|1.||Lee, Suk Hee: 1 article (12/2014)|
|2.||Parekh, Puja K: 1 article (12/2014)|
|3.||Liedtke, Wolfgang: 1 article (12/2014)|
|4.||Fang, Quan: 1 article (12/2014)|
|5.||Gereau, Robert W: 1 article (12/2014)|
|6.||Lee, Whasil: 1 article (12/2014)|
|7.||Kanju, Patrick: 1 article (12/2014)|
|8.||Brenner, Daniel: 1 article (12/2014)|
|9.||Moore, Carlene: 1 article (12/2014)|
|10.||Chen, Yong: 1 article (12/2014)|
04/01/2003 - "Recent advances in the understanding of the mechanisms of pain in general and chronic pain in particular, opened the field of analgesic therapy to newer pharmacological targets, which are aimed at improved efficacy and enhanced tolerability over conventional antipain treatments. "
11/01/2000 - "Positive results were due to restoration of self-regulation in pain and antipain systems which are disturbed in TN patients."
04/01/2009 - "We hypothesize that acupuncture may mediate its antipain, antianxiety, and other therapeutic effects via this intrinsic neural circuit that plays a central role in the affective and cognitive dimensions of pain as well as in the regulation and integration of emotion, memory processing, autonomic, endocrine, immunological, and sensorimotor functions."
01/01/2007 - "[Pain relief...quick!!!, an explanatory pamphlet on the role of each link in the antipain chain]."
10/01/2001 - "The main goal was to evaluate the motor and somatosensory systems by recording evoked motor responses (EMRs) during transcranial magnetic stimulation (TMS) and somatosensory evoked potentials (SEPs) in patients with neurogenic pain syndromes before and after implantation of the systems for chronic antipain epidural stimulation. "
|2.||Chromosome Aberrations (Chromosome Abnormalities)
07/01/1989 - "Although the exact mechanism whereby antipain decreases the yield of chromosome aberrations induced by the S-independent agent X-rays is unknown, these data indicate that the formation of chromosome aberrations by S-independent agents too can involve an antipain-sensitive process."
07/01/1989 - "Experiments have now been carried out to see if antipain might also effect the yield of aberrations induced by X-rays, which are S-independent and thus produce chromosomal aberrations by a different mechanism. "
07/01/1989 - "Antipain-mediated suppression of X-ray-induced chromosomal aberrations in human lymphocytes."
11/01/1980 - "Antipain inhibits N-methyl-N'-nitro-N-nitrosoguanidine-induced transformation and increases chromosomal aberrations."
06/01/1980 - "We show that antipain inhibited MNNG-induced chromosomal exchanges and all other chromosomal aberrations exclusively. "
|3.||Muscular Dystrophies (Muscular Dystrophy)
07/01/1988 - "This conclusion is supported by experiments with substances such as antipain and eicosa-5,8,11,14-tetraynoic acid which inhibit both tumor induction in initiated skin and the cytogenetic alterations induced by TPA in cultured keratinocytes. "
07/01/1980 - "Our results, together with the recent results of Loveday and Latt, may argue against the notion that TPA enhances the antipain-suppressible SCEs as an index of mitotic recombination in relevance with a tumor-promotion mechanism."
05/01/2010 - "An antipain care model is proposed for patients with chronic non-cancer pain, which makes it possible to optimize the treatment of the patients, to train pain specialists, and to enhance the economic efficiency of management."
12/01/1988 - "It was found that antipain suppressed the radiation-induced expression of the tumor-associated antigen in all treatment protocols. "
07/01/1980 - "The effects of a tumor promoter 12-O-tetradecanoylphorbol-13-acetate (TPA) and/or an anti-promoter antipain (protease inhibitor) on spontaneous and ultraviolet-induced sister-chromatid exchanges (SCEs) and 6-thioguanine-resistant (6TGr) recessive mutations were examined in V79 Chinese hamster cells in culture. "
|5.||Xeroderma Pigmentosum (Kaposi's Disease)
09/01/1980 - "Antipain had little effect on UV-survival and UV-induced sister chromatid exchanges in normal and xeroderma pigmentosum (XP) cells, suggesting that it may not affect DNA repair. "
09/01/1980 - "1. High sensitivity of xeroderma pigmentosum cells to antipain."
11/01/1994 - "Antipain also prevented the suppression of UV-mutagenicity by HuIFN-alpha in RSa and xeroderma pigmentosum-derived fibroblast cells, as shown by culturing cells in medium containing antipain immediately after UV exposure and evaluating the generation of clones resistant to ouabain- or 6-thioguanine-mediated cytotoxicity. "
|2.||pepstatin (pepstatin A)
|4.||Free Radical Scavengers
|5.||Dihydrotachysterol (AT 10)
|7.||Protease Inhibitors (Protease Inhibitor)