|1.||Bustos, Susan P: 2 articles (01/2011 - 01/2006)|
|2.||Reithmeier, Reinhart A F: 2 articles (01/2011 - 01/2006)|
|3.||Lutz, Hans U: 2 articles (02/2010 - 04/2007)|
|4.||Alper, Seth L: 2 articles (03/2004 - 01/2002)|
|5.||Nurgaliyeva, A S: 1 article (11/2015)|
|6.||Bakirova, R Y: 1 article (11/2015)|
|7.||Gogebashvili, N: 1 article (11/2015)|
|8.||Kolesnikova, Y A: 1 article (11/2015)|
|9.||Molotov-Luchanskiy, V B: 1 article (11/2015)|
|10.||Sujashvili, R: 1 article (11/2015)|
01/01/2003 - "Southeast Asian ovalocytosis (SAO) is a red blood cell abnormality common in malaria-endemic regions and caused by a 27 nt deletion of the band 3 protein gene. "
08/01/2002 - "We hypothesize that altered red-cell membrane band 3 protein may be a target for enhanced antibody binding to alpha-thalassaemic IRBCs and could be involved in the mechanism of malaria protection."
05/01/1995 - "Sera from an area highly endemic for malaria, The Gambia, were found to have strong reactivity to well-defined regions of the band 3 protein (some of which contain the adhesin), whereas minimal reactivity was observed with sera from individuals living in a geographic area where malaria transmission is rare (California). "
05/01/1995 - "Plasmodium falciparum: sera of individuals living in a malaria-endemic region recognize peptide motifs of the human erythrocyte anion transport protein."
04/01/1994 - "Cytoadherence-related neoantigens on Plasmodium falciparum (human malaria)-infected human erythrocytes result from the exposure of normally cryptic regions of the band 3 protein."
01/01/1998 - "Band 3 Tokyo: Thr837-->Ala837 substitution in erythrocyte band 3 protein associated with spherocytic hemolysis."
02/01/1993 - "Participation of band 3 protein in hypotonic hemolysis of human erythrocytes."
09/01/1977 - "These results suggest that the intracellular alkalinization process leading to a structural instability of the membrane band 3 protein is responsible for this calf cell hemolysis."
09/01/1977 - "Hemolysis is progressively inhibited as the band 3 protein undergoes proteolysis, until a total inhibition of hemolysis takes place when almost all of the band 3 protein is digested into smaller protein components with a mol wt of 65,000 and 35,000 daltons. "
11/25/1987 - "When pyridoxal 5'-phosphate (PLP) is covalently bound to band 3 protein in intact red blood cells and those cells are subjected to the osmotic hemolysis and resealing process, a significant reduction in the original PLP anion transport inhibitory potency occurs. "
05/01/2008 - "Study of a novel band 3 hereditary spherocytosis variant suggests that expression of mutant band 3 protein can be rescued by wild-type band 3. Other studies show that some mutant band 3 protein can mediate a cation conductance. "
01/24/2006 - "Structure and stability of hereditary spherocytosis mutants of the cytosolic domain of the erythrocyte anion exchanger 1 protein."
10/01/1999 - "Analysis of red cell membrane revealed deficiency of band 3 protein in all cases of silent form of hereditary spherocytosis."
01/01/1994 - "These results on structural and functional properties of protein band 3 deficient erythrocytes may contribute to a better understanding of this newly discovered form of hereditary spherocytosis."
01/01/1994 - "Hereditary spherocytosis associated with protein band 3 defect in a Swiss kindred."
|4.||Respiratory Insufficiency (Respiratory Failure)
08/01/1995 - "[A study on erythrocyte membrane band 3 protein anion transport function in chronic respiratory failure patients]."
08/01/1995 - "(2) band 3 protein anion transport function impairment in patients of cor pulmonale with type II respiratory failure is reversible. "
08/01/1995 - "The results showed that (1) anion transport function impairment of erythrocyte membrane band 3 protein and HCO3-/Cl- exchange restrain may be the reasons that aggravate CO2 retention and respiratory acidosis in cor pulmonale patients with type II respiratory failure. "
05/01/1992 - "These findings suggested that (1) The relative composition reduction of erythrocyte membrane band 3 protein and HCO3-/Cl- exchange restrain may be one of the reasons that aggravated CO2 retention and respiratory acidosis in cor pulmonale patients with type II respiratory failure. "
12/01/1995 - "[The effect of furosemide and valium on band 3 protein anion transport function in chronic respiratory failure patients]."
03/01/1994 - "Finally, recovery from intracellular alkalosis induced by incubation in 0 mM Cl-, 50 mM HCO3- extracellular solution required Cl- and was independent of Na+. These studies indicate that the major mechanism of HCO3- transport in primary cultures of the rabbit IMCD is via a band 3 protein-negative, Na(+)-independent, basolateral, Cl-/HCO3- exchanger."
02/01/1992 - "We examined the effect of Cl- depletion metabolic alkalosis (CDA) on H(+)-ATPase and band 3 protein localization in intercalated cells (IC) of the rat cortical collecting duct (CCD) and the outer medullary collecting duct (OMCD). "
01/01/1997 - "Using indirect immunofluorescence, we examined the localization and abundance of the vacuolar H(+)-ATPase and the AE1 anion exchanger in intercalated cells (IC) of rat kidney connecting segment (CNT) and CCD during acute (6 hr) metabolic (NH4Cl) acidosis and respiratory (NaHCO3) alkalosis. "
01/01/1997 - "Regulation of AE1 anion exchanger and H(+)-ATPase in rat cortex by acute metabolic acidosis and alkalosis."
|3.||Proteins (Proteins, Gene)
|6.||dirhodium tetraacetate (DRTA)
|8.||Membrane Proteins (Integral Membrane Proteins)
|9.||Proton-Translocating ATPases (ATPase, H+)
|10.||Messenger RNA (mRNA)
|1.||Renal Dialysis (Hemodialysis)