|1.||Inokuma, Hisashi: 7 articles (01/2013 - 01/2002)|
|2.||Streck, Emilio L: 6 articles (08/2013 - 05/2007)|
|3.||Scaini, Giselli: 5 articles (10/2013 - 05/2010)|
|4.||Quevedo, João: 5 articles (12/2011 - 05/2007)|
|5.||Herndon, David N: 3 articles (09/2015 - 08/2008)|
|6.||Clarke, Kieran: 3 articles (11/2014 - 12/2008)|
|7.||Tarnopolsky, Mark A: 3 articles (01/2014 - 04/2007)|
|8.||Krieger, José E: 3 articles (11/2012 - 11/2006)|
|9.||Gomes, Lara M: 3 articles (12/2011 - 05/2010)|
|10.||Tremblay, Angelo: 3 articles (03/2008 - 03/2002)|
09/01/2015 - "Hypoxia leads to an increase in citrate synthase activity of monocyte mitochondria as a marker of mitochondrial mass, which is not modified by antioxidant supplementation. "
01/01/2015 - "However, decreasing citrate synthase may be essential to preserve local PO2, minimising regions of hypoxia and hence maximising the area of myocardium able to preserve cardiac output following maternal hypoxia. "
11/01/2014 - "After 24 hours of hypoxia, mitochondrial networks were more fragmented but there was no change in citrate synthase activity, indicating that mitochondrial content was unchanged. "
05/01/2014 - "However, hypoxia moderately decreased the oxidative capacity (assessed from the activity of citrate synthase) of intact muscle and delayed its recovery in regenerated muscle. "
07/01/2013 - "In contrast, in striated muscle, hypoxia did not induce any significant modification of neither truncated AMPKα nor AKT protein content, and citrate synthase activity was altered after 6 h of hypoxia. "
|2.||Body Weight (Weight, Body)
08/01/1995 - "The efficacy of exercise training was demonstrated by a 12% increase (P < 0.05) in ventricular weight-to-body weight ratio and increases (P < 0.05) in citrate synthase activity in hindlimb skeletal muscles of ET. "
07/01/1997 - "During weeks 5-7, S5B/Pl exercisers tended to run more than did OM (approximately 60 vs. 45 km/week), but by the end of the study both groups had an equally greater heart weight (mg/g body weight) and planteris citrate synthase activity than their sedentary controls. "
01/01/2013 - "The peak power related to the body weight and citrate synthase activity was not significantly influenced in mice receiving AOX. "
03/01/2009 - "Haemodynamic parameters, soleus citrate synthase (SCS) activity and LVH (left ventricular/body weight ratio, mg/g) were evaluated. "
01/01/2008 - "After an 8-week training period, both exercise groups had significantly less body weight (BW) gain but higher aerobic capacity, shown by increased muscle citrate synthase (CS) activity. "
08/01/2009 - "To explore mechanisms, aortic ring vasodilator function and nitrate (NO(x)) activity, urinary excretion of NO(x), running muscle microvascular density and citrate synthase activity, as well as myocardial mass and histologic evidence of ischemia were measured. "
01/01/2009 - "Citrate synthase activity was also depressed by ischemia and 2 h reperfusion, but the activity recovered by 7 days. "
08/01/2006 - "Ischemia caused decreased recovery of citrate synthase in isolates of SSM (P < .05) but not of IFM. "
04/15/2000 - "Citrate synthase activity was decreased significantly only after 48 hr of ischemia and reperfusion, consistent with the loss of mitochondrial membrane integrity seen in electron micrographs of the transplanted 48-hr group. "
05/01/1996 - "Glycogen levels decreased with ischemia in all groups and returned to preischemic levels in groups 2, 3, and 4. The citrate synthase ratio was low in the control group and preserved in the groups showing improved functional recovery. "
08/01/2008 - "Surprisingly, AChE ablation does not modify twitch kinetics, absolute maximal power, fatigue resistance or citrate synthase activity, despite the reduced number of slow muscle fibres. "
06/01/2005 - "Scand 169: 157-165, 2000) showed that 2 wk of daily sprint interval training (SIT) increased citrate synthase (CS) maximal activity but did not change "anaerobic" work capacity, possibly because of chronic fatigue induced by daily training. "
01/01/1993 - "4. Different time courses of the changes in resistance to fatigue and citrate synthase activity were observed not only with prolonged stimulation periods but also during the first week, when pronounced increases in resistance to fatigue were accompanied by only moderate elevations in citrate synthase activity. "
01/01/1993 - "3. Resistance to fatigue reached maximal values in both rat and rabbit tibialis anterior muscles after stimulation periods of 14 days, whereas citrate synthase activity continued to increase with longer stimulation periods. "
12/01/1992 - "The fatigue index measured from 50 consecutive concentric contractions at 180 degrees/s decreased and the citrate synthase activity increased in all but one subject. "
09/01/1981 - "Muscle weights of the CA-treated freely eating N controls were significantly less than that of N runners that received CA. In plantaris muscles of both N and C animals that received CA, total protein concentration and citrate synthase activity, a mitochondrial marker, remained constant, but their amounts per muscle decreased in proportion to the atrophy. "
09/01/1986 - "Protein deprived rats showed atrophy of type 2 fibres predominantly, reduced histochemical activity of succinic dehydrogenase (SDH) and reduced biochemical activity of citrate synthase. "
05/01/2005 - "Of 26 patients, results of analysis of 3 patients (1 Duchenne muscular dystrophy, 1 spinal muscular atrophy and 1 type II fibre atrophy) were excluded because the citrate synthase activities in their muscle homogenate were less than third percentile of the normal controls. "
03/01/1996 - "It was found that, in sedentary rats, hypoxia results in stunted growth, adrenal atrophy, a significant reduction of cross-sectional area of fast-twitch (type II) fibres, a reduced capillary-to-fibre ratio (C/F), and a reduced oxidative capacity (decreases in citrate synthase and 3-hydroxy-Acyl CoA dehydrogenase activities) of the plantaris muscle. "
01/01/2014 - "Concurrent exercise altered the expression of genes involved in mitochondrial biogenesis (PGC-1α, PRC, PPARγ), hypertrophy (PGC-1α4, REDD2, Rheb) and atrophy (MuRF-1, Runx1), increased electron transport chain complex protein content, citrate synthase and mitochondrial cytochrome c oxidase enzyme activity, muscle mass, maximum isometric strength and VO 2peak. "
|1.||L-Lactate Dehydrogenase (Lactate Dehydrogenase)
|4.||Fatty Acid Synthetase Complex (Fatty Acid Synthase)
|6.||Acyl-CoA Dehydrogenase (Medium-Chain Acyl-Coenzyme A Dehydrogenase)
|8.||coenzyme Q10 (CoQ10)
|10.||Adrenergic Receptors (Adrenergic Receptor)
|2.||Knee Replacement Arthroplasty (Total Knee Replacement)
|4.||Heart Transplantation (Grafting, Heart)
|5.||Surgical Instruments (Clip)