|1.||Hu, Sen: 17 articles (07/2011 - 03/2003)|
|2.||Sheng, Zhi-yong: 9 articles (05/2010 - 03/2003)|
|3.||Costa, L G: 6 articles (01/2003 - 02/2000)|
|4.||Guizzetti, M: 6 articles (01/2003 - 02/2000)|
|5.||Sun, Dan: 5 articles (02/2005 - 03/2003)|
|6.||Yang, Li: 4 articles (05/2014 - 02/2011)|
|7.||Ikenoue, Tsuyomu: 4 articles (05/2014 - 02/2011)|
|8.||Furukawa, Seishi: 4 articles (05/2014 - 02/2011)|
|9.||Sameshima, Hiroshi: 4 articles (05/2014 - 02/2011)|
|10.||Tugay, Melih: 4 articles (05/2012 - 11/2003)|
01/01/2000 - "b) Responses to in vitro ischemia: After in vitro ischemia, contractile responses of both normal and hypertrophied tissues to FS were reduced to a significantly greater degree than were responses to carbachol and KCl. "
03/01/1999 - "Repetitive stimulation during in vitro ischemia resulted in a significantly greater decrease in the contractile responses to FS and carbachol than did in vitro ischemia without repetitive stimulation. "
01/01/1998 - "In functional studies, contractile responses to carbachol were cumulatively measured after the urinary bladder was treated with various duration (0, 30, 60, and 90 min) of ischemia. "
08/01/2007 - "To investigate the effects of carbachol on apoptosis of intestinal epithelial cells in rats after gut ischemia/reperfusion (I/R). "
08/01/2007 - "[Effects of carbachol on apoptosis of intestinal epithelial cells after gut ischemia/reperfusion in rat]."
03/01/2010 - "Predominantly in type I fibers, carbachol incubation resulted in strong contractures of the sarcomeres underlying the MEPs. "
05/01/2006 - "The time constants of changes to nerve-mediated contractions and carbachol contracture were also significantly different, at 147 (5) vs 1207 (386) s, respectively. "
05/01/2006 - "RESULTS Reducing extracellular [Na+] reversibly reduced the magnitude of nerve-mediated contractions but increased the resting tension and magnitude of carbachol-induced contracture. "
12/01/2005 - "In chick biventer cervicis preparations, MiDCA1 produced concentration- and time-dependent neuromuscular blockade that reached 100% after 120 min (2.4 microM, n = 6); contractures to exogenously applied carbachol (8 microM) and KCl (13 mM) were still seen after complete blockade. "
01/01/2005 - "Contractures elicited by carbachol, ABMA and KCl increased between Groups 1 and 2, but not thereafter. "
05/01/1995 - "Within the anatomical limits of the study, the zona incerta is the area most sensitive to carbachol-induced generalized seizures."
06/30/2010 - "In mice with recurrent seizures, carbachol-induced enhancement of spontaneous IPSCs (sIPSCs) originating from CCK-containing basket cells was accordingly reduced in CA1 pyramidal cells. "
12/01/2000 - "Since the forebrain seizures were induced by a single carbachol administration, it is proposed that the PAG serves as a pathway for caudal-rostral seizure generalization."
12/01/2000 - "Rats with seizures induced by PAG carbachol microinjections exhibited dense c-fos-like immunoreactivity in the dentate gyrus but not the CA(1) or CA(3) regions, amygdala, piriform cortex, perirhinal cortex or hypothalamus. "
12/01/2000 - "Focal microinjection of carbachol into the periaqueductal gray induces seizures in the forebrain of the rat."
09/01/2011 - "In the tissues related to antimuscarinic side effects, the inhibitory actions were measured each on salivary secretion by electrical stimulation of chorda tympani, on rhythmical contractions in colon, and on carbamylcholine-induced bradycardia. "
11/26/2010 - "Loss of RGS6 provoked dramatically exaggerated bradycardia in response to carbachol in mice and isolated perfused hearts and significantly enhanced the effect of carbachol on inhibition of spontaneous action potential firing in sinoatrial node cells. "
07/01/2010 - "In isolated, beating hearts, carbachol did not prolong action potentials per se, but caused bradycardia and rate-dependent action potential prolongation. "
07/31/2009 - "Using implantable EKG transmitters, we demonstrated that compared with wild-type, Ins2(Akita) type I diabetic mice demonstrated a decrease in the negative chronotropic response to carbamylcholine characterized by a 2.4-fold decrease in the duration of bradycardia, a 52+/-8% decrease in atrial expression of GIRK1 (P<0.01), and a 31.3+/-2.1% decrease in SREBP-1 (P<0.05). "
02/01/2007 - "In contrast, SHR atria were hyperresponsive to carbachol-induced bradycardia, with elevated production of atrial cGMP. "
02/01/2009 - "Carbachol (10 micromol.L(-1)) evoked a positive inotropic response only in muscles from rats with heart failure approximating 36% of that elicited by 1 micromol.L(-1) isoproterenol (20 +/- 1.5% and 56 +/- 6.1% above basal respectively). "
07/01/1996 - "Agonist binding with carbachol also revealed an increase in total muscarinic receptors in heart failure without a change in fraction of high- and low-affinity receptors. "
02/01/2009 - "Carbachol-evoked inotropic responses did not correlate with infarction size but did correlate with increased left ventricular end diastolic pressure, heart weight/body weight ratio and lung weight, primary indicators of the severity of heart failure. "
10/01/1997 - "The effects of carbachol and bradykinin but not SNAP were attenuated by NG-nitro-L-arginine (10(-4) mol/L), indicating inhibition of the formation of endogenous NO. The inhibitory effect on tissue O2 consumption in response to carbachol and bradykinin became significantly smaller in skeletal muscle from dogs with pacing-induced heart failure, but the effects of SNAP were unchanged. "
07/01/1996 - "Dose-response curves for carbachol (10-8 to 10-3 mol/L) inhibition of isoproterenol-stimulated adenylyl cyclase demonstrated significantly greater (P<.05) inhibition in heart failure compared with sham-operated dogs. "
|1.||Muscarinic Receptors (Muscarinic Acetylcholine Receptor)
|2.||Acetylcholine (Acetylcholine Chloride)
|5.||Sympathomimetics (Sympathomimetic Amines)