|1.||Boison, Detlev: 21 articles (08/2015 - 01/2004)|
|2.||Kowaluk, E A: 7 articles (06/2001 - 03/2000)|
|3.||Jarvis, M F: 7 articles (06/2001 - 03/2000)|
|4.||Li, Tianfu: 5 articles (02/2015 - 11/2007)|
|5.||Liou, Gregory I: 5 articles (01/2015 - 07/2013)|
|6.||Simon, Roger P: 5 articles (01/2008 - 01/2007)|
|7.||Ahmad, Saif: 4 articles (01/2015 - 07/2013)|
|8.||Fulzele, Sadanand: 4 articles (01/2015 - 07/2013)|
|9.||Elsherbiny, Nehal M: 4 articles (12/2014 - 07/2013)|
|10.||Williams, M: 4 articles (06/2001 - 12/2000)|
06/15/2008 - "HIF-1-dependent repression of adenosine kinase attenuates hypoxia-induced vascular leak."
02/01/1997 - "Adenosine kinase isolated from guinea pig hearts was inhibited by [P(i)] values seen during hypoxia or hypoperfusion. "
12/01/1994 - "In guinea pig isolated hearts, PD 81,723 (5 mumol/L) increased by twofold the stimulus-to-His bundle (S-H) interval prolongations induced by both a 5-minute period of hypoxia (25% O2/70% N2/5% CO2) and the administration of the adenosine kinase inhibitor iodotubercidin (40 to 70 nmol/L) but had no effect on coronary conductance. "
07/15/2014 - "Notably, UCP2 overexpression increased signaling from the master energy-regulating kinase, adenosine monophosphate-activated protein kinase, while downregulating expression of hypoxia-induced factor. "
08/01/1997 - "Hypoxia-induced inhibition of adenosine kinase potentiates cardiac adenosine release."
11/01/1997 - "An adenosine kinase-ineffective analog of neplanocin A was effective in inducing differentiation, suggesting that phosphorylation of the nucleoside is not essential for inducing the differentiation of leukemia cells. "
11/01/1979 - "Activities of adenosine deaminase (ADA), adenosine kinase (AK), adenine phosphoribosyltransferase (APRT), hypoxanthine guanine phosphoribosyltransferase (HGPRT), and purine nucleoside phosphorylase (PNP), all enzymes of the purine interconversion system, were determined in lymphocytes of 25 patients with chronic lymphatic leukemia (CLL) and in 23 controls. "
|3.||Septic Shock (Toxic Shock Syndrome)
10/01/2015 - "Adenosine kinase (AK) inhibitor is a potential candidate for controlling pain, but some AK inhibitors have problems of adverse effects such as motor impairment. "
01/01/2013 - "Preclinical neuropathic and postsurgical pain studies implicate the kinase adenosine monophosphate activated kinase (AMPK) as a potential pharmacological target for the treatment of chronic pain conditions. "
11/19/1999 - "Spinal administration of an adenosine kinase inhibitor, alone or in combination with an adenosine deaminase inhibitor, produces antinociception in inflammatory pain tests. "
04/19/2004 - "Adenosine kinase inhibition is an attractive therapeutic approach for several conditions for example, neurodegeneration, seizures, ischemia, inflammation and pain. "
09/19/1996 - "Inhibition of adenosine metabolism by administration of an adenosine kinase inhibitor, in the present study, decreased behavior induced by putative pain neurotransmitters providing additional support for an endogenous purinergic system. "
|10.||Purinergic P1 Receptors (Adenosine Receptor)