, Puay-Hoon: 1 article (07/2015)|
|5.||Bay, Boon Huat: 1 article (07/2015)|
|6.||Yu, Yingnan: 1 article (07/2015)|
|7.||Thike, Aye Aye: 1 article (07/2015)|
|8.||Matsumoto, Ken: 1 article (07/2015)|
|9.||Scully, Olivia Jane: 1 article (07/2015)|
|10.||Yip, George Wai-Cheong: 1 article (07/2015)|
09/01/2014 - "Hereditary angioedema is caused by a deficiency (type I) or dysfunction (type II) in complement C1 inhibitor. "
06/01/2013 - "Hereditary angioedema (HAE) is a genetic syndrome caused by a functional deficit in complement C1 inhibitor that results in recurrent episodes of nonpruritic swelling of the hands, feet, arms, legs, trunk, face, genitalia, bowels, and larynx beginning in childhood or adolescence and continuing throughout the patient's lifetime. "
11/01/2006 - "Hereditary angioedema (HAE) is an infrequent disorder characterized by abnormalities in the levels and/or function of complement C1 esterase inhibitor. "
01/01/2005 - "Hereditary angioedema is a disorder characterized by decreased levels or function of complement C1 esterase inhibitor. "
03/01/2002 - "Hereditary angioedema is a rare disorder characterized by quantitative or qualitative deficiency of complement C1 esterase inhibitor. "
05/28/1987 - "During the past 25 years, three forms of deficiency of the inhibitor of the first component of complement (C1 inhibitor) with angioedema have been recognized; two forms are hereditary and one is acquired. "
12/01/1993 - "Acquired complement component 1 (C1) inhibitor deficiency with consequent angioedema is a rare condition that may indicate an underlying neoplasm of B-lymphocytes or plasma cells. "
10/01/2013 - "Approximately 2% of angioedema (AE) patients have a hereditary or an acquired deficiency of the complement 1 (C1) esterase inhibitor (C1 INH) gene. "
01/01/2006 - "Angioedema from angiotensin-converting enzyme (ACE) inhibitor treated with complement 1 (C1) inhibitor concentrate."
08/01/1986 - "The first component of complement (C1) inhibitor plays a critical role in the regulation of the classical complement pathway and the contact system, and the deficiency of C1 inhibitor protein or function is associated with recurrent angioedema. "
05/01/2007 - "These data suggests that both adipose and immune cells are the sources for abnormal adipose tissue production of C1 complement and decorin in obesity. "
05/01/2007 - "In this study, we investigated adipose expression and regulation of C1 complement subcomponents and C1 activation regulator decorin in obesity and insulin resistance. "
10/01/2009 - "Three candidate genes not previously associated with obesity or type 2 diabetes, sushi domain containing 2, collagen and calcium-binding EGF domains 1 and periostin (Postn), as well as one gene known to be associated, complement component 1, were shown by RT-PCR to be differentially expressed in skeletal muscle of P. "
|4.||Systemic Lupus Erythematosus (Libman-Sacks Disease)
08/01/2010 - "Recent studies have tested genetic variation at the C1QA, C1QB and C1QC (complement component 1, q subcomponent, A chain, complement component 1, q subcomponent, B chain and complement component 1, q subcomponent, c chain) loci in relation to systemic lupus erythematosus (SLE) risk. "
01/01/1995 - "A large proportion of systemic lupus erythematosus (SLE) patients develop glomerulonephritis, coincident with the appearance of autoantibodies to C1q, the Fc-recognizing collagen-like subcomponent of the first component of complement, C1. "
|5.||Peripheral Nervous System Diseases (PNS Diseases)
02/01/1985 - "Previous studies show that only 27-50% of patients with acute demyelinating neuropathy or Guillain-Barré syndrome (GBS) had serum Abs to peripheral nerve or PNM as demonstrated by consumption of hemolytic activity of serum complement 1 (C1) fixation and transfer assay, quantitative determinations of anti-PNM Ab showed significantly high titers in the serum of patients with GBS, chronic and recurrent polyneuritis, and paraproteinemia associated with peripheral neuropathy. "
|1.||Bradykinin Receptors (Bradykinin Receptor)
|3.||Complement System Proteins (Complement)
|5.||Complement C4 (Complement Component 4)
|6.||Carrier Proteins (Binding Protein)
|7.||Complement C1 Inhibitor Protein (C1 Esterase Inhibitor)
|8.||Peptidyl-Dipeptidase A (Angiotensin Converting Enzyme)
|9.||Immunoglobulin G (IgG)
|10.||Glial Fibrillary Acidic Protein
|2.||Drug Therapy (Chemotherapy)