|1.||Contagious Ecthyma (Orf)
|1.||Lee, C W: 1 article (03/2013)|
|2.||Kim, C J: 1 article (03/2013)|
|3.||Kim, T G: 1 article (03/2013)|
|4.||Hur, S Y: 1 article (03/2013)|
|5.||Lee, S J: 1 article (03/2013)|
|6.||Park, J S: 1 article (03/2013)|
|7.||Park, T C: 1 article (03/2013)|
|8.||Kim, J H: 1 article (03/2013)|
|9.||Song, M J: 1 article (03/2013)|
|10.||Brännström, M: 1 article (06/2007)|
|2.||DNA (Deoxyribonucleic Acid)IBA
03/01/1993 - "HPV-16 and HCMV DNA homologous sequence and the status of cellular oncogene C-myc DNA were examined in uterine cervical diseases with dot blot hybridization and Southern hybridization. "
01/01/1998 - "Oncogenicity of human papillomavirus (HPV) DNA in premalignant and malignant uterine cervical diseases is mainly induced by E6/E7 open reading frame (ORF). "
|3.||HLA-C Antigens (HLA-C)IBA
|4.||Volatile Oils (Essential Oils)IBA
|5.||Mitochondrial DNA (mtDNA)IBA
04/01/2011 - "Methylation at 14 of the tested CpG sites within the HPV16 L1 region were significantly higher in CIN3+ compared to HPV16 genomes from women without CIN3+. In contrast, 2/16 CpG sites in HPV16 URR, 5/5 in TERT, 1/4 in DAPK1 and 1/3 mtDNA, and 2/5 in RARB were associated with increased methylation in CIN3+. These results indicate that increased methylation of CpG sites in the HPV16 L1 ORF is associated with CIN3+ and, thus, may constitute a potential biomarker for precancerous and cancerous cervix disease."